Atrial-selective sodium channel block as a strategy for suppression of atrial fibrillation - Wikidata - awgldk - TriplyDB

Atrial-selective sodium channel block as a strategy for suppression of atrial fibrillation

Atrial-selective sodium channel block as a strategy for suppression of atrial fibrillation

http://www.wikidata.org/entity/Q34765700

about

Ion Channels in the Heart Emerging clinical role of ranolazine in the management of angina.Novel pharmacological targets for the rhythm control management of atrial fibrillation.Atrial-selective sodium channel blockers: do they exist?PITX2 Modulates Atrial Membrane Potential and the Antiarrhythmic Effects of Sodium-Channel Blockers.Atrial-selective sodium channel block for the treatment of atrial fibrillation.New pharmacological strategies for the treatment of atrial fibrillation Atrial-selective sodium channel block as a novel strategy for the management of atrial fibrillation.New developments in atrial antiarrhythmic drug therapy.Mechanisms underlying atrial-selective block of sodium channels by Wenxin Keli: Experimental and theoretical analysis.Novel ion channel targets in atrial fibrillation.Tetrodotoxin-sensitive α-subunits of voltage-gated sodium channels are relevant for inhibition of cardiac sodium currents by local anesthetics.How Do Atrial-Selective Drugs Differ From Antiarrhythmic Drugs Currently Used in the Treatment of Atrial Fibrillation?Anti-arrhythmic strategies for atrial fibrillation: The role of computational modeling in discovery, development, and optimization.Effective suppression of atrial fibrillation by the antihistaminic agent antazoline: First experimental insights into a novel antiarrhythmic agent.Antiarrhythmic properties of ranolazine--from bench to bedside.Atrial-selective sodium channel block as a novel strategy for the management of atrial fibrillation.In silico optimization of atrial fibrillation-selective sodium channel blocker pharmacodynamics.How Do Atrial-Selective Drugs Differ From Antiarrhythmic Drugs Currently Used in the Treatment of Atrial Fibrillation?Atrial-selective targeting of arrhythmogenic phase-3 early afterdepolarizations in human myocytes Ranolazine inhibition of hERG potassium channels: drug-pore interactions and reduced potency against inactivation mutants.Potassium currents in the heart: functional roles in repolarization, arrhythmia and therapeutics.Atrial-ventricular differences in rabbit cardiac voltage-gated Na+ currents: Basis for atrial-selective block by ranolazine.Further insights into the underlying electrophysiological mechanisms for reduction of atrial fibrillation by ranolazine in an experimental model of chronic heart failure.

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P2860

Atrial-selective sodium channel block as a strategy for suppression of atrial fibrillation

http://www.wikidata.org/entity/Q34765700

description

2008 nî lūn-bûn

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2008 թուականի Մարտին հրատարակուած գիտական յօդուած

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2008 թվականի մարտին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Atrial-selective sodium channe ...... ression of atrial fibrillation

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Atrial-selective sodium channe ...... ression of atrial fibrillation

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Atrial-selective sodium channe ...... ression of atrial fibrillation

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type

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Atrial-selective sodium channe ...... ression of atrial fibrillation

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Atrial-selective sodium channe ...... ression of atrial fibrillation

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Atrial-selective sodium channe ...... ression of atrial fibrillation

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Atrial-selective sodium channe ...... ression of atrial fibrillation

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Atrial-selective sodium channe ...... ression of atrial fibrillation

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Atrial-selective sodium channe ...... ression of atrial fibrillation

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ANNALS.1420.012

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Annals of the New York Academy of Sciences

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Atrial-selective sodium channe ...... ression of atrial fibrillation

@en

P2093

Alexander Burashnikov

http://www.w3.org/2001/XMLSchema#string

Andrew C Zygmunt

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José M Di Diego

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Luiz Belardinelli

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P2860

Kv1.5 channelopathy due to KCNA5 loss-of-function mutation causes human atrial fibrillation

Comparative analysis of the action of class I antiarrhythmic drugs (lidocaine, quinidine, and prajmaline) in rabbit atrial and ventricular myocardium

The M cell: its contribution to the ECG and to normal and abnormal electrical function of the heart.

Electrophysiological effects of ranolazine, a novel antianginal agent with antiarrhythmic properties.

Preliminary report: effect of encainide and flecainide on mortality in a randomized trial of arrhythmia suppression after myocardial infarction.

Role of repolarization restitution in the development of coarse and fine atrial fibrillation in the isolated canine right atria.

Ranolazine improves abnormal repolarization and contraction in left ventricular myocytes of dogs with heart failure by inhibiting late sodium current

Electrophysiologic properties and antiarrhythmic actions of a novel antianginal agent.

Late-phase 3 EAD. A unique mechanism contributing to initiation of atrial fibrillation

Basic concepts in cellular cardiac electrophysiology: Part II: Block of ion channels by antiarrhythmic drugs.

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105-112

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scholarly article

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10.1196/ANNALS.1420.012

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1123

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Charles Antzelevitch

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2008-03-01T00:00:00Z

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5476182

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18375582

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P921

atrial fibrillation

P932

2366169

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