Biology and pathogenesis of lymphocytic choriomeningitis virus infection.
about
Arenavirus reverse genetics: new approaches for the investigation of arenavirus biology and development of antiviral strategiesPathogenesis of Lassa feverReporter-Expressing, Replicating-Competent Recombinant ArenavirusesHost-Species Transferrin Receptor 1 Orthologs Are Cellular Receptors for Nonpathogenic New World Clade B ArenavirusesResponse and recovery in the plasma metabolome tracks the acute LCMV-induced immune responseType I interferon suppresses virus-specific B cell responses by modulating CD8(+) T cell differentiationPlatelets prevent IFN-alpha/beta-induced lethal hemorrhage promoting CTL-dependent clearance of lymphocytic choriomeningitis virus.Immunosuppression during acute infection with foot-and-mouth disease virus in swine is mediated by IL-10.Inhibition of cellular entry of lymphocytic choriomeningitis virus by amphipathic DNA polymersMyD88 plays a critical T cell-intrinsic role in supporting CD8 T cell expansion during acute lymphocytic choriomeningitis virus infection.Conventional but not plasmacytoid dendritic cells foster the systemic virus-induced type I IFN response needed for efficient CD8 T cell priming.Posttranslational modification of alpha-dystroglycan, the cellular receptor for arenaviruses, by the glycosyltransferase LARGE is critical for virus binding.Molecular indetermination in the transition to error catastrophe: systematic elimination of lymphocytic choriomeningitis virus through mutagenesis does not correlate linearly with large increases in mutant spectrum complexity.Molecular anatomy and number of antigen specific CD8 T cells required to cause type 1 diabetesReverse genetics approaches to combat pathogenic arenavirusesDecoding arenavirus pathogenesis: essential roles for alpha-dystroglycan-virus interactions and the immune response.Antigen-driven effector CD8 T cell function regulated by T-bet.Targeting Schwann cells by nonlytic arenaviral infection selectively inhibits myelinationAltered central nervous system gene expression caused by congenitally acquired persistent infection with lymphocytic choriomeningitis virus.Lentiviral transduction of dendritic cells confers protective antiviral immunity in vivoA highly optimized DNA vaccine confers complete protective immunity against high-dose lethal lymphocytic choriomeningitis virus challenge.Novel mechanism of arenavirus-induced liver pathology.Blockade of interferon Beta, but not interferon alpha, signaling controls persistent viral infectionLymphocytic Choriomeningitis Virus Differentially Affects the Virus-Induced Type I Interferon Response and Mitochondrial Apoptosis Mediated by RIG-I/MAVS.Evaluation of the anti-arenaviral activity of the subtilisin kexin isozyme-1/site-1 protease inhibitor PF-429242.An intermediate dose of LCMV clone 13 causes prolonged morbidity that is maintained by CD4+ T cells.HLA-A2-restricted protection against lethal lymphocytic choriomeningitis.Lassa virus infection in experimentally infected marmosets: liver pathology and immunophenotypic alterations in target tissuesOld World and clade C New World arenaviruses mimic the molecular mechanism of receptor recognition used by alpha-dystroglycan's host-derived ligands.Chimeric Mice with Competent Hematopoietic Immunity Reproduce Key Features of Severe Lassa Fever.Interaction between unrelated viruses during in vivo co-infection to limit pathology and immunityThe Roles and Perspectives of Toll-Like Receptors and CD4(+) Helper T Cell Subsets in Acute Viral Encephalitis.A Jekyll and Hyde Profile: Type 1 Interferon Signaling Plays a Prominent Role in the Initiation and Maintenance of a Persistent Virus Infection.Old World arenavirus infection interferes with the expression of functional alpha-dystroglycan in the host cellResolution of a chronic viral infection after interleukin-10 receptor blockadeHigh basal STAT4 balanced by STAT1 induction to control type 1 interferon effects in natural killer cells.D471G mutation in LCMV-NP affects its ability to self-associate and results in a dominant negative effect in viral RNA synthesisGeneration and maintenance of Listeria-specific CD8+ T cell responses in perforin-deficient mice chronically infected with LCMVUnraveling the functions of plasmacytoid dendritic cells during viral infections, autoimmunity, and tolerance.Natural killer cells act as rheostats modulating antiviral T cells.
P2860
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P2860
Biology and pathogenesis of lymphocytic choriomeningitis virus infection.
description
2002 nî lūn-bûn
@nan
2002 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Biology and pathogenesis of lymphocytic choriomeningitis virus infection.
@ast
Biology and pathogenesis of lymphocytic choriomeningitis virus infection.
@en
Biology and pathogenesis of lymphocytic choriomeningitis virus infection.
@nl
type
label
Biology and pathogenesis of lymphocytic choriomeningitis virus infection.
@ast
Biology and pathogenesis of lymphocytic choriomeningitis virus infection.
@en
Biology and pathogenesis of lymphocytic choriomeningitis virus infection.
@nl
prefLabel
Biology and pathogenesis of lymphocytic choriomeningitis virus infection.
@ast
Biology and pathogenesis of lymphocytic choriomeningitis virus infection.
@en
Biology and pathogenesis of lymphocytic choriomeningitis virus infection.
@nl
P1476
Biology and pathogenesis of lymphocytic choriomeningitis virus infection
@en
P2093
M B A Oldstone
P304
P356
10.1007/978-3-642-56055-2_6
P577
2002-01-01T00:00:00Z