Glutamic acid decarboxylase and glutamate receptor changes during tolerance and dependence to benzodiazepines. - Wikidata - awgldk - TriplyDB

Glutamic acid decarboxylase and glutamate receptor changes during tolerance and dependence to benzodiazepines.

Glutamic acid decarboxylase and glutamate receptor changes during tolerance and dependence to benzodiazepines.

http://www.wikidata.org/entity/Q34742725

about

Mechanisms Underlying Tolerance after Long-Term Benzodiazepine Use: A Future for Subtype-Selective GABA(A) Receptor Modulators?Abuse and dependence liability of benzodiazepine-type drugs: GABA(A) receptor modulation and beyond Associations of glutamate decarboxylase genes with initial sensitivity and age-at-onset of alcohol dependence in the Irish Affected Sib Pair Study of Alcohol Dependence.Calcium/calmodulin-dependent protein kinase II mediates hippocampal glutamatergic plasticity during benzodiazepine withdrawal.Immunogold electron microscopic evidence of differential regulation of GluN1, GluN2A, and GluN2B, NMDA-type glutamate receptor subunits in rat hippocampal CA1 synapses during benzodiazepine withdrawal.Selective changes in sensitivity to benzodiazepines, and not other positive GABA(A) modulators, in rats receiving flunitrazepam chronically Effects of estrogen treatment on expression of brain-derived neurotrophic factor and cAMP response element-binding protein expression and phosphorylation in rat amygdaloid and hippocampal structures Down-regulation of synaptic GluN2B subunit-containing N-methyl-D-aspartate receptors: a physiological brake on CA1 neuron α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid hyperexcitability during benzodiazepine withdrawal Tolerance to the rate-increasing and not rate-decreasing effects of pregnanolone in rats GABAA receptor drugs and neuronal plasticity in reward and aversion: focus on the ventral tegmental area Down-regulation of dendritic spine and glutamic acid decarboxylase 67 expressions in the reelin haploinsufficient heterozygous reeler mouse.Neuronal gamma-aminobutyric acid (GABA) type A receptors undergo cognate ligand chaperoning in the endoplasmic reticulum by endogenous GABA.Regulation of Ca²⁺/calmodulin-dependent protein kinase II signaling within hippocampal glutamatergic postsynapses during flurazepam withdrawal.Abnormal cerebellar cytoarchitecture and impaired inhibitory signaling in adult mice lacking TR4 orphan nuclear receptor.Increased AMPA receptor GluR1 subunit incorporation in rat hippocampal CA1 synapses during benzodiazepine withdrawal Reduced phosphorylation of GluA1 subunits relates to anxiety-like behaviours in mice.A neurochemical basis for an epigenetic vision of psychiatric disorders (1994-2009).Differential effects of two chronic diazepam treatment regimes on withdrawal anxiety and AMPA receptor characteristics.Duration of treatment and activation of α1-containing GABAA receptors variably affect the level of anxiety and seizure susceptibility after diazepam withdrawal in rats.Cognate Ligand Chaperoning: a Novel Mechanism for the Post-translational Regulation of Neurotransmitter Receptor Biogenesis.Intrathecal midazolam regulates spinal AMPA receptor expression and function after nerve injury in rats.Adaptive changes in the rat hippocampal glutamatergic neurotransmission are observed during long-term treatment with lorazepam.The decreased phosphorylation of cyclic adenosine monophosphate (cAMP) response element binding (CREB) protein in the central amygdala acts as a molecular substrate for anxiety related to ethanol withdrawal in rats.Transient plasticity of hippocampal CA1 neuron glutamate receptors contributes to benzodiazepine withdrawal-anxiety.Long-lasting modulation of glutamatergic transmission in VTA dopamine neurons after a single dose of benzodiazepine agonists.Innate differences in the expression of brain-derived neurotrophic factor in the regions within the extended amygdala between alcohol preferring and nonpreferring rats.Changes of brain neuropeptide Y and its receptors in rats with flurazepam tolerance and dependence.Neonatal Clonazepam Administration Induces Long-Lasting Changes in Glutamate Receptors

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Glutamic acid decarboxylase and glutamate receptor changes during tolerance and dependence to benzodiazepines.

http://www.wikidata.org/entity/Q34742725

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2001 nî lūn-bûn

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Proceedings of the National Academy of Sciences of the United States of America

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A Guidotti

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P2860

Imidazenil, a partial positive allosteric modulator of GABAA receptors, exhibits low tolerance and dependence liabilities in the rat

Binding of the GABA(A) receptor-associated protein (GABARAP) to microtubules and microfilaments suggests involvement of the cytoskeleton in GABARAPGABA(A) receptor interaction

Loss of postsynaptic GABA(A) receptor clustering in gephyrin-deficient mice

Protein-synthetic machinery beneath postsynaptic sites on CNS neurons: association between polyribosomes and other organelles at the synaptic site.

AMPA receptors unbound: membrane cycling and synaptic plasticity.

Sedative but not anxiolytic properties of benzodiazepines are mediated by the GABA(A) receptor alpha1 subtype.

Local protein synthesis and its role in synapse-specific plasticity.

The NMDA receptor competitive antagonist CPP modulates benzodiazepine tolerance and discontinuation.

Benzodiazepines: use, abuse, and consequences.

Diazepam dependence prevented by glutamate antagonists

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11248104

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30679

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