Reactive oxygen species, mitochondria, and NAD(P)H oxidases in the development and progression of heart failure.
about
Histone deacetylase signaling in cardioprotectionNitric oxide and peroxynitrite in health and diseaseTherapeutic effects of xanthine oxidase inhibitors: renaissance half a century after the discovery of allopurinolThioredoxin and ventricular remodelingNovel Perspectives in Redox Biology and Pathophysiology of Failing Myocytes: Modulation of the Intramyocardial Redox Milieu for Therapeutic Interventions-A Review Article from the Working Group of Cardiac Cell Biology, Italian Society of CardiologyNutrient- and other stress-responsive microRNAs in plants: Role for thiol-based redox signalingThe efficacy of edaravone (radicut), a free radical scavenger, for cardiovascular diseaseIndomethacin potentiates acetylcholine-induced vasodilation by increasing free radical productionThe electrophile responsive proteome: integrating proteomics and lipidomics with cellular functionTherapeutic inhibition of mitochondrial reactive oxygen species with mito-TEMPO reduces diabetic cardiomyopathyEffects of shear stresses and antioxidant concentrations on the production of reactive oxygen species in lung cancer cellsRevisiting Kadenbach: Electron flux rate through cytochrome c-oxidase determines the ATP-inhibitory effect and subsequent production of ROSThe Role of Overweight and Obesity in the Cardiorenal Syndrome.Delivering regenerative cues to the heart: cardiac drug delivery by microspheres and peptide nanofibers.Cardiac CD47 drives left ventricular heart failure through Ca2+-CaMKII-regulated induction of HDAC3.Catestatin-A Novel Predictor of Left Ventricular Remodeling After Acute Myocardial InfarctionMonoamine oxidase A-mediated enhanced catabolism of norepinephrine contributes to adverse remodeling and pump failure in hearts with pressure overload.Effects of exercise and L-arginine on ventricular remodeling and oxidative stress.LOX-1 abrogation reduces cardiac hypertrophy and collagen accumulation following chronic ischemia in the mouseOxidant stress from nitric oxide synthase-3 uncoupling stimulates cardiac pathologic remodeling from chronic pressure load.Molecular Signals Elicited by GPCR Agonists in Hypertension, Cardiovascular Remodeling: Are MMPs and ADAMs Elusive Therapeutic Targets?Mitochondria and arrhythmias.{beta}1-Adrenergic receptor activation induces mouse cardiac myocyte death through both L-type calcium channel-dependent and -independent pathways.Redox activation of DUSP4 by N-acetylcysteine protects endothelial cells from Cd²⁺-induced apoptosis.Long-term obesity promotes alterations in diastolic function induced by reduction of phospholamban phosphorylation at serine-16 without affecting calcium handling.Pressure overload induces IL-18 and IL-18R expression, but markedly suppresses IL-18BP expression in a rabbit model. IL-18 potentiates TNF-α-induced cardiomyocyte deathMitochondrial division/mitophagy inhibitor (Mdivi) ameliorates pressure overload induced heart failureTargeting cellular energy production in neurological disorders.Inhibition of endogenous thioredoxin in the heart increases oxidative stress and cardiac hypertrophy.Human stanniocalcin-1 suppresses angiotensin II-induced superoxide generation in cardiomyocytes through UCP3-mediated anti-oxidant pathway.Modulation of signaling mechanisms in the heart by thioredoxin 1.Cardiac-specific overexpression of metallothionein rescues against cigarette smoking exposure-induced myocardial contractile and mitochondrial damageSympathetic hyperactivity differentially affects skeletal muscle mass in developing heart failure: role of exercise training.Cardiac-specific overexpression of metallothionein rescues nicotine-induced cardiac contractile dysfunction and interstitial fibrosisDo mitochondria limit hot fish hearts? Understanding the role of mitochondrial function with heat stress in Notolabrus celidotus.Myocardial repair/remodelling following infarction: roles of local factors.Celastrol protects ischaemic myocardium through a heat shock response with up-regulation of haeme oxygenase-1.Beyond blood pressure: the endothelium and atherosclerosis progression.Cardiac remodeling and physical training post myocardial infarctionCardiac steatosis potentiates angiotensin II effects in the heart.
P2860
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P2860
Reactive oxygen species, mitochondria, and NAD(P)H oxidases in the development and progression of heart failure.
description
2002 nî lūn-bûn
@nan
2002 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Reactive oxygen species, mitoc ...... progression of heart failure.
@ast
Reactive oxygen species, mitoc ...... progression of heart failure.
@en
Reactive oxygen species, mitoc ...... progression of heart failure.
@nl
type
label
Reactive oxygen species, mitoc ...... progression of heart failure.
@ast
Reactive oxygen species, mitoc ...... progression of heart failure.
@en
Reactive oxygen species, mitoc ...... progression of heart failure.
@nl
prefLabel
Reactive oxygen species, mitoc ...... progression of heart failure.
@ast
Reactive oxygen species, mitoc ...... progression of heart failure.
@en
Reactive oxygen species, mitoc ...... progression of heart failure.
@nl
P1476
Reactive oxygen species, mitoc ...... progression of heart failure.
@en
P2093
Dan Sorescu
Kathy K Griendling
P304
P356
10.1111/J.1527-5299.2002.00717.X
P577
2002-05-01T00:00:00Z