TLR2 is constitutively expressed within the kidney and participates in ischemic renal injury through both MyD88-dependent and -independent pathways. - Wikidata - awgldk - TriplyDB

TLR2 is constitutively expressed within the kidney and participates in ischemic renal injury through both MyD88-dependent and -independent pathways.

TLR2 is constitutively expressed within the kidney and participates in ischemic renal injury through both MyD88-dependent and -independent pathways.

http://www.wikidata.org/entity/Q34625047

about

A TLR5 agonist inhibits acute renal ischemic failure How the Innate Immune System Senses Trouble and Causes Trouble Reverse signaling through the co-stimulatory ligand, CD137L, as a critical mediator of sterile inflammation Emerging therapies targeting intra-organ inflammation in transplantation Eya1 controls cell polarity, spindle orientation, cell fate and Notch signaling in distal embryonic lung epithelium Deconvoluting post-transplant immunity: cell subset-specific mapping reveals pathways for activation and expansion of memory T, monocytes and B cells Inflammation in AKI: Current Understanding, Key Questions, and Knowledge Gaps.Sisters in arms: myeloid and tubular epithelial cells shape renal innate immunity.Increased endothelial expression of Toll-like receptor 2 at sites of disturbed blood flow exacerbates early atherogenic events.Toll-like receptor-4 coordinates the innate immune response of the kidney to renal ischemia/reperfusion injury.Biglycan-triggered TLR-2- and TLR-4-signaling exacerbates the pathophysiology of ischemic acute kidney injury.Acute kidney injury: what part do toll-like receptors play?Glycyrrhizin Ameliorate Ischemia Reperfusion Lung Injury through Downregulate TLR2 Signaling Cascade in Alveolar Macrophages.Toll-like receptors in kidney disease.Amelioration of acute kidney injury in lipopolysaccharide-induced systemic inflammatory response syndrome by an aldose reductase inhibitor, fidarestat.Toll-like receptor 2 mediates inhibition of HCO(3)(-) absorption by bacterial lipoprotein in medullary thick ascending limb Multi-step pathogenesis and induction of local immune response by systemic Candida albicans infection in an intravenous challenge mouse model.Fresh and cryopreserved, uncultured adipose tissue-derived stem and regenerative cells ameliorate ischemia-reperfusion-induced acute kidney injury.HMGB1 contributes to kidney ischemia reperfusion injury.An inflammasome-independent role for epithelial-expressed Nlrp3 in renal ischemia-reperfusion injury Nod1 and nod2 are expressed in human and murine renal tubular epithelial cells and participate in renal ischemia reperfusion injury.Proximal tubule epithelial cell specific ablation of the spermidine/spermine N1-acetyltransferase gene reduces the severity of renal ischemia/reperfusion injury TLR2 and TLR4 mediate differential responses to limb ischemia through MyD88-dependent and independent pathways Type I interferon pathway mediates renal ischemia/reperfusion injury.Donor Toll-like receptor 4 contributes to ischemia and reperfusion injury following human kidney transplantation Toll-like receptors 2 and 4: initiators of non-septic inflammation in critical care medicine?TLR2 modulates antibodies required for intestinal ischemia/reperfusion-induced damage and inflammation Ischemia-reperfusion lung injury is attenuated in MyD88-deficient mice.Disruption of downstream MyD88 or TRIF Toll-like receptor signaling does not protect against cerebral ischemia The early activation of toll-like receptor (TLR)-3 initiates kidney injury after ischemia and reperfusion Differential activation of Toll-like receptor-mediated apoptosis induced by hypoxia Extracellular histones are mediators of death through TLR2 and TLR4 in mouse fatal liver injury Toll-like receptors (TLRs) in transplantation Different expressions of TLRs and related factors in peripheral blood of preterm infants.Reverse signaling through the costimulatory ligand CD137L in epithelial cells is essential for natural killer cell-mediated acute tissue inflammation.Toll-like receptor 4 promotes tubular inflammation in diabetic nephropathy.Differential toll-like receptor activation in lung ischemia reperfusion injury.Toll-like receptors in ischaemia and its potential role in the pathophysiology of muscle damage in critical limb ischaemia.Innate immunity in donor procurement.miR-628, a microRNA that is induced by Toll-like receptor stimulation, regulates porcine innate immune responses

