SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants - Wikidata - awgldk - TriplyDB

SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants

SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants

http://www.wikidata.org/entity/Q34738296

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Phenotypic and Genotypic Characteristics of Small Colony Variants and Their Role in Chronic Infection The SaeRS Two-Component System of Staphylococcus aureus Sigma Factor SigB Is Crucial to Mediate Staphylococcus aureus Adaptation during Chronic Infections The Staphylococcal Biofilm: Adhesins, Regulation, and Host Response Staphylococcus aureus Small Colony Variants (SCVs): a road map for the metabolic pathways involved in persistent infections The Agr quorum-sensing system regulates fibronectin binding but not hemolysis in the absence of a functional electron transport chain.Prolonged growth of a clinical Staphylococcus aureus strain selects for a stable small-colony-variant cell type.Staphylococcus aureus Transcriptome Architecture: From Laboratory to Infection-Mimicking Conditions.Characterization of a vraG Mutant in a Genetically Stable Staphylococcus aureus Small-Colony Variant and Preliminary Assessment for Use as a Live-Attenuated Vaccine against Intrammamary Infections.Clinical Significance and Pathogenesis of Staphylococcal Small Colony Variants in Persistent Infections.Staphylococcus aureus adapts to oxidative stress by producing H2O2-resistant small-colony variants via the SOS response.Staphylococcus aureus dynamically adapts global regulators and virulence factor expression in the course from acute to chronic infection.Vascular graft infection: a new model for treatment management?Resilience in the Face of Uncertainty: Sigma Factor B Fine-Tunes Gene Expression To Support Homeostasis in Gram-Positive Bacteria Staphylococcus aureus-Associated Musculoskeletal Infections.The role of biofilms in persistent infections and factors involved in ica-independent biofilm development and gene regulation in Staphylococcus aureus.Impact of Staphylococcus aureus regulatory mutations that modulate biofilm formation in the USA300 strain LAC on virulence in a murine bacteremia model.Cell differentiation defines acute and chronic infection cell types in Staphylococcus aureus.Staphylococcus aureus Regulator Sigma B is Important to Develop Chronic Infections in Hematogenous Murine Osteomyelitis Model.Novel Pathways for Ameliorating the Fitness Cost of Gentamicin Resistant Small Colony Variants.The Electron Transport Chain Sensitizes Staphylococcus aureus and Enterococcus faecalis to the Oxidative Burst.Bicarbonate induces high-level resistance to the human antimicrobial peptide LL-37 in Staphylococcus aureus small colony variants.A novel SigB(Q225P) mutation in Staphylococcus aureus retains virulence but promotes biofilm formation.

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P2860

SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants

http://www.wikidata.org/entity/Q34738296

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2013 nî lūn-bûn

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2013 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2013 թվականի մայիսին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants

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SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants

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SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants

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type

label

SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants

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SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants

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SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants

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prefLabel

SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants

@ast

SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants

@en

SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants

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JOURNAL.PONE.0065018

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SigB is a dominant regulator of virulence in Staphylococcus aureus small-colony variants

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Alexandre Fugère

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André M Cantin

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Eric Brouillette

http://www.w3.org/2001/XMLSchema#string

Gabriel Mitchell

http://www.w3.org/2001/XMLSchema#string

Karine Pépin Gaudreau

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P2860

RNAIII-independent target gene control by the agr quorum-sensing system: insight into the evolution of virulence regulation in Staphylococcus aureus

Staphylococcus aureus biofilms: properties, regulation, and roles in human disease

Bacterial biofilms: a common cause of persistent infections

Modulation of eDNA release and degradation affects Staphylococcus aureus biofilm maturation

Interconnections between Sigma B, agr, and proteolytic activity in Staphylococcus aureus biofilm maturation

Autoinduction and signal transduction in the regulation of staphylococcal virulence

A potential new pathway for Staphylococcus aureus dissemination: the silent survival of S. aureus phagocytosed by human monocyte-derived macrophages.

Staphylococcus aureus sigma B-dependent emergence of small-colony variants and biofilm production following exposure to Pseudomonas aeruginosa 4-hydroxy-2-heptylquinoline-N-oxide.

agr expression precedes escape of internalized Staphylococcus aureus from the host endosome.

Waves of resistance: Staphylococcus aureus in the antibiotic era.

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e65018

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scholarly article

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10.1371/JOURNAL.PONE.0065018

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5

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8

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Francois Malouin

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2013-05-21T00:00:00Z

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236909664

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23705029

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3660380

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