The EBV Latent Antigen 3C Inhibits Apoptosis through Targeted Regulation of Interferon Regulatory Factors 4 and 8.
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The Modulation of Apoptotic Pathways by GammaherpesvirusesRole of EBNA-3 Family Proteins in EBV Associated B-cell LymphomagenesisThe Role of Gammaherpesviruses in Cancer PathogenesisEpstein-Barr Virus: Diseases Linked to Infection and TransformationEpstein-Barr virus latent genesThe EBNA3 family of Epstein-Barr virus nuclear proteins associates with the USP46/USP12 deubiquitination complexes to regulate lymphoblastoid cell line growthDownregulation of IRF4 induces lytic reactivation of KSHV in primary effusion lymphoma cellsEpstein-Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes.Modulation of enhancer looping and differential gene targeting by Epstein-Barr virus transcription factors directs cellular reprogramming.EBNA3C augments Pim-1 mediated phosphorylation and degradation of p21 to promote B-cell proliferationLMP1-deficient Epstein-Barr virus mutant requires T cells for lymphomagenesis.EBV Nuclear Antigen 3C Mediates Regulation of E2F6 to Inhibit E2F1 Transcription and Promote Cell Proliferation.Epstein-Barr Virus Nuclear Antigen 3 (EBNA3) Proteins Regulate EBNA2 Binding to Distinct RBPJ Genomic SitesEpstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A.Recent progress in omics-driven analysis of MS to unravel pathological mechanisms.Dissecting the contribution of EBNA3C domains important for EBV-induced B-cell growth and proliferation.IRF4 and IRF8: Governing the virtues of B Lymphocytes.Epstein-Barr virus infection induces aberrant TLR activation pathway and fibroblast-myofibroblast conversion in sclerodermaEpstein-Barr virus latency: current and future perspectives.An essential EBV latent antigen 3C binds Bcl6 for targeted degradation and cell proliferation.Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expressionLMP1 signaling pathway activates IRF4 in latent EBV infection and a positive circuit between PI3K and Src is required.An EBV recombinant deleted for residues 130-159 in EBNA3C can deregulate p53/Mdm2 and Cyclin D1/CDK6 which results in apoptosis and reduced cell proliferation.Epstein-Barr virus essential antigen EBNA3C attenuates H2AX expression.EBNA3C regulates p53 through induction of Aurora kinase B.Interferon regulatory factor 4 is activated through c-Src-mediated tyrosine phosphorylation in virus-transformed cells.Epstein-Barr Virus LMP1 Mediated Oncogenicity.Epstein-Barr Virus Rta-Mediated Accumulation of DNA Methylation Interferes with CTCF Binding in both Host and Viral Genomes.Latent Membrane Protein 1 (LMP1) and LMP2A Collaborate To Promote Epstein-Barr Virus-Induced B Cell Lymphomas in a Cord Blood-Humanized Mouse Model but Are Not Essential.A study of the mutational landscape of pediatric-type follicular lymphoma and pediatric nodal marginal zone lymphoma.The Epstein-Barr Virus Regulome in Lymphoblastoid Cells.CRISPR-Cas9 Genetic Analysis of Virus-Host Interactions.Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV.Interferon regulatory factor 8 regulates caspase-1 expression to facilitate Epstein-Barr virus reactivation in response to B cell receptor stimulation and chemical induction.Current Progress in EBV-Associated B-Cell Lymphomas.Upregulation of Interferon Regulatory Factor 6 Promotes Neuronal Apoptosis After Traumatic Brain Injury in Adult Rats.Transcriptional and epigenetic modulation of autophagy promotes EBV oncoprotein EBNA3C induced B-cell survival.Counteracting survival functions of EBNA3C in Epstein-Barr virus (EBV)-driven lymphoproliferative diseases by combination of SAHA and bortezomib.An EBNA3C-deleted Epstein-Barr virus (EBV) mutant causes B-cell lymphomas with delayed onset in a cord blood-humanized mouse model
P2860
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P2860
The EBV Latent Antigen 3C Inhibits Apoptosis through Targeted Regulation of Interferon Regulatory Factors 4 and 8.
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2013 nî lūn-bûn
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2013 թուականի Մայիսին հրատարակուած գիտական յօդուած
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2013 թվականի մայիսին հրատարակված գիտական հոդված
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2013年の論文
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2013年論文
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2013年論文
@zh-hant
2013年論文
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2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
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name
The EBV Latent Antigen 3C Inhi ...... on Regulatory Factors 4 and 8.
@ast
The EBV Latent Antigen 3C Inhi ...... on Regulatory Factors 4 and 8.
@en
The EBV Latent Antigen 3C Inhi ...... on Regulatory Factors 4 and 8.
@nl
type
label
The EBV Latent Antigen 3C Inhi ...... on Regulatory Factors 4 and 8.
@ast
The EBV Latent Antigen 3C Inhi ...... on Regulatory Factors 4 and 8.
@en
The EBV Latent Antigen 3C Inhi ...... on Regulatory Factors 4 and 8.
@nl
prefLabel
The EBV Latent Antigen 3C Inhi ...... on Regulatory Factors 4 and 8.
@ast
The EBV Latent Antigen 3C Inhi ...... on Regulatory Factors 4 and 8.
@en
The EBV Latent Antigen 3C Inhi ...... on Regulatory Factors 4 and 8.
@nl
P2093
P2860
P1433
P1476
The EBV Latent Antigen 3C Inhi ...... on Regulatory Factors 4 and 8.
@en
P2093
Abhik Saha
Hem Chandra Jha
Qiliang Cai
Richard Kuo Dzeng
Shuvomoy Banerjee
P2860
P304
P356
10.1371/JOURNAL.PPAT.1003314
P577
2013-05-02T00:00:00Z