Analysis of phosphotyrosine signaling in glioblastoma identifies STAT5 as a novel downstream target of ΔEGFR.
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HER2 and uPAR cooperativity contribute to metastatic phenotype of HER2-positive breast cancerNeutralizing the EGF receptor in glioblastoma cells stimulates cell migration by activating uPAR-initiated cell signaling.Nuclear EGFRvIII-STAT5b complex contributes to glioblastoma cell survival by direct activation of the Bcl-XL promoter.Signal transducer and activator of transcription 5b drives malignant progression in a PDGFB-dependent proneural glioma model by suppressing apoptosis.Forced dimerization increases the activity of ΔEGFR/EGFRvIII and enhances its oncogenicity.Crosstalk between the urokinase-type plasminogen activator receptor and EGF receptor variant III supports survival and growth of glioblastoma cells.Optimization of titanium dioxide and immunoaffinity-based enrichment procedures for tyrosine phosphopeptide using matrix-assisted laser desorption/ionization time-of-flight mass spectrometry.Soluble Urokinase Receptor Is Released Selectively by Glioblastoma Cells That Express Epidermal Growth Factor Receptor Variant III and Promotes Tumor Cell Migration and Invasion.Integrative and regularized principal component analysis of multiple sources of dataCucurbitacin-I inhibits Aurora kinase A, Aurora kinase B and survivin, induces defects in cell cycle progression and promotes ABT-737-induced cell death in a caspase-independent manner in malignant human glioma cellsMolecular characterization of EGFR and EGFRvIII signaling networks in human glioblastoma tumor xenografts.Regulation of HGF expression by ΔEGFR-mediated c-Met activation in glioblastoma cellsEGFR wild type antagonizes EGFRvIII-mediated activation of Met in glioblastoma.DARPP32, STAT5 and STAT3 mRNA expression ratios in glioblastomas are associated with patient outcome.Combination of an anti-EGFRvIII antibody CH12 with Rapamycin synergistically inhibits the growth of EGFRvIII+PTEN-glioblastoma in vivo.EGFR-dependent mechanisms in glioblastoma: towards a better therapeutic strategy.Phosphoproteomic studies of receptor tyrosine kinases: future perspectives.A phase I study of temozolomide and lapatinib combination in patients with recurrent high-grade gliomas.Identification of a novel fusion gene HMGA2-EGFR in glioblastoma.EGFRvIII-Stat5 Signaling Enhances Glioblastoma Cell Migration and Survival.
P2860
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P2860
Analysis of phosphotyrosine signaling in glioblastoma identifies STAT5 as a novel downstream target of ΔEGFR.
description
2011 nî lūn-bûn
@nan
2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Analysis of phosphotyrosine si ...... el downstream target of ΔEGFR.
@ast
Analysis of phosphotyrosine si ...... el downstream target of ΔEGFR.
@en
Analysis of phosphotyrosine si ...... el downstream target of ΔEGFR.
@nl
type
label
Analysis of phosphotyrosine si ...... el downstream target of ΔEGFR.
@ast
Analysis of phosphotyrosine si ...... el downstream target of ΔEGFR.
@en
Analysis of phosphotyrosine si ...... el downstream target of ΔEGFR.
@nl
prefLabel
Analysis of phosphotyrosine si ...... el downstream target of ΔEGFR.
@ast
Analysis of phosphotyrosine si ...... el downstream target of ΔEGFR.
@en
Analysis of phosphotyrosine si ...... el downstream target of ΔEGFR.
@nl
P2093
P2860
P356
P1476
Analysis of phosphotyrosine si ...... el downstream target of ΔEGFR.
@en
P2093
Keith Baggerly
Khatri Latha
Lauren Hawley
Lixia Diao
Raymond Sawaya
Rebecca Maywald
Vaibhav Chumbalkar
Webster K Cavenee
YeoHyeon Hwang
P2860
P304
P356
10.1021/PR101075E
P577
2011-02-14T00:00:00Z