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Epigenetic mechanisms in schizophreniaDNA Damage and Repair in Schizophrenia and Autism: Implications for Cancer Comorbidity and BeyondTherapeutic Targets for Neurodevelopmental Disorders Emerging from Animal Models with Perinatal Immune ActivationEpidemiologic studies of exposure to prenatal infection and risk of schizophrenia and autismImmunologic and neurodevelopmental susceptibilities of autismH3N2 influenza A virus replicates in immortalized human first trimester trophoblast cell lines and induces their rapid apoptosis.Prenatal exposure to a viral mimetic alters behavioural flexibility in male, but not female, rats.Mouse models of gene-environment interactions in schizophrenia.Maternal immune activation causes age- and region-specific changes in brain cytokines in offspring throughout development.Viral infection, inflammation and schizophreniaModeling an autism risk factor in mice leads to permanent immune dysregulation.Prenatal interaction of mutant DISC1 and immune activation produces adult psychopathology.Pallidal hyperdopaminergic innervation underlying D2 receptor-dependent behavioral deficits in the schizophrenia animal model established by EGFEvaluating early preventive antipsychotic and antidepressant drug treatment in an infection-based neurodevelopmental mouse model of schizophrenia.Combined effect of neonatal immune activation and mutant DISC1 on phenotypic changes in adulthood.Animal models of gene-environment interactions in schizophrenia.Induction of Toll-like receptor 3-mediated immunity during gestation inhibits cortical neurogenesis and causes behavioral disturbancesInteraction between environmental and genetic factors modulates schizophrenic endophenotypes in the Snap-25 mouse mutant blind-drunk.Neuroimmunological aberrations and cerebral asymmetry abnormalities in schizophrenia: select perspectives on pathogenesisBCG vaccine-induced neuroprotection in a mouse model of Parkinson's disease.Maternal immune activation alters nonspatial information processing in the hippocampus of the adult offspring.Identifying novel interventional strategies for psychiatric disorders: integrating genomics, 'enviromics' and gene-environment interactions in valid preclinical models.Synaptotagmin I regulates patterned spontaneous activity in the developing rat retina via calcium binding to the C2AB domains.Toll-like receptors at the maternal-fetal interface in normal pregnancy and pregnancy disorders.Late prenatal immune activation in mice leads to behavioral and neurochemical abnormalities relevant to the negative symptoms of schizophrenia.The α7 nicotinic acetylcholine receptor and the acute stress response: maternal genotype determines offspring phenotype.Deficient maternal care resulting from immunological stress during pregnancy is associated with a sex-dependent enhancement of conditioned fear in the offspringRisperidone administered during asymptomatic period of adolescence prevents the emergence of brain structural pathology and behavioral abnormalities in an animal model of schizophrenia.Diminished cerebral inhibition in neonates associated with risk factors for schizophrenia: parental psychosis, maternal depression, and nicotine use.Neural systems governed by nicotinic acetylcholine receptors: emerging hypotheses.Prenatal maternal immune disruption and sex-dependent risk for psychoses.Mapping Alterations to the Endogenous Elemental Distribution within the Lateral Ventricles and Choroid Plexus in Brain Disorders Using X-Ray Fluorescence Imaging.Major histocompatibility complex class I proteins in brain development and plasticity.Does Systemic Inflammation Play a Role in Pediatric Psychosis?A review of the fetal brain cytokine imbalance hypothesis of schizophrenia.Expression and function of toll-like receptors at the maternal-fetal interface.Neonatal systemic exposure to lipopolysaccharide enhances susceptibility of nigrostriatal dopaminergic neurons to rotenone neurotoxicity in later life.Decanalization mediating gene-environment interactions in schizophrenia and other psychiatric disorders with neurodevelopmental etiology.Potential microbial origins of schizophrenia and their treatments.Abnormal expression of myelination genes and alterations in white matter fractional anisotropy following prenatal viral influenza infection at E16 in mice.
P2860
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P2860
description
2007 nî lūn-bûn
@nan
2007 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Neuroscience. Maternal effects on schizophrenia risk.
@ast
Neuroscience. Maternal effects on schizophrenia risk.
@en
Neuroscience. Maternal effects on schizophrenia risk.
@nl
type
label
Neuroscience. Maternal effects on schizophrenia risk.
@ast
Neuroscience. Maternal effects on schizophrenia risk.
@en
Neuroscience. Maternal effects on schizophrenia risk.
@nl
prefLabel
Neuroscience. Maternal effects on schizophrenia risk.
@ast
Neuroscience. Maternal effects on schizophrenia risk.
@en
Neuroscience. Maternal effects on schizophrenia risk.
@nl
P356
P1433
P1476
Neuroscience. Maternal effects on schizophrenia risk.
@en
P2093
Paul H Patterson
P304
P356
10.1126/SCIENCE.1150196
P407
P577
2007-10-01T00:00:00Z