Substance P signaling contributes to the vascular and nociceptive abnormalities observed in a tibial fracture rat model of complex regional pain syndrome type I.
about
The NALP1 inflammasome controls cytokine production and nociception in a rat fracture model of complex regional pain syndromeOrthopedic surgery and bone fracture pain are both significantly attenuated by sustained blockade of nerve growth factor.Regulation of peripheral blood flow in complex regional pain syndrome: clinical implication for symptomatic relief and pain management.Activation of cutaneous immune responses in complex regional pain syndrome.Brain neuroplastic changes accompany anxiety and memory deficits in a model of complex regional pain syndromeImmobilization contributes to exaggerated neuropeptide signaling, inflammatory changes, and nociceptive sensitization after fracture in rats.Autoimmunity contributes to nociceptive sensitization in a mouse model of complex regional pain syndrome.Neuropeptides contribute to peripheral nociceptive sensitization by regulating interleukin-1β production in keratinocytesEndoneurial pathology of the needlestick-nerve-injury model of Complex Regional Pain Syndrome, including rats with and without pain behaviorsSubstance P signaling controls mast cell activation, degranulation, and nociceptive sensitization in a rat fracture model of complex regional pain syndrome.Sex differences in a Murine Model of Complex Regional Pain Syndrome.Prolonged maintenance of capsaicin-induced hyperalgesia by brief daily vibration stimuli.Facilitated spinal neuropeptide signaling and upregulated inflammatory mediator expression contribute to postfracture nociceptive sensitizationSubstance P spinal signaling induces glial activation and nociceptive sensitization after fracture.Protective Effects of Alisma orientale Extract against Hepatic Steatosis via Inhibition of Endoplasmic Reticulum Stress.New Concepts in Complex Regional Pain Syndrome.Differential Efficacy of Ketamine in the Acute versus Chronic Stages of Complex Regional Pain Syndrome in MiceCaMKII Controls Whether Touch Is PainfulMast Cell-Mediated Mechanisms of Nociception.Acute versus chronic phase mechanisms in a rat model of CRPS.Neuropeptide deficient mice have attenuated nociceptive, vascular, and inflammatory changes in a tibia fracture model of complex regional pain syndrome.Post-traumatic complex regional pain syndrome: clinical features and epidemiology.Applied information retrieval and multidisciplinary research: new mechanistic hypotheses in complex regional pain syndromeChanges resembling complex regional pain syndrome following surgery and immobilization.TNF signaling contributes to the development of nociceptive sensitization in a tibia fracture model of complex regional pain syndrome type IEffect of anti-NGF antibodies in a rat tibia fracture model of complex regional pain syndrome type I.Epidermal adrenergic signaling contributes to inflammation and pain sensitization in a rat model of complex regional pain syndromeAcute and chronic phases of complex regional pain syndrome in mice are accompanied by distinct transcriptional changes in the spinal cord.Pentoxifylline attenuates nociceptive sensitization and cytokine expression in a tibia fracture rat model of complex regional pain syndrome.Post-junctional facilitation of Substance P signaling in a tibia fracture rat model of complex regional pain syndrome type I.The role of enhanced cutaneous IL-1beta signaling in a rat tibia fracture model of complex regional pain syndrome.Role of substance P signaling in enhanced nociceptive sensitization and local cytokine production after incision.Orthopedic surgery modulates neuropeptides and BDNF expression at the spinal and hippocampal levels.Replacement, refinement, and reduction: necessity of standardization and computational models for long bone fracture repair in animals.Preclinical Pain Research: Can We Do Better?The inflammasome as a target for pain therapy.Bisphosphonates Inhibit Pain, Bone Loss, and Inflammation in a Rat Tibia Fracture Model of Complex Regional Pain Syndrome.Effective dosage and administration schedule of oral alendronate for non-nociceptive symptoms in rats with chronic constriction injury.[EXPRESS] The Chemokine Receptor CXCR2 Supports Nociceptive Sensitization after Traumatic Brain Injury.Activated spinal astrocytes are involved in the maintenance of chronic widespread mechanical hyperalgesia after cast immobilization.
