Emodin ameliorates LPS-induced acute lung injury, involving the inactivation of NF-κB in mice. - Wikidata - awgldk - TriplyDB

Emodin ameliorates LPS-induced acute lung injury, involving the inactivation of NF-κB in mice.

Emodin ameliorates LPS-induced acute lung injury, involving the inactivation of NF-κB in mice.

http://www.wikidata.org/entity/Q34684913

about

San-Huang-Xie-Xin-Tang Constituents Exert Drug-Drug Interaction of Mutual Reinforcement at Both Pharmacodynamics and Pharmacokinetic Level: A Review Silybin attenuates LPS-induced lung injury in mice by inhibiting NF-κB signaling and NLRP3 activation.INHIBITORY EFFECT OF EMODIN ON RAW 264.7 ACTIVATED WITH DOUBLE STRANDED RNA ANALOGUE POLY I:C Chemical Profiles and Protective Effect of Hedyotis diffusa Willd in Lipopolysaccharide-Induced Renal Inflammation Mice Glucagon Like Peptide-1 (GLP-1) Modulates OVA-Induced Airway Inflammation and Mucus Secretion Involving a Protein Kinase A (PKA)-Dependent Nuclear Factor-κB (NF-κB) Signaling Pathway in Mice.CXCL10/IP-10 Neutralization Can Ameliorate Lipopolysaccharide-Induced Acute Respiratory Distress Syndrome in Rats Antimicrobial Peptide CMA3 Derived from the CA-MA Hybrid Peptide: Antibacterial and Anti-inflammatory Activities with Low Cytotoxicity and Mechanism of Action in Escherichia coli Emodin Bidirectionally Modulates Macrophage Polarization and Epigenetically Regulates Macrophage Memory.Preventive Effects of the Intestine Function Recovery Decoction, a Traditional Chinese Medicine, on Postoperative Intra-Abdominal Adhesion Formation in a Rat Model Ugonin M, a Helminthostachys zeylanica Constituent, Prevents LPS-Induced Acute Lung Injury through TLR4-Mediated MAPK and NF-κB Signaling Pathways.Emodin and Its Role in Chronic Diseases.Tight junctions in pulmonary epithelia during lung inflammation.Scutellaria baicalensis Ameliorates Acute Lung Injury by Suppressing Inflammation In Vitro and In Vivo.Inhibition of nuclear factor-κB signal by pyrrolidine dithiocarbamate alleviates lipopolysaccharide-induced acute lung injury.Runt-Related Transcription Factor 1 Regulates LPS-Induced Acute Lung Injury via NF-κB Signaling.Depletion of circulating monocytes suppresses IL-17 and HMGB1 expression in mice with LPS-induced acute lung injury.Fisetin-treatment alleviates airway inflammation through inhbition of MyD88/NF-κB signaling pathway.Juglanin suppresses fibrosis and inflammation response caused by LPS in acute lung injury.Preventive Effects of Velvet Antler (Cervus elaphus) against Lipopolysaccharide-Induced Acute Lung Injury in Mice by Inhibiting MAPK/NF-κB Activation and Inducing AMPK/Nrf2 Pathways.GLP-1 Analogue Liraglutide Enhances SP-A Expression in LPS-Induced Acute Lung Injury through the TTF-1 Signaling Pathway.Emodin Attenuates Lipopolysaccharide-Induced Acute Liver Injury via Inhibiting the TLR4 Signaling Pathway and Ginsenoside Rh2 Ameliorates Lipopolysaccharide-Induced Acute Lung Injury by Regulating the TLR4/PI3K/Akt/mTOR, Raf-1/MEK/ERK, and Keap1/Nrf2/HO-1 Signaling Pathways in Mice

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P2860

Emodin ameliorates LPS-induced acute lung injury, involving the inactivation of NF-κB in mice.

http://www.wikidata.org/entity/Q34684913

description

2014 nî lūn-bûn

@nan

2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

@zh-hk

2014年論文

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2014年論文

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2014年论文

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name

Emodin ameliorates LPS-induced ...... inactivation of NF-κB in mice.

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Emodin ameliorates LPS-induced ...... inactivation of NF-κB in mice.

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Emodin ameliorates LPS-induced ...... inactivation of NF-κB in mice.

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type

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Emodin ameliorates LPS-induced ...... inactivation of NF-κB in mice.

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Emodin ameliorates LPS-induced ...... inactivation of NF-κB in mice.

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Emodin ameliorates LPS-induced ...... inactivation of NF-κB in mice.

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Emodin ameliorates LPS-induced ...... inactivation of NF-κB in mice.

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Emodin ameliorates LPS-induced ...... inactivation of NF-κB in mice.

@en

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Fu-Qiang Wen

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Min Xiao

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Qiong-Fang Wan

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Tao Wang

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Tao Zhu

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Wei Zhang

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Yong-Chun Shen

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Pharmacological treatments in ARDS; a state-of-the-art update

Andrographolide protects against LPS-induced acute lung injury by inactivation of NF-κB

Deregulation of inflammatory response in the diabetic condition is associated with increased ischemic brain injury.

Effect of siRNA against NF-κB on sepsis‑induced acute lung injury in a mouse model.

The Berlin definition of ARDS: an expanded rationale, justification, and supplementary material.

Identification of plumericin as a potent new inhibitor of the NF-κB pathway with anti-inflammatory activity in vitro and in vivo

Novel interventional approaches for ALI/ARDS: cell-based gene therapy

Year in review in Intensive Care Medicine 2011: III. ARDS and ECMO, weaning, mechanical ventilation, noninvasive ventilation, pediatrics and miscellanea

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10.3390/IJMS151119355

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11

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