Crosstalk between sentinel and helper macrophages permits neutrophil migration into infected uroepithelium. - Wikidata - awgldk - TriplyDB

Crosstalk between sentinel and helper macrophages permits neutrophil migration into infected uroepithelium.

Crosstalk between sentinel and helper macrophages permits neutrophil migration into infected uroepithelium.

http://www.wikidata.org/entity/Q34643625

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Innate cell communication kick-starts pathogen-specific immunity Macrophages Subvert Adaptive Immunity to Urinary Tract Infection Pathogenesis of Streptococcus urinary tract infection depends on bacterial strain and β-hemolysin/cytolysin that mediates cytotoxicity, cytokine synthesis, inflammation and virulence Pathogenesis of human diffusely adhering Escherichia coli expressing Afa/Dr adhesins (Afa/Dr DAEC): current insights and future challenges Ly6C(low) and not Ly6C(high) macrophages accumulate first in the heart in a model of murine pressure-overload.VEGF-production by CCR2-dependent macrophages contributes to laser-induced choroidal neovascularization Are you experienced? Understanding bladder innate immunity in the context of recurrent urinary tract infection.Diagnosing inflammation and infection in the urinary system via proteomics.Inhibition of Cyclooxygenase-2 Prevents Chronic and Recurrent Cystitis.Splenic red pulp macrophages are intrinsically superparamagnetic and contaminate magnetic cell isolates The role of tissue resident cells in neutrophil recruitment Similar Neutrophil-Driven Inflammatory and Antibacterial Responses in Elderly Patients with Symptomatic and Asymptomatic Bacteriuria Endothelial CD36 Contributes to Postischemic Brain Injury by Promoting Neutrophil Activation via CSF3.Characterization of Early-Phase Neutrophil Extracellular Traps in Urinary Tract Infections Macrophage origin limits functional plasticity in helminth-bacterial co-infection Neutrophil Migration into the Infected Uroepithelium Is Regulated by the Crosstalk between Resident and Helper Macrophages.Histone Deacetylase 6 Regulates Bladder Architecture and Host Susceptibility to Uropathogenic Escherichia coli.The nature of immune responses to urinary tract infections Patrolling monocytes promote intravascular neutrophil activation and glomerular injury in the acutely inflamed glomerulus.Yersinia enterocolitica YopH-Deficient Strain Activates Neutrophil Recruitment to Peyer's Patches and Promotes Clearance of the Virulent Strain.Macrophagic control of the response to uropathogenic E. coli infection by regulation of iron retention in an IL-6-dependent manner.The Pathogenic Potential of Proteus mirabilis Is Enhanced by Other Uropathogens during Polymicrobial Urinary Tract Infection.High-Fat Diet Induces Unexpected Fatal Uterine Infections in Mice with aP2-Cre-mediated Deletion of Estrogen Receptor Alpha.Neutrophil migration in infection and wound repair: going forward in reverse.Human dendritic cell functional specialization in steady-state and inflammation.Neutrophils at work.Dendritic Cells and Macrophages: Sentinels in the Kidney.Sequential Immune Responses: The Weapons of Immunity Macrophages: sentinels and regulators of the immune system.Therapies in early development for the treatment of urinary tract inflammation.Innate Immune Responses to Bladder Infection.Macrophage heterogeneity and renin-angiotensin system disorders.Cryopreserved IL-4-treated macrophages attenuate murine colitis in an integrin β7-dependent manner Opposing role of tumor necrosis factor receptor 1 signaling in T cell-mediated hepatitis and bacterial infection in mice.UroPathogenic Escherichia coli (UPEC) Infections: Virulence Factors, Bladder Responses, Antibiotic, and Non-antibiotic Antimicrobial Strategies.Urinary tract infection: recent insight into the evolutionary arms race between uropathogenic Escherichia coli and our immune system.Inflammation drives renal scarring in experimental pyelonephritis.New insights into the multidimensional concept of macrophage ontogeny, activation and function.Uropathogenic Escherichia coli Engages CD14-Dependent Signaling to Enable Bladder-Macrophage-Dependent Control of Acute Urinary Tract Infection.Strain- and host species-specific inflammasome activation, IL-1β release, and cell death in macrophages infected with uropathogenic Escherichia coli.

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Crosstalk between sentinel and helper macrophages permits neutrophil migration into infected uroepithelium.

http://www.wikidata.org/entity/Q34643625

description

2014 nî lūn-bûn

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2014 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2014 թվականի հունվարին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年论文

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Crosstalk between sentinel and ...... n into infected uroepithelium.

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Crosstalk between sentinel and ...... n into infected uroepithelium.

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Crosstalk between sentinel and ...... n into infected uroepithelium.

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Crosstalk between sentinel and ...... n into infected uroepithelium.

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Crosstalk between sentinel and ...... n into infected uroepithelium.

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Crosstalk between sentinel and ...... n into infected uroepithelium.

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J.CELL.2014.01.006

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Akanksha Dixit

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Catherine Meyer-Schwesinger

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Christina Weisheit

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Marzena Schiwon

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Biochemistry and molecular biology of gelatinase B or matrix metalloproteinase-9 (MMP-9)

A toll-like receptor that prevents infection by uropathogenic bacteria

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Ghislain Opdenakker

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Martin Fuhrmann

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2014-01-01T00:00:00Z

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