IL-1β production through the NLRP3 inflammasome by hepatic macrophages links hepatitis C virus infection with liver inflammation and disease - Wikidata - awgldk - TriplyDB

IL-1β production through the NLRP3 inflammasome by hepatic macrophages links hepatitis C virus infection with liver inflammation and disease

IL-1β production through the NLRP3 inflammasome by hepatic macrophages links hepatitis C virus infection with liver inflammation and disease

http://www.wikidata.org/entity/Q34697945

about

Inflammatory status in human hepatic cirrhosis Interleukin-1 Family Cytokines in Liver Diseases Regulation of the Host Antiviral State by Intercellular Communications Regulation of hepatic innate immunity by hepatitis C virus Immune responses to HCV and other hepatitis viruses Interplay between Inflammation and Cellular Stress Triggered by Flaviviridae Viruses Emerging concepts in immunity to hepatitis C virus infection.Ubiquitin in Influenza Virus Entry and Innate Immunity Innate and Adaptive Immune Regulation During Chronic Viral Infections Inflammasome control of viral infection HCV RNA Activates APCs via TLR7/TLR8 While Virus Selectively Stimulates Macrophages Without Inducing Antiviral Responses Reactive oxygen species-induced TXNIP drives fructose-mediated hepatic inflammation and lipid accumulation through NLRP3 inflammasome activation Defective mitophagy driven by dysregulation of rheb and KIF5B contributes to mitochondrial reactive oxygen species (ROS)-induced nod-like receptor 3 (NLRP3) dependent proinflammatory response and aggravates lipotoxicity Upregulation of interleukin-1β/transforming growth factor-β1 and hypoxia relate to molecular mechanisms underlying immobilization-induced muscle contracture Immunoregulatory Role of NK Cells in Tissue Inflammation and Regeneration.The innate immunity of guinea pigs against highly pathogenic avian influenza virus infection Rapid inflammasome activation in microglia contributes to brain disease in HIV/AIDS Cellular visualization of macrophage pyroptosis and interleukin-1β release in a viral hemorrhagic infection in zebrafish larvae HIV and HCV activate the inflammasome in monocytes and macrophages via endosomal Toll-like receptors without induction of type 1 interferon.Depression and Chronic Liver Diseases: Are There Shared Underlying Mechanisms?Regulated Entry of Hepatitis C Virus into Hepatocytes.Changes in inflammatory biomarkers in HCV-infected patients undergoing direct acting antiviral-containing regimens with or without interferon Oxidative stress and hepatic Nox proteins in chronic hepatitis C and hepatocellular carcinoma.Molecular mechanisms of liver fibrosis in HIV/HCV coinfection HIV-1 infection induces interleukin-1β production via TLR8 protein-dependent and NLRP3 inflammasome mechanisms in human monocytes Prolactin prevents hepatocellular carcinoma by restricting innate immune activation of c-Myc in mice.Hepatitis C virus core protein enhances HIV-1 replication in human macrophages through TLR2, JNK, and MEK1/2-dependent upregulation of TNF-α and IL-6.Innate immune cell networking in hepatitis C virus infection Natural Killer Cells in Viral Hepatitis.Macrophages in Tissue Repair, Regeneration, and Fibrosis.A balanced IL-1β activity is required for host response to Citrobacter rodentium infection.RNA sequencing of sessile serrated colon polyps identifies differentially expressed genes and immunohistochemical markers.Transmitted/founder hepatitis C viruses induce cell-type- and genotype-specific differences in innate signaling within the liver.Hepatic inflammation and fibrosis: functional links and key pathways.Impact of hepatitis C seropositivity on the risk of coronary heart disease events.Hepatitis C virus and antiviral innate immunity: who wins at tug-of-war?Hepatitis C Virus. Strategies to Evade Antiviral Responses Activation and evasion of antiviral innate immunity by hepatitis C virus.Monocytes activate natural killer cells via inflammasome-induced interleukin 18 in response to hepatitis C virus replication.Interleukin 1β-Responsive MicroRNA-146a Is Critical for the Cytokine-Induced Tolerance and Cross-Tolerance to Toll-Like Receptor Ligands

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IL-1β production through the NLRP3 inflammasome by hepatic macrophages links hepatitis C virus infection with liver inflammation and disease

http://www.wikidata.org/entity/Q34697945

description

2013 nî lūn-bûn

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2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2013 թվականի ապրիլին հրատարակված գիտական հոդված

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年學術文章

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IL-1β production through the N ...... liver inflammation and disease

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IL-1β production through the N ...... liver inflammation and disease

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IL-1β production through the N ...... liver inflammation and disease

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Amina A Negash

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Brian Doehle

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Curt H Hagedorn

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Daryl T Y Lau

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Don A Delker

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Hilario J Ramos

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Juandy Jo

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Nanette Crochet

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Neven Papic

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P2860

The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

Viral and therapeutic control of IFN-beta promoter stimulator 1 during hepatitis C virus infection

Regulating intracellular antiviral defense and permissiveness to hepatitis C virus RNA replication through a cellular RNA helicase, RIG-I

Cytosolic DNA triggers inflammasome activation in keratinocytes in psoriatic lesions

Innate immunity induced by composition-dependent RIG-I recognition of hepatitis C virus RNA

IL-1β signaling promotes CNS-intrinsic immune control of West Nile virus infection

Highly permissive cell lines for subgenomic and genomic hepatitis C virus RNA replication.

Activation of anti-hepatitis C virus responses via Toll-like receptor 7.

Cell culture-produced hepatitis C virus does not infect peripheral blood mononuclear cells

Apolipoprotein E on hepatitis C virion facilitates infection through interaction with low-density lipoprotein receptor

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10.1371/JOURNAL.PPAT.1003330

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4

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Antonio Bertoletti

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2013-04-25T00:00:00Z

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236600567

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23633957

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Hepatitis C virus

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3635973

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