A novel phosphodiesterase type 4 inhibitor, HT-0712, enhances rehabilitation-dependent motor recovery and cortical reorganization after focal cortical ischemia. - Wikidata - awgldk - TriplyDB

A novel phosphodiesterase type 4 inhibitor, HT-0712, enhances rehabilitation-dependent motor recovery and cortical reorganization after focal cortical ischemia.

A novel phosphodiesterase type 4 inhibitor, HT-0712, enhances rehabilitation-dependent motor recovery and cortical reorganization after focal cortical ischemia.

http://www.wikidata.org/entity/Q34682160

about

Emergent properties of neural repair: elemental biology to therapeutic concepts Psychiatric aspects of phosphodiesterases: An overview.Translating the frontiers of brain repair to treatments: starting not to break the rules Phosphodiesterase Inhibitors as a Therapeutic Approach to Neuroprotection and Repair.Promoting axonal rewiring to improve outcome after stroke The PDE4 inhibitor HT-0712 improves hippocampus-dependent memory in aged mice.ABCD of the phosphodiesterase family: interaction and differential activity in COPD Neuron-targeted caveolin-1 protein enhances signaling and promotes arborization of primary neurons.Fatigue versus activity-dependent fatigability in patients with central or peripheral motor impairments.What do motor "recovery" and "compensation" mean in patients following stroke?Robotic devices as therapeutic and diagnostic tools for stroke recovery cAMP-specific phosphodiesterase inhibitors: promising drugs for inflammatory and neurological diseases.Dose of arm activity training during acute and subacute rehabilitation post stroke: a systematic review of the literature.Investigational phosphodiesterase inhibitors in phase I and phase II clinical trials for Alzheimer's disease.Consideration of Dose and Timing When Applying Interventions After Stroke and Spinal Cord Injury.Pathophysiology Associated with Traumatic Brain Injury: Current Treatments and Potential Novel Therapeutics.What is current practice for upper limb rehabilitation in the acute hospital setting following stroke? A systematic review.Perisynaptic GABA Receptors The Overzealous Protector.Simulations suggest pharmacological methods for rescuing long-term potentiation.Membrane lipid rafts and neurobiology: age-related changes in membrane lipids and loss of neuronal function.Prevention of cerebral ischemia-induced memory deficits by inhibition of phosphodiesterase-4 in rats.Targeting phosphodiesterase 4 as a potential therapeutic strategy for enhancing neuroplasticity following ischemic stroke

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P2860

A novel phosphodiesterase type 4 inhibitor, HT-0712, enhances rehabilitation-dependent motor recovery and cortical reorganization after focal cortical ischemia.

http://www.wikidata.org/entity/Q34682160

description

2007 nî lūn-bûn

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2007 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2007 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

A novel phosphodiesterase type ...... after focal cortical ischemia.

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A novel phosphodiesterase type ...... after focal cortical ischemia.

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A novel phosphodiesterase type ...... after focal cortical ischemia.

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type

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A novel phosphodiesterase type ...... after focal cortical ischemia.

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A novel phosphodiesterase type ...... after focal cortical ischemia.

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A novel phosphodiesterase type ...... after focal cortical ischemia.

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prefLabel

A novel phosphodiesterase type ...... after focal cortical ischemia.

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A novel phosphodiesterase type ...... after focal cortical ischemia.

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A novel phosphodiesterase type ...... after focal cortical ischemia.

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Neurorehabilitation and Neural Repair

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A novel phosphodiesterase type ...... after focal cortical ischemia.

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Christy Cole

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David Pocock

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Erin MacDonald

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Heidi Van der Lee

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Jeffrey A Kleim

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Nagheme Thomas

http://www.w3.org/2001/XMLSchema#string

Penny M VandenBerg

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Rusiko Bourtchouladze

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P2860

Rolipram, a type IV-specific phosphodiesterase inhibitor, facilitates the establishment of long-lasting long-term potentiation and improves memory

A mouse model of Rubinstein-Taybi syndrome: defective long-term memory is ameliorated by inhibitors of phosphodiesterase 4

Outcome measures in acute stroke trials: a systematic review and some recommendations to improve practice.

Cortical synaptogenesis and motor map reorganization occur during late, but not early, phase of motor skill learning.

Injection of the cAMP-responsive element into the nucleus of Aplysia sensory neurons blocks long-term facilitation.

Amyloid beta -peptide inhibition of the PKA/CREB pathway and long-term potentiation: reversibility by drugs that enhance cAMP signaling

Reorganization of remote cortical regions after ischemic brain injury: a potential substrate for stroke recovery.

Induction of a dominant negative CREB transgene specifically blocks long-term memory in Drosophila.

Neural substrates for the effects of rehabilitative training on motor recovery after ischemic infarct.

D1/D5 receptor agonists induce a protein synthesis-dependent late potentiation in the CA1 region of the hippocampus.

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6

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21

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