PEAK: a randomized, multicenter phase II study of panitumumab plus modified fluorouracil, leucovorin, and oxaliplatin (mFOLFOX6) or bevacizumab plus mFOLFOX6 in patients with previously untreated, unresectable, wild-type KRAS exon 2 metastatic color
about
Treatment of Metastatic Colorectal Cancer: Standard of Care and Future PerspectivesGenomic diversity of colorectal cancer: Changing landscape and emerging targetsMolecular Subtypes and Personalized Therapy in Metastatic Colorectal CancerAdvances of Targeted Therapy in Treatment of Unresectable Metastatic Colorectal CancerAdvances in targeted and immunobased therapies for colorectal cancer in the genomic eraProfile of panitumumab as first-line treatment in patients with wild-type KRAS metastatic colorectal cancerAdjuvant chemotherapy for resected colorectal cancer metastases: Literature review and meta-analysisTropomyosin-related kinase B/brain derived-neurotrophic factor signaling pathway as a potential therapeutic target for colorectal cancerA Comprehensive Review of Clinical Trials on EGFR Inhibitors Such as Cetuximab and Panitumumab as Monotherapy and in Combination for Treatment of Metastatic Colorectal CancerA Systematic Review and Network Meta-Analysis of Biologic Agents in the First Line Setting for Advanced Colorectal CancerPrecision medicine in colorectal cancer: the molecular profile alters treatment strategiesNew findings on primary and acquired resistance to anti-EGFR therapy in metastatic colorectal cancer: do all roads lead to RAS?Management of colorectal cancerTreatment of metastatic colorectal cancer: focus on panitumumabESMO - Magnitude of Clinical Benefit Scale V.1.0 questions and answersTherapeutic options for peritoneal metastasis arising from colorectal cancerRole of targeted therapy in metastatic colorectal cancerMolecular Diagnostics for Precision Medicine in Colorectal Cancer: Current Status and Future PerspectiveTherapeutic value of EGFR inhibition in CRC and NSCLC: 15 years of clinical evidenceMechanisms of resistance to anti-EGFR therapy in colorectal cancerRAS and BRAF in metastatic colorectal cancer managementMechanisms of resistance to anti-epidermal growth factor receptor inhibitors in metastatic colorectal cancerRole of Deficient Mismatch Repair in the Personalized Management of Colorectal CancerPanitumumab in Metastatic Colorectal Cancer: The Importance of Tumour RAS StatusIs conversion therapy possible in stage IV gastric cancer: the proposal of new biological categories of classificationExtended RAS testing in metastatic colorectal cancer-Refining the predictive molecular biomarkersTranslating genomic profiling to gastrointestinal cancer treatment.Targeted therapies in metastatic colorectal cancer: a systematic review and assessment of currently available dataRAS Mutations Beyond KRAS Exon 2: A Review and Discussion of Clinical Trial Data.A multi-institutional feasibility study of S-1/oxaliplatin plus bevacizumab in patients with advanced/metastatic colorectal cancer: the HiSCO-02 prospective phase II study.Recent Advances in Targeting the EGFR Signaling Pathway for the Treatment of Metastatic Colorectal CancerReply to S. Zhang, L. Fornaro et al, and H.J. Lee et alPrimary and acquired resistance to biologic therapies in gastrointestinal cancers.Metastatic colorectal cancer: ESMO Clinical Practice Guidelines for diagnosis, treatment and follow-up.Expanded RAS: refining the patient populationCorrelation of extended RAS and PIK3CA gene mutation status with outcomes from the phase III AGITG MAX STUDY involving capecitabine alone or in combination with bevacizumab plus or minus mitomycin C in advanced colorectal cancerJapanese Society of Medical Oncology Clinical Guidelines: RAS (KRAS/NRAS) mutation testing in colorectal cancer patientsA retrospective observational study of clinicopathological features of KRAS, NRAS, BRAF and PIK3CA mutations in Japanese patients with metastatic colorectal cancer.Consensus statement: the 16th Annual Western Canadian Gastrointestinal Cancer Consensus Conference; Saskatoon, Saskatchewan; September 5-6, 2014Extended RAS and BRAF Mutation Analysis Using Next-Generation Sequencing.
P2860
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P2860
PEAK: a randomized, multicenter phase II study of panitumumab plus modified fluorouracil, leucovorin, and oxaliplatin (mFOLFOX6) or bevacizumab plus mFOLFOX6 in patients with previously untreated, unresectable, wild-type KRAS exon 2 metastatic color
description
2014 nî lūn-bûn
@nan
2014 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մարտին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
PEAK: a randomized, multicente ...... e KRAS exon 2 metastatic color
@ast
PEAK: a randomized, multicente ...... e KRAS exon 2 metastatic color
@en
PEAK: a randomized, multicente ...... e KRAS exon 2 metastatic color
@nl
type
label
PEAK: a randomized, multicente ...... e KRAS exon 2 metastatic color
@ast
PEAK: a randomized, multicente ...... e KRAS exon 2 metastatic color
@en
PEAK: a randomized, multicente ...... e KRAS exon 2 metastatic color
@nl
prefLabel
PEAK: a randomized, multicente ...... e KRAS exon 2 metastatic color
@ast
PEAK: a randomized, multicente ...... e KRAS exon 2 metastatic color
@en
PEAK: a randomized, multicente ...... e KRAS exon 2 metastatic color
@nl
P2093
P921
P356
P1476
PEAK: a randomized, multicente ...... e KRAS exon 2 metastatic color
@en
P2093
Fernando Rivera
Gianpiero Fasola
J Randolph Hecht
Jean-Luc Canon
Kelly S Oliner
Lee S Schwartzberg
Meinolf Karthaus
William Y Go
P304
P356
10.1200/JCO.2013.53.2473
P407
P577
2014-03-31T00:00:00Z