Value of serum nonceruloplasmin copper for prediction of mild cognitive impairment conversion to Alzheimer disease.
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Copper-2 Ingestion, Plus Increased Meat Eating Leading to Increased Copper Absorption, Are Major Factors Behind the Current Epidemic of Alzheimer's DiseaseDivalent Copper as a Major Triggering Agent in Alzheimer's DiseaseThe Case for Abandoning Therapeutic Chelation of Copper Ions in Alzheimer's DiseaseSerum calreticulin is a negative biomarker in patients with Alzheimer's disease.Airborne copper exposure in school environments associated with poorer motor performance and altered basal ganglia.Biological metals and metal-targeting compounds in major neurodegenerative diseases.Zinc in Alzheimer's Disease: A Meta-Analysis of Serum, Plasma, and Cerebrospinal Fluid Studies.Chitooligosaccharides attenuate Cu2+-induced cellular oxidative damage and cell apoptosis involving Nrf2 activation.Association Between Serum Ceruloplasmin Specific Activity and Risk of Alzheimer's Disease.The Influence of Cigarette Smoke Exposure on the Copper Concentration in the Serum Depending on the Use of Menopausal Hormone Therapy.Copper Exposure Perturbs Brain Inflammatory Responses and Impairs Clearance of Amyloid-Beta.Copper and Alzheimer's Disease.An exploratory study of BDNF and oxidative stress marker alterations in subacute and chronic stroke patients affected by neuropathic pain.The effect of copper from water and food: changes of serum nonceruloplasmin copper and brain's amyloid-beta in mice.Serum Non-Ceruloplasmin Non-Albumin Copper Elevation in Mild Cognitive Impairment and Dementia due to Alzheimer's Disease: A Case Control Study.Environmental and dietary exposure to copper and its cellular mechanisms linking to Alzheimer disease.Non-ceruloplasmin bound copper and ATP7B gene variants in Alzheimer's disease.Risk Factors Associated with Cortical Thickness and White Matter Hyperintensities in Dementia Free Okinawan Elderly.Tetrathiomolybdate Treatment Leads to the Suppression of Inflammatory Responses through the TRAF6/NFκB Pathway in LPS-Stimulated BV-2 Microglia.Non-Ceruloplasmin Copper Distincts Subtypes in Alzheimer's Disease: a Genetic Study of ATP7B Frequency.Spectrometric-based techniques for metal-binding protein assessment in clinical, environmental, and food samples
P2860
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P2860
Value of serum nonceruloplasmin copper for prediction of mild cognitive impairment conversion to Alzheimer disease.
description
2014 nî lūn-bûn
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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Value of serum nonceruloplasmi ...... nversion to Alzheimer disease.
@ast
Value of serum nonceruloplasmi ...... nversion to Alzheimer disease.
@en
Value of serum nonceruloplasmi ...... nversion to Alzheimer disease.
@nl
type
label
Value of serum nonceruloplasmi ...... nversion to Alzheimer disease.
@ast
Value of serum nonceruloplasmi ...... nversion to Alzheimer disease.
@en
Value of serum nonceruloplasmi ...... nversion to Alzheimer disease.
@nl
prefLabel
Value of serum nonceruloplasmi ...... nversion to Alzheimer disease.
@ast
Value of serum nonceruloplasmi ...... nversion to Alzheimer disease.
@en
Value of serum nonceruloplasmi ...... nversion to Alzheimer disease.
@nl
P2093
P2860
P50
P356
P1433
P1476
Value of serum nonceruloplasmi ...... onversion to Alzheimer disease
@en
P2093
Deborah Caprara
Fabrizio Vernieri
Mariacarla Ventriglia
Mariachiara Magri
Paolo M Rossini
P2860
P304
P356
10.1002/ANA.24136
P50
P577
2014-04-14T00:00:00Z