Interleukin-17 contributes to generation of Th1 immunity and neutrophil recruitment during Chlamydia muridarum genital tract infection but is not required for macrophage influx or normal resolution of infection - Wikidata - awgldk - TriplyDB

Interleukin-17 contributes to generation of Th1 immunity and neutrophil recruitment during Chlamydia muridarum genital tract infection but is not required for macrophage influx or normal resolution of infection

Interleukin-17 contributes to generation of Th1 immunity and neutrophil recruitment during Chlamydia muridarum genital tract infection but is not required for macrophage influx or normal resolution of infection

http://www.wikidata.org/entity/Q34739767

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Evaluation of the sublingual route for administration of influenza H5N1 virosomes in combination with the bacterial second messenger c-di-GMP Mucosal resident memory CD4 T cells in protection and immunopathology Cytokines in the host response to Candida vaginitis: Identifying a role for non-classical immune mediators, S100 alarmins Steroids alone or as adjunctive therapy with doxycycline fail to improve oviduct damage in mice infected with Chlamydia muridarum.Chlamydial infection in vitamin D receptor knockout mice is more intense and prolonged than in wild-type mice.Interruption of CXCL13-CXCR5 axis increases upper genital tract pathology and activation of NKT cells following chlamydial genital infection.Anti-chlamydial Th17 responses are controlled by the inducible costimulator partially through phosphoinositide 3-kinase signaling Innate and adaptive immunity at mucosal surfaces of the female reproductive tract: stratification and integration of immune protection against the transmission of sexually transmitted infections The duration of Chlamydia muridarum genital tract infection and associated chronic pathological changes are reduced in IL-17 knockout mice but protection is not increased further by immunization.Protective immunity against Chlamydia trachomatis can engage both CD4+ and CD8+ T cells and bridge the respiratory and genital mucosae Enhanced neutrophil longevity and recruitment contribute to the severity of oviduct pathology during Chlamydia muridarum infection.Interferon regulatory transcription factor 3 protects mice from uterine horn pathology during Chlamydia muridarum genital infection Protective immunity against mouse upper genital tract pathology correlates with high IFNγ but low IL-17 T cell and anti-secretion protein antibody responses induced by replicating chlamydial organisms in the airway.Plac8-dependent and inducible NO synthase-dependent mechanisms clear Chlamydia muridarum infections from the genital tract Frequency of Chlamydia trachomatis-specific T cell interferon-γ and interleukin-17 responses in CD4-enriched peripheral blood mononuclear cells of sexually active adolescent females.Hematopoetic prostaglandin D synthase: an ESR1-dependent oviductal epithelial cell synthase.IL-23 dampens the allergic response to Cryptococcus neoformans through IL-17-independent and -dependent mechanisms.Antibody and Cytokine Responses of Koalas (Phascolarctos cinereus) Vaccinated with Recombinant Chlamydial Major Outer Membrane Protein (MOMP) with Two Different Adjuvants Comparable Genital Tract Infection, Pathology, and Immunity in Rhesus Macaques Inoculated with Wild-Type or Plasmid-Deficient Chlamydia trachomatis Serovar D Modeling the transcriptome of genital tract epithelial cells and macrophages in healthy mucosa versus mucosa inflamed by Chlamydia muridarum infection.Th1 and Th17 cells regulate innate immune responses and bacterial clearance during central nervous system infection Contribution of interleukin-12 p35 (IL-12p35) and IL-12p40 to protective immunity and pathology in mice infected with Chlamydia muridarum.Immunity to infection in IL-17-deficient mice and humans Immunity, immunopathology, and human vaccine development against sexually transmitted Chlamydia trachomatis CD4+ T cell expression of MyD88 is essential for normal resolution of Chlamydia muridarum genital tract infection.IFNγ is Required for Optimal Antibody-Mediated Immunity against Genital Chlamydia Infection.IL-23 induces IL-22 and IL-17 production in response to Chlamydia muridarum genital tract infection, but the absence of these cytokines does not influence disease pathogenesis.CCR7 Deficiency Allows Accelerated Clearance of Chlamydia from the Female Reproductive Tract.Neutrophils are Central to Antibody-Mediated Protection against Genital Chlamydia Sensing the enemy, containing the threat: cell-autonomous immunity to Chlamydia trachomatis.Immunopathogenesis of Chlamydial Infections.Murine MicroRNA-214 regulates intracellular adhesion molecule (ICAM1) gene expression in genital Chlamydia muridarum infection.High Chlamydia Burden Promotes Tumor Necrosis Factor-Dependent Reactive Arthritis in SKG Mice.Immunity against a Chlamydia infection and disease may be determined by a balance of IL-17 signaling.Progesterone activates multiple innate immune pathways in Chlamydia trachomatis-infected endocervical cells.Th17 cells confer long-term adaptive immunity to oral mucosal Candida albicans infections.A novel role for IL-17 in enhancing type 1 helper T cell immunity in the female genital tract following mucosal HSV-2 vaccination.Development of a contemporary animal model of Candida albicans-associated denture stomatitis using a novel intraoral denture system.Transcervical inoculation with Chlamydia trachomatis induces infertility in HLA-DR4-transgenic and wild type mice.Mucocutaneous IL-17 immunity in mice and humans: host defense vs. excessive inflammation.

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P2860

Interleukin-17 contributes to generation of Th1 immunity and neutrophil recruitment during Chlamydia muridarum genital tract infection but is not required for macrophage influx or normal resolution of infection

http://www.wikidata.org/entity/Q34739767

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2010 nî lūn-bûn

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2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Interleukin-17 contributes to ...... normal resolution of infection

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Infection and Immunity

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Interleukin-17 contributes to ...... normal resolution of infection

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Amy M Scurlock

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Catherine M O'Connell

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Charles W Andrews

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Isaac P Foote

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Jay K Kolls

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Kumar Chandra-Kuntal

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Lauren C Frazer

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Sarabeth L Bailey

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Toni Darville

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P2860

A receptor for the heterodimeric cytokine IL-23 is composed of IL-12Rbeta1 and a novel cytokine receptor subunit, IL-23R

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

Transforming growth factor-beta induces development of the T(H)17 lineage

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17

Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages

IL-17 family cytokines and the expanding diversity of effector T cell lineages

A MyD88-dependent early IL-17 production protects mice against airway infection with the obligate intracellular pathogen Chlamydia muridarum.

Th17 cells at the crossroads of innate and adaptive immunity against infectious diseases at the mucosa.

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1349-1362

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10.1128/IAI.00984-10

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3

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79

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21149587

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