P2860

Q24635867-45906E75-D018-46DC-96FF-1CC7D197B0B8 Q27016140-772715CB-E00E-4C84-9278-D7F6E1116B51 Q27022830-FA42FE50-7365-455C-B4F3-1258F6EA1F3A Q28085496-41B3F8C8-D234-4533-BADE-07074039D291 Q28593989-30866F03-D883-4E42-8C11-767E80E727AB Q28748866-FB871909-9D43-4080-91C0-32EF9595EF47 Q30278352-08447569-5D16-4F38-A9C7-00EDB5BC4362 Q30539710-6EA53D49-5EF7-481E-AE6D-64DC82A84E05 Q33318184-369BC014-882C-438E-B26C-FA2AE52F70BA Q33381642-43E13F4B-05AB-4FA3-AB07-B9E658C5EC76 Q33752945-7E65E44D-5F44-491F-88D8-07CFB3F5D535 Q33800669-0B3F987E-0485-4B91-AC45-559FA5A124DF Q33804507-EC91FF9E-8FC2-4CE3-BB63-80B53DD249B2 Q33960964-15553026-74A7-4594-BEE7-0833510F7BB7 Q34131121-993F7067-680D-4CFB-800D-3A3304D9DD7B Q34149622-EA415F56-3A5C-4749-9A15-D41025889839 Q34158734-35798C77-9C35-4BBE-8094-D9884312E83D Q34340727-664D56C2-5664-4ED2-9881-AB7D205DBBD9 Q34457038-BAD21445-0EA7-4998-B051-7F43527FE4F3 Q34485850-57F27812-1098-4588-9E2D-E32BF0D9C29E Q34485856-58826740-AC5B-43C7-94EF-83C48634F5E1 Q34497328-A06D1D04-0DCC-440C-B44D-7A17E432B82A Q34500561-C2CE30F1-CD1E-4297-A50F-C719BF13560E Q34557372-3D15AE7F-F0F1-4D6F-9D17-FDE2BC8D99B4 Q34945273-E726C20A-0CFE-48CC-8FD7-BA359E0737EC Q34956130-9DE955B3-0BAF-413D-8534-D311DD016BE9 Q34980207-0EFD959D-E48E-4725-80C3-C70B559679B5 Q35023053-FC90CB2E-60E2-4C21-9E27-DC32B4537DF1 Q35076476-475C8B5B-C115-4929-9A4A-D902415E9F5F Q35149040-0C6EA4A5-1AFA-4FA2-B8F0-599518BE56E0 Q35169612-24815810-476B-49FC-9FB2-399623D1B7DB Q35175597-EB268109-8BD5-439D-8EA6-3C1E062DC197 Q35581147-C14E3BF2-090F-49D9-BAF2-FA8EB8BC4C65 Q35643924-E0C466FA-8E1C-4410-918C-EB17D927FAB9 Q35657838-E2F2EFBA-CDF1-472D-892A-3CC0AAB463AF Q35720839-E788721B-7440-4CD7-B069-D5748334D223 Q35732768-BE9D3791-9C53-4F85-98A4-903E8A51796B Q35791682-19726CCC-9CA4-45A0-8B67-73A928B56598 Q35889959-4E99616C-EBFE-4B68-89D5-DC3BAEBD7DDA Q35905794-1F7A010C-5341-4D9A-90E7-F535B0B40262

P2860

TLR2 is constitutively expressed within the kidney and participates in ischemic renal injury through both MyD88-dependent and -independent pathways.

http://www.wikidata.org/entity/Q34625047

description

2007 nî lūn-bûn

@nan

2007 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2007年の論文

@ja

2007年論文

@yue

2007年論文

@zh-hant

2007年論文

@zh-hk

2007年論文

@zh-mo

2007年論文

@zh-tw

2007年论文

@wuu

name

TLR2 is constitutively express ...... ent and -independent pathways.

