Recruitment of damaged DNA to the nuclear matrix in hamster cells following ultraviolet irradiation. - Wikidata - awgldk - TriplyDB

Recruitment of damaged DNA to the nuclear matrix in hamster cells following ultraviolet irradiation.

Recruitment of damaged DNA to the nuclear matrix in hamster cells following ultraviolet irradiation.

http://www.wikidata.org/entity/Q34605597

about

Translocation of Cockayne syndrome group A protein to the nuclear matrix: possible relevance to transcription-coupled DNA repair DNA end-independent activation of DNA-PK mediated via association with the DNA-binding protein C1D Functional TFIIH is required for UV-induced translocation of CSA to the nuclear matrix Human alpha spectrin II and the Fanconi anemia proteins FANCA and FANCC interact to form a nuclear complex The Pso4 mRNA splicing and DNA repair complex interacts with WRN for processing of DNA interstrand cross-links Transcription-coupled DNA repair: two decades of progress and surprises Radiation down-regulates replication origin activity throughout the S phase in mammalian cells.Reduced extractability of the XPA DNA repair protein in ultraviolet light-irradiated mammalian cells.DNA interstrand crosslink repair in mammalian cells: step by step.DNA-PK-dependent binding of DNA ends to plasmids containing nuclear matrix attachment region DNA sequences: evidence for assembly of a repair complex.Regulation of nucleotide excision repair in bacteria and mammalian cells.Effects of heat shock on the Mre11/Rad50/Nbs1 complex in irradiated or unirradiated cells.DNA break repair: refined rules of an already complicated game.Clusters of S1 nuclease-hypersensitive sites induced in vivo by DNA damage Maintenance of genome stability by Fanconi anemia proteins.Nuclear organization of nucleotide excision repair is mediated by RING1B dependent H2A-ubiquitylation.Transcription-coupled repair is inducible in hamster cells.Dephosphorylation and subcellular compartment change of the mitotic Bloom's syndrome DNA helicase in response to ionizing radiation.Transcriptionally active and inactive mouse beta-globin gene loci are repaired at similar rates after ultraviolet irradiation.Selected nuclear matrix proteins are targets for poly(ADP-ribose)-binding.Expression of wild-type p53 is required for efficient global genomic nucleotide excision repair in UV-irradiated human fibroblasts.KIAA1530 protein is recruited by Cockayne syndrome complementation group protein A (CSA) to participate in transcription-coupled repair (TCR).Analysis of nucleotide excision repair by detection of single-stranded DNA transients.Nuclear α-catenin mediates the DNA damage response via β-catenin and nuclear actin.

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Recruitment of damaged DNA to the nuclear matrix in hamster cells following ultraviolet irradiation.

http://www.wikidata.org/entity/Q34605597

description

1996 nî lūn-bûn

@nan

1996 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

1996 թվականի օգոստոսին հրատարակված գիտական հոդված

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1996年の論文

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1996年論文

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1996年論文

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1996年論文

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1996年論文

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1996年論文

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1996年论文

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name

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Recruitment of damaged DNA to ...... owing ultraviolet irradiation.

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Recruitment of damaged DNA to ...... owing ultraviolet irradiation.

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Recruitment of damaged DNA to ...... owing ultraviolet irradiation.

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Nucleic Acids Research

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Hanawalt PC

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Koehler DR

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P2860

Formation of a ternary complex by human XPA, ERCC1, and ERCC4(XPF) excision repair proteins

XPG endonuclease makes the 3' incision in human DNA nucleotide excision repair

Human DNA repair excision nuclease. Analysis of the roles of the subunits involved in dual incisions by using anti-XPG and anti-ERCC1 antibodies

Specific association between the human DNA repair proteins XPA and ERCC1

The general transcription-repair factor TFIIH is recruited to the excision repair complex by the XPA protein independent of the TFIIE transcription factor

Specific cleavage of model recombination and repair intermediates by the yeast Rad1-Rad10 DNA endonuclease

Dual role of TFIIH in DNA excision repair and in transcription by RNA polymerase II

Enhancement of damage-specific DNA binding of XPA by interaction with the ERCC1 DNA repair protein

Selective removal of transcription-blocking DNA damage from the transcribed strand of the mammalian DHFR gene

Matrix attachment regions are positioned near replication initiation sites, genes, and an interamplicon junction in the amplified dihydrofolate reductase domain of Chinese hamster ovary cells.

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