P2860
Q28568698-69634153-6DE7-4DB9-A632-691AFF525383Q30657180-7952869A-9A94-4251-9C5F-4CDA0D8C0C1DQ33506195-22B67191-54C2-4C76-A923-07845071B054Q33573783-71672B0E-DD3D-4361-AE27-013DE7CA7B59Q34244823-1095BAD5-70F2-4EC5-BDA8-35A3AB1D9B2EQ34276067-92CEB696-8268-4199-8022-960B213C575CQ34614091-EC2C2041-CB9D-428E-B2D8-7FA9363DB05EQ35068859-9ECD04B8-A70E-46FC-B474-8B974F36D8D4Q35223732-2FDC9B80-1881-4B96-9061-C8EEC1DC4011Q35850819-6FC5FEC6-31E7-4B18-BCC0-5B5F4DB14BD4Q35931545-898A3E16-BE8B-413F-B5AC-5888FACE73C1Q35950461-28581672-A9BF-446E-BEF8-94E00688C258Q36083373-83FD01BC-22D2-4000-8EAC-94CE393DA883Q36249124-2D6846B5-97DF-477E-ADD8-92469F510955Q36324486-4EB44B48-CF6E-479F-A208-593CAD938357Q36327294-483A40DC-0B77-48D1-9BEA-8973E092AB76Q36372728-107A4BB9-840E-4378-AD67-0572AF13CA65Q36383254-8CC01F57-D681-42D8-94B5-6FB82D418853Q36404141-447E0FA2-ED2C-4DA9-A598-59FAA5E0038FQ36480963-495518F2-AA5E-470A-B84B-603DB7424608Q36527054-DE97D634-519D-45C4-A77D-5D5ED05B0C50Q36800198-A1659E57-4661-4062-935E-3323BE3242DAQ36811556-33E000A9-0F0E-4DF4-BEAE-D1A66FD3471DQ36816589-89913E5A-727A-4B60-BA81-3B28E415F6D9Q36862101-394454EC-A77C-4F6E-AD31-79F26F131C71Q36881864-0E12F7C8-D10F-44C7-913F-A2AE93BF5C57Q37020591-F230EFFF-2ED3-4CC3-9B73-9A2BAE80C5E8Q37116137-5F0944D8-8FA7-4EEA-8C80-7BF6F48DC57AQ37169930-1831E62C-4745-40EE-B83C-39C8A5A65F3BQ37253305-E9C17475-2D80-4FE6-84B3-F6E70E90A96EQ37345168-98F2F79F-BA83-44A9-B5F1-DFC8BB4AF44DQ37351925-95305B47-B1C1-436E-A555-C6E5D0CBED2EQ37379656-32C1471C-0FEF-4033-84CC-AF09833007E0Q38129027-7137F8C9-6671-49B5-9E7F-025BED1F7A44Q38940989-2410A557-8988-4DF3-B6C4-2BC02125249CQ39036848-856AB751-E680-44C7-8FC0-E6D7BC0D53A2Q39381651-CAEBCF84-8416-463A-A372-8B962393032BQ40774771-B3440F61-FB26-45E2-84EE-2ABA4060BF82Q41664958-DC58AE98-05CF-42FE-B00A-C94B661DB7CDQ41901127-D290BC7A-8A8D-4E68-95B5-4402E9751B0C
P2860
Substance P signaling contributes to the vascular and nociceptive abnormalities observed in a tibial fracture rat model of complex regional pain syndrome type I.
description
2004 nî lūn-bûn
@nan
2004 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի մարտին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Substance P signaling contribu ...... regional pain syndrome type I.
@ast
Substance P signaling contribu ...... regional pain syndrome type I.
@en
Substance P signaling contribu ...... regional pain syndrome type I.
@nl
type
label
Substance P signaling contribu ...... regional pain syndrome type I.
@ast
Substance P signaling contribu ...... regional pain syndrome type I.
@en
Substance P signaling contribu ...... regional pain syndrome type I.
@nl
prefLabel
Substance P signaling contribu ...... regional pain syndrome type I.
@ast
Substance P signaling contribu ...... regional pain syndrome type I.
@en
Substance P signaling contribu ...... regional pain syndrome type I.
@nl
P2093
P1433
P1476
Substance P signaling contribu ...... regional pain syndrome type I.
@en
P2093
Christopher R Jacobs
Erin A Boyd
Sarah C Offley
Tian-Zhi Guo
Wade S Kingery
P304
P356
10.1016/J.PAIN.2003.12.010
P407
P577
2004-03-01T00:00:00Z