TGFβ-dependent induction of interleukin-11 and interleukin-8 involves SMAD and p38 MAPK pathways in breast tumor models with varied bone metastases potential.
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Exposure to biomass smoke extract enhances fibronectin release from fibroblastsMutations in protein-binding hot-spots on the hub protein Smad3 differentially affect its protein interactions and Smad3-regulated gene expressionEmdogain-regulated gene expression in palatal fibroblasts requires TGF-βRI kinase signalingIL-11 produced by breast cancer cells augments osteoclastogenesis by sustaining the pool of osteoclast progenitor cells.Oncolytic adenovirus expressing soluble TGFβ receptor II-Fc-mediated inhibition of established bone metastases: a safe and effective systemic therapeutic approach for breast cancer.Breast cancer cells promote osteoblastic differentiation via Sema 3A signaling pathway in vitro.Cytokine biomarkers and chronic pain: association of genes, transcription, and circulating proteins with temporomandibular disorders and widespread palpation tendernessSystemic delivery of oncolytic adenoviruses targeting transforming growth factor-β inhibits established bone metastasis in a prostate cancer mouse model.Intravenous administration of adenoviruses targeting transforming growth factor beta signaling inhibits established bone metastases in 4T1 mouse mammary tumor model in an immunocompetent syngeneic host.Morphoproteomics provides support for TGF-β pathway signaling in the osteoclastogenesis and immune dysregulation of osteolytic Langerhans cell histiocytosis.Systemic Delivery of an Oncolytic Adenovirus Expressing Decorin for the Treatment of Breast Cancer Bone MetastasesCharacterization of breast cancer interstitial fluids by TmT labeling, LTQ-Orbitrap Velos mass spectrometry, and pathway analysis.TGF-β inhibition enhances chemotherapy action against triple-negative breast cancer.Autocrine production of IL-11 mediates tumorigenicity in hypoxic cancer cells.Regulation of SPARC by transforming growth factor β2 in human trabecular meshworkInterleukin-11 promotes epithelial-mesenchymal transition in anaplastic thyroid carcinoma cells through PI3K/Akt/GSK3β signaling pathway activation.RUNX2 in mammary gland development and breast cancer.Genetics of breast cancer bone metastasis: a sequential multistep pattern.Nerve growth factor exposure promotes tubular epithelial-mesenchymal transition via TGF-β1 signaling activation.Substance P increases CCN2 dependent on TGF-beta yet Collagen Type I via TGF-beta1 dependent and independent pathways in tenocytes.Bone metastasis from breast cancer involves elevated IL-11 expression and the gp130/STAT3 pathway.Lysophosphatidic acid enhances breast cancer cells-mediated osteoclastogenesis
P2860
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P2860
TGFβ-dependent induction of interleukin-11 and interleukin-8 involves SMAD and p38 MAPK pathways in breast tumor models with varied bone metastases potential.
description
2011 nî lūn-bûn
@nan
2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
TGFβ-dependent induction of in ...... ied bone metastases potential.
@ast
TGFβ-dependent induction of in ...... ied bone metastases potential.
@en
TGFβ-dependent induction of in ...... ied bone metastases potential.
@nl
type
label
TGFβ-dependent induction of in ...... ied bone metastases potential.
@ast
TGFβ-dependent induction of in ...... ied bone metastases potential.
@en
TGFβ-dependent induction of in ...... ied bone metastases potential.
@nl
prefLabel
TGFβ-dependent induction of in ...... ied bone metastases potential.
@ast
TGFβ-dependent induction of in ...... ied bone metastases potential.
@en
TGFβ-dependent induction of in ...... ied bone metastases potential.
@nl
P2093
P2860
P356
P1476
TGFβ-dependent induction of in ...... ied bone metastases potential.
@en
P2093
Janhavi Gupta
John Robbins
Tamas Jilling
P2860
P304
P356
10.4161/CBT.11.3.14096
P577
2011-02-01T00:00:00Z