Let-7: a regulator of the ERα signaling pathway in human breast tumors and breast cancer stem cells.
about
The insights of Let-7 miRNAs in oncogenesis and stem cell potencySide population cells as prototype of chemoresistant, tumor-initiating cellsPRMT2 and RORγ expression are associated with breast cancer survival outcomes.Abundant circulating microRNAs in breast cancer patients fluctuate considerably during neoadjuvant chemotherapy.MicroRNA-26a inhibits the growth and invasiveness of malignant melanoma and directly targets on MITF gene.MicroRNA profile in very young women with breast cancerSex, epilepsy, and epigenetics.DICER1 regulated let-7 expression levels in p53-induced cancer repression requires cyclin D1.miRNAs regulated by estrogens, tamoxifen, and endocrine disruptors and their downstream gene targets.Let-7 inhibits self-renewal of hepatocellular cancer stem-like cells through regulating the epithelial-mesenchymal transition and the Wnt signaling pathwayInhibition of mTOR sensitizes breast cancer stem cells to radiation-induced repression of self-renewal through the regulation of MnSOD and Akt.MiR-208a stimulates the cocktail of SOX2 and β-catenin to inhibit the let-7 induction of self-renewal repression of breast cancer stem cells and formed miR208a/let-7 feedback loop via LIN28 and DICER1Pri-let-7a-1 rs10739971 polymorphism is associated with gastric cancer prognosis and might affect mature let-7a expression.Higher expression of circulating miR-182 as a novel biomarker for breast cancer.Let-7a functions as a tumor suppressor in Ewing's sarcoma cell lines partly by targeting cyclin-dependent kinase 6.Role of microRNAs in maintaining cancer stem cells.Hyperthermia-driven aberrations of secreted microRNAs in breast cancer in vitro.TLR7 agonist induced repression of hepatocellular carcinoma via the TLR7-IKK-NF-κB-IL6 signaling pathway.Analysis of risk factors for post-operative complications and prognostic predictors of disease recurrence following definitive treatment of patients with esophageal cancer from two medical centers in Northwest China.The cell cycle- and insulin-signaling-inhibiting miRNA expression pattern of very small embryonic-like stem cells contributes to their quiescent state.Breast cancer stem-like cells are sensitized to tamoxifen induction of self-renewal inhibition with enforced Let-7c dependent on Wnt blocking.miR-367 stimulates Wnt cascade activation through degrading FBXW7 in NSCLC stem cells.Aberrant miRNA promoter methylation and EMT-involving miRNAs in breast cancer metastasis: Diagnosis and therapeutic implications.
P2860
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P2860
Let-7: a regulator of the ERα signaling pathway in human breast tumors and breast cancer stem cells.
description
2013 nî lūn-bûn
@nan
2013 թուականի Մարտին հրատարակուած գիտական յօդուած
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2013 թվականի մարտին հրատարակված գիտական հոդված
@hy
2013年の論文
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2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
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2013年论文
@wuu
name
Let-7: a regulator of the ERα ...... and breast cancer stem cells.
@ast
Let-7: a regulator of the ERα ...... and breast cancer stem cells.
@en
Let-7: a regulator of the ERα ...... and breast cancer stem cells.
@nl
type
label
Let-7: a regulator of the ERα ...... and breast cancer stem cells.
@ast
Let-7: a regulator of the ERα ...... and breast cancer stem cells.
@en
Let-7: a regulator of the ERα ...... and breast cancer stem cells.
@nl
prefLabel
Let-7: a regulator of the ERα ...... and breast cancer stem cells.
@ast
Let-7: a regulator of the ERα ...... and breast cancer stem cells.
@en
Let-7: a regulator of the ERα ...... and breast cancer stem cells.
@nl
P2093
P356
P1433
P1476
Let-7: a regulator of the ERα ...... and breast cancer stem cells.
@en
P2093
P304
P356
10.3892/OR.2013.2330
P577
2013-03-06T00:00:00Z