miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines. - Wikidata - awgldk - TriplyDB

miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines.

miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines.

http://www.wikidata.org/entity/Q34745553

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The emerging role of miR-506 in cancer MicroRNA Regulation of Epithelial to Mesenchymal Transition Key nodes of a microRNA network associated with the integrated mesenchymal subtype of high-grade serous ovarian cancer MiR-101 suppresses the epithelial-to-mesenchymal transition by targeting ZEB1 and ZEB2 in ovarian carcinoma Regulation of the mRNA half-life in breast cancer MiR-506 suppresses proliferation of hepatoma cells through targeting YAP mRNA 3'UTR.MiR-506 inhibits multiple targets in the epithelial-to-mesenchymal transition network and is associated with good prognosis in epithelial ovarian cancer Post-translational regulation of the type III inositol 1,4,5-trisphosphate receptor by miRNA-506.Comprehensive proteome quantification reveals NgBR as a new regulator for epithelial-mesenchymal transition of breast tumor cells MiR-506 is down-regulated in clear cell renal cell carcinoma and inhibits cell growth and metastasis via targeting FLOT1 MicroRNA-506 suppresses growth and metastasis of oral squamous cell carcinoma via targeting GATA6.MiR-506: A Multitasker in Suppression of the Epithelial-to-Mesenchymal Transition MiR-506 Over-Expression Inhibits Proliferation and Metastasis of Breast Cancer Cells.An update on miRNAs as biological and clinical determinants in colorectal cancer: a bench-to-bedside approach.Up-regulation of miR-506 inhibits cell growth and disrupt the cell cycle by targeting YAP in breast cancer cells.microRNA-506 regulates proliferation, migration and invasion in hepatocellular carcinoma by targeting F-spondin 1 (SPON1)MicroRNA-506 suppresses tumor proliferation and metastasis in colon cancer by directly targeting the oncogene EZH2 Meta-Analysis of Differential Connectivity in Gene Co-Expression Networks in Multiple Sclerosis.Plasma miRNA-506 as a Prognostic Biomarker for Esophageal Squamous Cell Carcinoma Downregulated miR-506 expression facilitates pancreatic cancer progression and chemoresistance via SPHK1/Akt/NF-κB signaling.Regulation of the unfolded protein response by microRNAs.Post-transcriptional regulatory network of epithelial-to-mesenchymal and mesenchymal-to-epithelial transitions.Corepressor metastasis-associated protein 3 modulates epithelial-to-mesenchymal transition and metastasis.Genetic and epigenetic silencing of mircoRNA-506-3p enhances COTL1 oncogene expression to foster non-small lung cancer progression.miRNAs and their role in the correlation between schizophrenia and cancer (Review).The role of microRNAs in human breast cancer progression.Identification of breast cancer mechanism based on weighted gene coexpression network analysis.MiR-506 inhibits PRRSV replication in MARC-145 cells via CD151.Selective killing of lung cancer cells by miRNA-506 molecule through inhibiting NF-κB p65 to evoke reactive oxygen species generation and p53 activation.Biomarkers for EMT and MET in breast cancer: An update.Overexpression of miR-506 suppresses proliferation and promotes apoptosis of osteosarcoma cells by targeting astrocyte elevated gene-1 Genome wide predictions of miRNA regulation by transcription factors.miR-506 inhibits the proliferation and invasion by targeting IGF2BP1 in glioblastoma.Evaluation of miR-506 and miR-4316 expression in early and non-invasive diagnosis of colorectal cancer.The miR-506-Induced Epithelial-Mesenchymal Transition is Involved in Poor Prognosis for Patients with Gastric Cancer.Bioinformatics analysis of gene expression data for the identification of critical genes in breast invasive carcinoma.miR-506 suppresses neuroblastoma metastasis by targeting ROCK1 Therapeutic impacts of microRNAs in breast cancer by their roles in regulating processes involved in this disease.Aberrant miRNA promoter methylation and EMT-involving miRNAs in breast cancer metastasis: Diagnosis and therapeutic implications.Anti-neoplastic drugs increase caveolin-1-dependent migration, invasion and metastasis of cancer cells.

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P2860

miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines.

http://www.wikidata.org/entity/Q34745553

description

2013 nî lūn-bûn

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2013 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2013 թվականի մայիսին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines.

@ast

miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines.

@en

miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines.

@nl

type

label

miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines.

@ast

miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines.

@en

miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines.

@nl

prefLabel

miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines.

@ast

miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines.

@en

miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines.

@nl

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miR-506 regulates epithelial mesenchymal transition in breast cancer cell lines.

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Himanshu Arora

http://www.w3.org/2001/XMLSchema#string

Rehana Qureshi

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Woong-Yang Park

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P2860

Epithelial-mesenchymal transitions in tumour progression

Role of estrogen receptor alpha in modulating IGF-I receptor signaling and function in breast cancer

EDG-1 links the PDGF receptor to Src and focal adhesion kinase activation leading to lamellipodia formation and cell migration

microRNA functions

Inhibitory effect of HGF on invasiveness of aggressive MDA-MB231 breast carcinoma cells, and role of HDACs.

Sustained c-Jun-NH2-kinase activity promotes epithelial-mesenchymal transition, invasion, and survival of breast cancer cells by regulating extracellular signal-regulated kinase activation.

Involvement of NF-κB/miR-448 regulatory feedback loop in chemotherapy-induced epithelial-mesenchymal transition of breast cancer cells.

Epithelial-mesenchymal transition: NF-kappaB takes center stage.

Role of tissue stroma in cancer cell invasion.

MicroRNA regulation of cancer stem cells

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