Pathogenic effect of interleukin-17A in induction of Sjögren's syndrome-like disease using adenovirus-mediated gene transfer - Wikidata - awgldk - TriplyDB

Pathogenic effect of interleukin-17A in induction of Sjögren's syndrome-like disease using adenovirus-mediated gene transfer

Pathogenic effect of interleukin-17A in induction of Sjögren's syndrome-like disease using adenovirus-mediated gene transfer

http://www.wikidata.org/entity/Q34614045

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T Regulatory and T Helper 17 Cells in Primary Sjögren's Syndrome: Facts and Perspectives The osteopontin transgenic mouse is a new model for Sjögren's syndrome NCR3/NKp30 contributes to pathogenesis in primary Sjogren's syndrome.The ratio of Th17/Treg cells as a risk indicator in early acute respiratory distress syndrome Altered balance of interleukin-13/interferon-gamma contributes to lacrimal gland destruction and secretory dysfunction in CD25 knockout model of Sjögren's syndrome.Sjögren's syndrome: studying the disease in mice.Mouse Models of Primary Sjogren's Syndrome.Expression of IL-17, IL-23 and their receptors in minor salivary glands of patients with primary Sjögren's syndrome IL-17 sequestration via salivary gland gene therapy in a mouse model of Sjogren's syndrome suppresses disease-associated expression of the putative autoantigen Klk1b22 The Clinical Relevance of IL-17-Producing CD4+CD161+ Cell and Its Subpopulations in Primary Sjögren's Syndrome Local delivery of AAV2-CTLA4IgG decreases sialadenitis and improves gland function in the C57BL/6.NOD-Aec1Aec2 mouse model of Sjögren's syndrome.Sjögren syndrome: what and where are we looking for?Inducible tertiary lymphoid structures, autoimmunity, and exocrine dysfunction in a novel model of salivary gland inflammation in C57BL/6 mice Gene therapy using IL-27 ameliorates Sjögren's syndrome-like autoimmune exocrinopathy.IL-22 regulation of functional gene expression in salivary gland cells.T Cell-Associated Cytokines in the Pathogenesis of Sjögren's Syndrome Animal models in autoimmune diseases: lessons learned from mouse models for Sjögren's syndrome.γδ T cells are essential effectors of type 1 diabetes in the nonobese diabetic mouse model.Effects of muscarinic acetylcholine 3 receptor(208-227) peptide immunization on autoimmune response in nonobese diabetic mice.Sexual dimorphic function of IL-17 in salivary gland dysfunction of the C57BL/6.NOD-Aec1Aec2 model of Sjögren's syndrome.Interferon-gamma deficiency protects against aging-related goblet cell loss.Pathogenic role of endogenous TNF-α in the development of Sjögren's-like sialadenitis and secretory dysfunction in non-obese diabetic mice.The T cell in Sjogren's syndrome: force majeure, not spectateur.The role of viruses in autoreactive B cell activation within tertiary lymphoid structures in autoimmune diseases.Autoimmunity in the pathogenesis and treatment of keratoconjunctivitis sicca.EBV and other viruses as triggers of tertiary lymphoid structures in primary Sjögren's syndrome.Th17 cells in autoimmune diseases.Update on Pathogenesis of Sjögren's syndrome.Clinical relevance of RORγ positive and negative subsets of CD161+CD4+ T cells in primary Sjögren's syndrome.Single-cell analysis reveals sexually dimorphic repertoires of Interferon-γ and IL-17A producing T cells in salivary glands of Sjögren's syndrome mice A case for IL-6, IL-17A, and nitric oxide in the pathophysiology of Sjögren's syndrome.The Pathophysiology of Dry Eye Disease: What We Know and Future Directions for Research.cDNA microarray analysis identifies NR4A2 as a novel molecule involved in the pathogenesis of Sjögren's syndrome.Proteasome inhibition suppresses Th17 cell generation and ameliorates autoimmune development in experimental Sjögren's syndrome.Interleukin-17 Impairs Salivary Tight Junction Integrity in Sjögren's Syndrome.Interleukin (IL)-17-producing pathogenic T lymphocytes co-express CD20 and are depleted by rituximab in primary Sjögren's syndrome: a pilot study.Dysregulation of the suppressor of cytokine signalling 3-signal transducer and activator of transcription-3 pathway in the aetiopathogenesis of Sjögren's syndrome.Expression of Gαq Is Decreased in Lymphocytes from Primary Sjögren's Syndrome Patients and Related to Increased IL-17A Expression.Beyond Horses to Zebras: Sicca Syndrome

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Pathogenic effect of interleukin-17A in induction of Sjögren's syndrome-like disease using adenovirus-mediated gene transfer

http://www.wikidata.org/entity/Q34614045

description

2010 nî lūn-bûn

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2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Pathogenic effect of interleuk ...... novirus-mediated gene transfer

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Pathogenic effect of interleuk ...... novirus-mediated gene transfer

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Pathogenic effect of interleuk ...... novirus-mediated gene transfer

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type

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Pathogenic effect of interleuk ...... novirus-mediated gene transfer

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Pathogenic effect of interleuk ...... novirus-mediated gene transfer

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Pathogenic effect of interleuk ...... novirus-mediated gene transfer

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Pathogenic effect of interleuk ...... novirus-mediated gene transfer

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Pathogenic effect of interleuk ...... novirus-mediated gene transfer

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Pathogenic effect of interleuk ...... novirus-mediated gene transfer

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Arthritis Research and Therapy

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Pathogenic effect of interleuk ...... novirus-mediated gene transfer

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Ammon B Peck

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Byung Ha Lee

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Cuong Q Nguyen

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Hongen Yin

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John A Chiorini

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Wendy C Carcamo

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P2860

TGF-beta-induced Foxp3 inhibits T(H)17 cell differentiation by antagonizing RORgammat function

Interleukin-23 drives innate and T cell-mediated intestinal inflammation

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

Transforming growth factor-beta induces development of the T(H)17 lineage

A genome-wide association study identifies IL23R as an inflammatory bowel disease gene

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17

Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages

Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain

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10.1186/AR3207

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6

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12

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3046533

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