Muscle ring finger 1 mediates cardiac atrophy in vivo. - Wikidata - awgldk - TriplyDB

Muscle ring finger 1 mediates cardiac atrophy in vivo.

Muscle ring finger 1 mediates cardiac atrophy in vivo.

http://www.wikidata.org/entity/Q34603561

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Atrogin-1 and MuRF1 regulate cardiac MyBP-C levels via different mechanisms The ubiquitin-proteasome system and cardiovascular disease Targeting the ubiquitin-proteasome system in heart disease: the basis for new therapeutic strategies Activation of AMPK inhibits cardiomyocyte hypertrophy by modulating of the FOXO1/MuRF1 signaling pathway in vitro Muscle RING finger-1 promotes a maladaptive phenotype in chronic hypoxia-induced right ventricular remodeling.Sent to destroy: the ubiquitin proteasome system regulates cell signaling and protein quality control in cardiovascular development and disease.MAFbx/Atrogin-1 is required for atrophic remodeling of the unloaded heart.MuRF1 activity is present in cardiac mitochondria and regulates reactive oxygen species production in vivo MuRF1 mono-ubiquitinates TRα to inhibit T3-induced cardiac hypertrophy in vivo.The ubiquitin proteasome system in human cardiomyopathies and heart failure.Cardiac muscle ring finger-1--friend or foe?Natural aminoacyl tRNA synthetase fragment enhances cardiac function after myocardial infarction.Tearin' up my heart: proteolysis in the cardiac sarcomere The ubiquitin ligase MuRF1 protects against cardiac ischemia/reperfusion injury by its proteasome-dependent degradation of phospho-c-Jun.NF-κB inhibition protects against tumor-induced cardiac atrophy in vivo.Tear me down: role of calpain in the development of cardiac ventricular hypertrophy.Regulation of the calpain and ubiquitin-proteasome systems in a canine model of muscular dystrophy.Cardiac proteasome activity in muscle ring finger-1 null mice at rest and following synthetic glucocorticoid treatment.Proteasome functional insufficiency in cardiac pathogenesis.JNK modulates FOXO3a for the expression of the mitochondrial death and mitophagy marker BNIP3 in pathological hypertrophy and in heart failure.IMP 2.0: a multi-species functional genomics portal for integration, visualization and prediction of protein functions and networks.The ubiquitin ligase MuRF1 regulates PPARα activity in the heart by enhancing nuclear export via monoubiquitination.Non-targeted metabolomics of Brg1/Brm double-mutant cardiomyocytes reveals a novel role for SWI/SNF complexes in metabolic homeostasis.Non-targeted metabolomics analysis of cardiac Muscle Ring Finger-1 (MuRF1), MuRF2, and MuRF3 in vivo reveals novel and redundant metabolic changes.Fenofibrate unexpectedly induces cardiac hypertrophy in mice lacking MuRF1.Breaking down protein degradation mechanisms in cardiac muscle.Oral resveratrol therapy inhibits cancer-induced skeletal muscle and cardiac atrophy in vivo Mechanical unloading activates FoxO3 to trigger Bnip3-dependent cardiomyocyte atrophy.Transcriptional activation of muscle atrophy promotes cardiac muscle remodeling during mammalian hibernation.Chronic alcohol consumption disrupts myocardial protein balance and function in aged, but not adult, female F344 rats.Muscle RING finger-1 attenuates IGF-I-dependent cardiomyocyte hypertrophy by inhibiting JNK signaling.The role of ubiquitin ligases in cardiac disease.Ubiquitin and ubiquitin-like proteins in cardiac disease and protection.Atrophied cardiomyocytes and their potential for rescue and recovery of ventricular function.Cardiac ubiquitin ligases: Their role in cardiac metabolism, autophagy, cardioprotection and therapeutic potential.Ubiquitin Ligases and Posttranslational Regulation of Energy in the Heart: The Hand that Feeds.The Calcineurin-FoxO-MuRF1 signaling pathway regulates myofibril integrity in cardiomyocytes.Atrophic cardiomyocyte signaling in hypertensive heart disease Small dedifferentiated cardiomyocytes bordering on microdomains of fibrosis: evidence for reverse remodeling with assisted recovery.Developmental MYH3 Myopathy Associated with Expression of Mutant Protein and Reduced Expression Levels of Embryonic MyHC.

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P2860

Muscle ring finger 1 mediates cardiac atrophy in vivo.

http://www.wikidata.org/entity/Q34603561

description

2009 nî lūn-bûn

@nan

2009 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Muscle ring finger 1 mediates cardiac atrophy in vivo.

@ast

Muscle ring finger 1 mediates cardiac atrophy in vivo.

@en

Muscle ring finger 1 mediates cardiac atrophy in vivo.

@nl

type

label

Muscle ring finger 1 mediates cardiac atrophy in vivo.

@ast

Muscle ring finger 1 mediates cardiac atrophy in vivo.

@en

Muscle ring finger 1 mediates cardiac atrophy in vivo.

@nl

prefLabel

Muscle ring finger 1 mediates cardiac atrophy in vivo.

@ast

Muscle ring finger 1 mediates cardiac atrophy in vivo.

@en

Muscle ring finger 1 mediates cardiac atrophy in vivo.

@nl

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AJPHEART.00660.2008

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American Journal of Physiology - Heart and Circulatory Physiology

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Muscle ring finger 1 mediates cardiac atrophy in vivo.

@en

P2093

Cam Patterson

http://www.w3.org/2001/XMLSchema#string

Craig H Selzman

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David J Glass

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Jessica E Rodriguez

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Luge Li

http://www.w3.org/2001/XMLSchema#string

Mauricio Rojas

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Monte S Willis

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Ru-Hang Tang

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William E Stansfield

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P2860

Identification of muscle specific ring finger proteins as potential regulators of the titin kinase domain

Muscle-specific RING finger-1 interacts with titin to regulate sarcomeric M-line and thick filament structure and may have nuclear functions via its interaction with glucocorticoid modulatory element binding protein-1

Muscle-specific RING finger 1 is a bona fide ubiquitin ligase that degrades cardiac troponin I

Myosin accumulation and striated muscle myopathy result from the loss of muscle RING finger 1 and 3

Molecular determinants for the recruitment of the ubiquitin-ligase MuRF-1 onto M-line titin

MURF-1 and MURF-2 target a specific subset of myofibrillar proteins redundantly: towards understanding MURF-dependent muscle ubiquitination

Hypertrophic cardiomyopathy associated with dexamethasone therapy for bronchopulmonary dysplasia

Effect of clenbuterol on skeletal muscle atrophy in mice induced by the glucocorticoid dexamethasone

Identification of ubiquitin ligases required for skeletal muscle atrophy

Prognostic implications of echocardiographically determined left ventricular mass in the Framingham Heart Study

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10.1152/AJPHEART.00660.2008

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4

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