@ast

TLR2 is constitutively express ...... ent and -independent pathways.

@en

TLR2 is constitutively express ...... ent and -independent pathways.

@nl

type

label

TLR2 is constitutively express ...... ent and -independent pathways.

@ast

TLR2 is constitutively express ...... ent and -independent pathways.

@en

TLR2 is constitutively express ...... ent and -independent pathways.

@nl

prefLabel

TLR2 is constitutively express ...... ent and -independent pathways.

@ast

TLR2 is constitutively express ...... ent and -independent pathways.

@en

TLR2 is constitutively express ...... ent and -independent pathways.

@nl

P1433

Q34625047-CC27F60D-9113-4B0A-86AD-5EEEB6091CA7

P1476

Q34625047-A8BA728E-6840-4FD7-A40B-3FCE9FF9D809

P2093

Q34625047-092D37E9-9DD8-4FDC-8F2D-DAF50B5C3D57

Q34625047-35AFC54C-09E1-4A28-9EEF-1F23128F3A87

Q34625047-41CAD7B6-BAD5-4CFB-93D6-3451DA5E35D7

Q34625047-6F616656-7DCB-4D38-9982-7C7D5641D122

Q34625047-B0886599-6DB3-404E-BABA-08D2330D2B38

Q34625047-B925FDC6-6408-4626-AA69-A1BC99AA4DFF

Q34625047-C3E36E78-C65D-42DF-982A-BFC9EB0F035E

Q34625047-FF1470D8-D09A-474F-BD3D-AF1F21D53D84

P304

Q34625047-DEA51FCF-A22A-4A4F-8614-54D5C5459D9E

P31

Q34625047-4CBC045E-7222-4D4F-B470-C83EB67C8C62

P356

Q34625047-6EB1C145-7E15-4BA8-8E2C-2F2D02003741

P407

Q34625047-D9860200-B551-4413-A83A-A84E51C2A41B

P433

Q34625047-B20A320B-A2CE-47C9-9248-0F3DABE60736

P478

Q34625047-5BE7381A-EBDC-4B91-8468-E1C9ACBDB05D

P577

Q34625047-1A2D9F86-F98A-47FE-BC76-18D303199582

P5875

Q34625047-7E7AB228-7BD4-438E-A319-81F930784E6D

P698

Q34625047-9795A275-B14F-4276-8D0B-952F6009A49C

P921

Q34625047-A3640E9C-F589-4BBF-A6C8-64514A001D2C

P356

JIMMUNOL.178.10.6252

P1433

Journal of Immunology

P1476

TLR2 is constitutively express ...... ent and -independent pathways.

@en

P2093

Alana A Shigeoka

http://www.w3.org/2001/XMLSchema#string

Andrew J King

http://www.w3.org/2001/XMLSchema#string

Dianne B McKay

http://www.w3.org/2001/XMLSchema#string

Frank W Hall

http://www.w3.org/2001/XMLSchema#string

Nigel Mackman

http://www.w3.org/2001/XMLSchema#string

Peter S Tobias

http://www.w3.org/2001/XMLSchema#string

Todd D Holscher

http://www.w3.org/2001/XMLSchema#string

William B Kiosses

http://www.w3.org/2001/XMLSchema#string

P304

6252-6258

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.4049/JIMMUNOL.178.10.6252

http://www.w3.org/2001/XMLSchema#string

P407

P433

10

http://www.w3.org/2001/XMLSchema#string

P478

178

http://www.w3.org/2001/XMLSchema#string

P577

2007-05-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

6354069

http://www.w3.org/2001/XMLSchema#string

P698

17475853

http://www.w3.org/2001/XMLSchema#string

P921

Toll-like receptor