Chemokine receptor 2-mediated accumulation of fungicidal exudate macrophages in mice that clear cryptococcal lung infection. - Wikidata - awgldk - TriplyDB

Chemokine receptor 2-mediated accumulation of fungicidal exudate macrophages in mice that clear cryptococcal lung infection.

Chemokine receptor 2-mediated accumulation of fungicidal exudate macrophages in mice that clear cryptococcal lung infection.

http://www.wikidata.org/entity/Q34756043

about

Pattern recognition scavenger receptor A/CD204 regulates airway inflammatory homeostasis following organic dust extract exposures Chemokine receptors and their therapeutic opportunities in diseased lung: far beyond leukocyte trafficking Immunoregulation in Fungal Diseases Scavenger receptor A modulates the immune response to pulmonary Cryptococcus neoformans infection.Analysis of cell cycle and replication of mouse macrophages after in vivo and in vitro Cryptococcus neoformans infection using laser scanning cytometry Resident alveolar macrophages suppress, whereas recruited monocytes promote, allergic lung inflammation in murine models of asthma Macrophage mitochondrial and stress response to ingestion of Cryptococcus neoformans.Cryptococcus neoformans-induced macrophage lysosome damage crucially contributes to fungal virulence.Mononuclear phagocyte-mediated antifungal immunity: the role of chemotactic receptors and ligands.Cryptococcal heat shock protein 70 homolog Ssa1 contributes to pulmonary expansion of Cryptococcus neoformans during the afferent phase of the immune response by promoting macrophage M2 polarization Type I IFN Induction via Poly-ICLC Protects Mice against Cryptococcosis.Distinct Upstream Role of Type I IFN Signaling in Hematopoietic Stem Cell-Derived and Epithelial Resident Cells for Concerted Recruitment of Ly-6Chi Monocytes and NK Cells via CCL2-CCL3 Cascade Early induction of CCL7 downstream of TLR9 signaling promotes the development of robust immunity to cryptococcal infection.The Cnes2 locus on mouse chromosome 17 regulates host defense against cryptococcal infection through pleiotropic effects on host immunity Susceptibility to progressive Cryptococcus neoformans pulmonary infection is regulated by loci on mouse chromosomes 1 and 9.PAI-1 promotes the accumulation of exudate macrophages and worsens pulmonary fibrosis following type II alveolar epithelial cell injury.Cryptococcus neoformans growth and protection from innate immunity are dependent on expression of a virulence-associated DEAD-box protein, Vad1.Implicating exudate macrophages and Ly-6C(high) monocytes in CCR2-dependent lung fibrosis following gene-targeted alveolar injury.Altered immune response differentially enhances susceptibility to Cryptococcus neoformans and Cryptococcus gattii infection in mice expressing the HIV-1 transgene.Macrophage M1/M2 polarization dynamically adapts to changes in cytokine microenvironments in Cryptococcus neoformans infection.Selective and inducible targeting of CD11b+ mononuclear phagocytes in the murine lung with hCD68-rtTA transgenic systems.Interleukin-17A enhances host defense against cryptococcal lung infection through effects mediated by leukocyte recruitment, activation, and gamma interferon production Induction of protective immunity against cryptococcosis.Local GM-CSF-Dependent Differentiation and Activation of Pulmonary Dendritic Cells and Macrophages Protect against Progressive Cryptococcal Lung Infection in Mice.Role of dendritic cell-pathogen interactions in the immune response to pulmonary cryptococcal infection.Sodium chloride promotes tissue inflammation via osmotic stimuli in subtotal-nephrectomized mice.Monocyte-mediated defense against bacteria, fungi, and parasites.T Cell-Restricted Notch Signaling Contributes to Pulmonary Th1 and Th2 Immunity during Cryptococcus neoformans Infection.Scavenger Receptor MARCO Orchestrates Early Defenses and Contributes to Fungal Containment during Cryptococcal Infection.Lung epithelium is the major source of IL-33 and is regulated by IL-33-dependent and IL-33-independent mechanisms in pulmonary cryptococcosis.Innate Immune Responses to Cryptococcus.Early or late IL-10 blockade enhances Th1 and Th17 effector responses and promotes fungal clearance in mice with cryptococcal lung infection Exploitation of Scavenger Receptor, Macrophage Receptor with Collagenous Structure, by Cryptococcus neoformans Promotes Alternative Activation of Pulmonary Lymph Node CD11b+ Conventional Dendritic Cells and Non-Protective Th2 Bias.The F-Box Protein Fbp1 Shapes the Immunogenic Potential of Cryptococcus neoformans.B Cells Protect T Cell-deficient mice from Cryptococcal Brain Invasion.Contribution of IL-1RI Signaling to Protection against Cryptococcus neoformans 52D in a Mouse Model of Infection.Host response to pulmonary fungal infections: A highlight on cell-driven immunity to Cryptococcus species and Aspergillus fumigatus.IL-1α induces CD11blow alveolar macrophage proliferation and maturation during granuloma formation

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Chemokine receptor 2-mediated accumulation of fungicidal exudate macrophages in mice that clear cryptococcal lung infection.

http://www.wikidata.org/entity/Q34756043

description

2010 nî lūn-bûn

@nan

2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

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2010年論文

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2010年论文

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name

Chemokine receptor 2-mediated ...... r cryptococcal lung infection.

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Chemokine receptor 2-mediated ...... r cryptococcal lung infection.

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Chemokine receptor 2-mediated ...... r cryptococcal lung infection.

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type

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Chemokine receptor 2-mediated ...... r cryptococcal lung infection.

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Chemokine receptor 2-mediated ...... r cryptococcal lung infection.

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Chemokine receptor 2-mediated ...... r cryptococcal lung infection.

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Chemokine receptor 2-mediated ...... r cryptococcal lung infection.

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J.AJPATH.2010.11.006

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The American Journal of Pathology

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Chemokine receptor 2-mediated ...... r cryptococcal lung infection.

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Galen B Toews

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Gary B Huffnagle

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Gwo-Hsiao Chen

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John J Osterholzer

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Yan-Mei Zhang

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P2860

Blood monocytes: development, heterogeneity, and relationship with dendritic cells

Identification of splenic reservoir monocytes and their deployment to inflammatory sites

Severe reduction in leukocyte adhesion and monocyte extravasation in mice deficient in CC chemokine receptor 2.

CCR2 expression determines T1 versus T2 polarization during pulmonary Cryptococcus neoformans infection

Monocyte emigration from bone marrow during bacterial infection requires signals mediated by chemokine receptor CCR2

Transient neutralization of tumor necrosis factor alpha can produce a chronic fungal infection in an immunocompetent host: potential role of immature dendritic cells.

Pulmonary infection with an interferon-gamma-producing Cryptococcus neoformans strain results in classical macrophage activation and protection.

Cryptococcus neoformans is a facultative intracellular pathogen in murine pulmonary infection.

Accumulation of CD11b+ lung dendritic cells in response to fungal infection results from the CCR2-mediated recruitment and differentiation of Ly-6Chigh monocytes.

The gamma interferon receptor is required for the protective pulmonary inflammatory response to Cryptococcus neoformans.

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