Nox2 modification of LDL is essential for optimal apolipoprotein B-mediated control of agr type III Staphylococcus aureus quorum-sensing.
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What a Dinner Party! Mechanisms and Functions of Interkingdom Signaling in Host-Pathogen Associations.Selective chemical inhibition of agr quorum sensing in Staphylococcus aureus promotes host defense with minimal impact on resistanceThe Staphylococcal Biofilm: Adhesins, Regulation, and Host ResponseVLP-based vaccine induces immune control of Staphylococcus aureus virulence regulation.Antiinfective therapy with a small molecule inhibitor of Staphylococcus aureus sortase.Rapid in vivo detection of isoniazid-sensitive Mycobacterium tuberculosis by breath testDevelopment of a mimotope vaccine targeting the Staphylococcus aureus quorum sensing pathway.Targeting Staphylococcus aureus quorum sensing with nonpeptidic small molecule inhibitors.ω-Hydroxyemodin limits staphylococcus aureus quorum sensing-mediated pathogenesis and inflammation.Apolipoprotein B48, the Structural Component of Chylomicrons, Is Sufficient to Antagonize Staphylococcus aureus Quorum-Sensing.Role of Phenol-Soluble Modulins in Formation of Staphylococcus aureus Biofilms in Synovial Fluid.Serum Lipoproteins Are Critical for Pulmonary Innate Defense against Staphylococcus aureus Quorum Sensing.Impact of Environmental Cues on Staphylococcal Quorum Sensing and Biofilm Development.Novel approaches for the design and discovery of quorum-sensing inhibitors.Interfering with Bacterial Quorum Sensing.Cross-Talk between Staphylococcus aureus and Other Staphylococcal Species via the agr Quorum Sensing SystemTranscriptional downregulation of agr expression in Staphylococcus aureus during growth in human serum can be overcome by constitutively active mutant forms of the sensor kinase AgrC.Tuning of the Lethal Response to Multiple Stressors with a Single-Site Mutation during Clinical Infection by Staphylococcus aureus.Targeting Virulence in Staphylococcus aureus by Chemical Inhibition of the Accessory Gene Regulator System In Vivo.Staphylococcus aureus utilizes host-derived lipoprotein particles as sources of exogenous fatty acids.Host Nitric Oxide Disrupts Microbial Cell-to-Cell Communication to Inhibit Staphylococcal Virulence.
P2860
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P2860
Nox2 modification of LDL is essential for optimal apolipoprotein B-mediated control of agr type III Staphylococcus aureus quorum-sensing.
description
2013 nî lūn-bûn
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2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2013年の論文
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2013年論文
@yue
2013年論文
@zh-hant
2013年論文
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2013年論文
@zh-mo
2013年論文
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2013年论文
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name
Nox2 modification of LDL is es ...... ococcus aureus quorum-sensing.
@ast
Nox2 modification of LDL is es ...... ococcus aureus quorum-sensing.
@en
Nox2 modification of LDL is es ...... ococcus aureus quorum-sensing.
@nl
type
label
Nox2 modification of LDL is es ...... ococcus aureus quorum-sensing.
@ast
Nox2 modification of LDL is es ...... ococcus aureus quorum-sensing.
@en
Nox2 modification of LDL is es ...... ococcus aureus quorum-sensing.
@nl
prefLabel
Nox2 modification of LDL is es ...... ococcus aureus quorum-sensing.
@ast
Nox2 modification of LDL is es ...... ococcus aureus quorum-sensing.
@en
Nox2 modification of LDL is es ...... ococcus aureus quorum-sensing.
@nl
P2093
P2860
P50
P1433
P1476
Nox2 modification of LDL is es ...... lococcus aureus quorum-sensing
@en
P2093
Bradley O Elmore
Brett C Manifold-Wheeler
Cynthia H Spang
Erin K Sully
Hattie D Gresham
Jon K Femling
M Michal Peterson
Susan M Alexander
P2860
P304
P356
10.1371/JOURNAL.PPAT.1003166
P577
2013-02-14T00:00:00Z