Cooperativity within and among Pten, p53, and Rb pathways induces high-grade astrocytoma in adult brain. - Wikidata - awgldk - TriplyDB

Cooperativity within and among Pten, p53, and Rb pathways induces high-grade astrocytoma in adult brain.

Cooperativity within and among Pten, p53, and Rb pathways induces high-grade astrocytoma in adult brain.

http://www.wikidata.org/entity/Q34697339

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The oncoprotein HBXIP uses two pathways to up-regulate S100A4 in promotion of growth and migration of breast cancer cells An extensive microRNA-mediated network of RNA-RNA interactions regulates established oncogenic pathways in glioblastoma In vivo models of brain tumors: roles of genetically engineered mouse models in understanding tumor biology and use in preclinical studies Mouse models to interrogate the implications of the differentiation status in the ontogeny of gliomas The role of IDO in brain tumor immunotherapy Will kinase inhibitors make it as glioblastoma drugs?Systems cancer medicine: towards realization of predictive, preventive, personalized and participatory (P4) medicine Contemporary murine models in preclinical astrocytoma drug development Precision targeted therapy of ovarian cancer Cell of origin for malignant gliomas and its implication in therapeutic development Contribution of classical end-joining to PTEN inactivation in p53-mediated glioblastoma formation and drug-resistant survival.Identification of mutated core cancer modules by integrating somatic mutation, copy number variation, and gene expression data.Growth arrest and forced differentiation of human primary glioblastoma multiforme by a novel small molecule.Glioblastoma models reveal the connection between adult glial progenitors and the proneural phenotype The probable cell of origin of NF1- and PDGF-driven glioblastomas.Discovery of co-occurring driver pathways in cancer JAK2/STAT3 targeted therapy suppresses tumor invasion via disruption of the EGFRvIII/JAK2/STAT3 axis and associated focal adhesion in EGFRvIII-expressing glioblastoma.Determining PTEN functional status by network component deduced transcription factor activities The cellular origin for malignant glioma and prospects for clinical advancements.Orthotopic models of pediatric brain tumors in zebrafish Sleeping Beauty mouse models identify candidate genes involved in gliomagenesis.ASYMMETRIC CELL DIVISION: IMPLICATIONS FOR GLIOMA DEVELOPMENT AND TREATMENT.Glial progenitors as targets for transformation in glioma.Transcriptional analysis of aggressiveness and heterogeneity across grades of astrocytomas.Inhibition of oxidative metabolism leads to p53 genetic inactivation and transformation in neural stem cells.ICan: an integrated co-alteration network to identify ovarian cancer-related genes A computational model incorporating neural stem cell dynamics reproduces glioma incidence across the lifespan in the human population Qualitative network modeling of the Myc-p53 control system of cell proliferation and differentiation Clinical stratification of glioblastoma based on alterations in retinoblastoma tumor suppressor protein (RB1) and association with the proneural subtype Advances toward regenerative medicine in the central nervous system: challenges in making stem cell therapy a viable clinical strategy.Cross-Species Genomics Identifies TAF12, NFYC, and RAD54L as Choroid Plexus Carcinoma Oncogenes.Expression Analysis of Genes Involved in the RB/E2F Pathway in Astrocytic Tumors.Capturing the molecular and biological diversity of high-grade astrocytoma in genetically engineered mouse models Review: insights gained from modelling high-grade glioma in the mouse.Emerging insights into the molecular and cellular basis of glioblastoma.Differential localization of glioblastoma subtype: implications on glioblastoma pathogenesis.Antiangiogenic therapy for glioma.Preclinical pharmacological evaluation of letrozole as a novel treatment for gliomas.Rapid tumor induction in zebrafish by TALEN-mediated somatic inactivation of the retinoblastoma1 tumor suppressor rb1 A novel technique of serial biopsy in mouse brain tumour models

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Cooperativity within and among Pten, p53, and Rb pathways induces high-grade astrocytoma in adult brain.

http://www.wikidata.org/entity/Q34697339

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2011 nî lūn-bûn

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J.CCR.2011.01.039

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Alberto Broniscer

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Chunxu Qu

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David W Ellison

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Junyuan Zhang

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Lionel M L Chow

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Raelene Endersby

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Sherri Rankin

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Suzanne J Baker

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Xiaoyan Zhu

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P2860

An integrated genomic analysis of human glioblastoma multiforme

IDH1 and IDH2 mutations in gliomas

Integrated genomic analysis identifies clinically relevant subtypes of glioblastoma characterized by abnormalities in PDGFRA, IDH1, EGFR, and NF1

Comprehensive genomic characterization defines human glioblastoma genes and core pathways

Expression of mutant p53 proteins implicates a lineage relationship between neural stem cells and malignant astrocytic glioma in a murine model

The 2007 WHO classification of tumours of the central nervous system

PTEN is essential for cell migration but not for fate determination and tumourigenesis in the cerebellum

Malignant gliomas in adults

Malignant astrocytic glioma: genetics, biology, and paths to treatment

Molecular subclasses of high-grade glioma predict prognosis, delineate a pattern of disease progression, and resemble stages in neurogenesis

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305-316

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10.1016/J.CCR.2011.01.039

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