One process for pancreatic beta-cell coalescence into islets involves an epithelial-mesenchymal transition.
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Regulation of CTNNB1 signaling in gastric cancer and stem cellsPancreas development in humansThe Beta Cell in Its Cluster: Stochastic Graphs of Beta Cell Connectivity in the Islets of LangerhansInsm1 promotes endocrine cell differentiation by modulating the expression of a network of genes that includes Neurog3 and Ripply3Chronic exposure to elevated norepinephrine suppresses insulin secretion in fetal sheep with placental insufficiency and intrauterine growth restriction.Evidence for epithelial-mesenchymal transition in adult human pancreatic exocrine cells.Increased adrenergic signaling is responsible for decreased glucose-stimulated insulin secretion in the chronically hyperinsulinemic ovine fetusCharacterization of glucose-insulin responsiveness and impact of fetal number and sex difference on insulin response in the sheep fetus.Fetal adrenal demedullation lowers circulating norepinephrine and attenuates growth restriction but not reduction of endocrine cell mass in an ovine model of intrauterine growth restrictionPlacental insufficiency decreases pancreatic vascularity and disrupts hepatocyte growth factor signaling in the pancreatic islet endothelial cell in fetal sheepG protein-coupled receptor signaling and sphingosine-1-phosphate play a phylogenetically conserved role in endocrine pancreas morphogenesisRole of epithelial-mesenchymal transition in repair of the lacrimal gland after experimentally induced injury.The transcriptional co-repressor Grg3/Tle3 promotes pancreatic endocrine progenitor delamination and β-cell differentiation.Insulin-like growth factor and fibroblast growth factor expression profiles in growth-restricted fetal sheep pancreas.Increased maternal nighttime cortisol concentrations in late gestation alter glucose and insulin in the neonatal lamb.A Synthetic Heterobivalent Ligand Composed of Glucagon-Like Peptide 1 and Yohimbine Specifically Targets β Cells Within the PancreasConsequences of a compromised intrauterine environment on islet function.Increased fetal insulin concentrations for one week fail to improve insulin secretion or β-cell mass in fetal sheep with chronically reduced glucose supplyIncreased amino acid supply potentiates glucose-stimulated insulin secretion but does not increase β-cell mass in fetal sheep.Establishment and characterization of immortalized human amniotic epithelial cells.ALDH1B1 is a potential stem/progenitor marker for multiple pancreas progenitor pools.Reductions in insulin concentrations and β-cell mass precede growth restriction in sheep fetuses with placental insufficiencyIntrauterine growth-restricted sheep fetuses exhibit smaller hindlimb muscle fibers and lower proportions of insulin-sensitive Type I fibers near term.Tissue engineering 2.0: guiding self-organization during pluripotent stem cell differentiation.Notch signaling in the pancreas: patterning and cell fate specification.Transcriptional control of mammalian pancreas organogenesis.Diallyl disulfide inhibits growth and metastatic potential of human triple-negative breast cancer cells through inactivation of the β-catenin signaling pathway.Mechanisms of adult human β-cell in vitro dedifferentiation and redifferentiation.Chrysin inhibits metastatic potential of human triple-negative breast cancer cells by modulating matrix metalloproteinase-10, epithelial to mesenchymal transition, and PI3K/Akt signaling pathway.Overexpression of NKX6.1 is closely associated with progressive features and predicts unfavorable prognosis in human primary hepatocellular carcinoma.Differential effects of chronic pulsatile versus chronic constant maternal hyperglycemia on fetal pancreatic β-cells.Neurogenin3 initiates stepwise delamination of differentiating endocrine cells during pancreas development.Islet adaptations in fetal sheep persist following chronic exposure to high norepinephrine.β2-Adrenergic receptor desensitization in perirenal adipose tissue in fetuses and lambs with placental insufficiency-induced intrauterine growth restriction.Myoblasts from intrauterine growth-restricted sheep fetuses exhibit intrinsic deficiencies in proliferation that contribute to smaller semitendinosus myofibres.In vivo imaging of emerging endocrine cells reveals a requirement for PI3K-regulated motility in pancreatic islet morphogenesis.GLIS1-3: emerging roles in reprogramming, stem and progenitor cell differentiation and maintenance.Cellular and molecular mechanisms coordinating pancreas development.Hypoxaemia-induced catecholamine secretion from adrenal chromaffin cells inhibits glucose-stimulated hyperinsulinaemia in fetal sheep.Enhanced insulin secretion and insulin sensitivity in young lambs with placental insufficiency-induced intrauterine growth restriction.
P2860
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P2860
One process for pancreatic beta-cell coalescence into islets involves an epithelial-mesenchymal transition.
description
2009 nî lūn-bûn
@nan
2009 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
One process for pancreatic bet ...... helial-mesenchymal transition.
@ast
One process for pancreatic bet ...... helial-mesenchymal transition.
@en
One process for pancreatic bet ...... helial-mesenchymal transition.
@nl
type
label
One process for pancreatic bet ...... helial-mesenchymal transition.
@ast
One process for pancreatic bet ...... helial-mesenchymal transition.
@en
One process for pancreatic bet ...... helial-mesenchymal transition.
@nl
prefLabel
One process for pancreatic bet ...... helial-mesenchymal transition.
@ast
One process for pancreatic bet ...... helial-mesenchymal transition.
@en
One process for pancreatic bet ...... helial-mesenchymal transition.
@nl
P2093
P2860
P356
P1476
One process for pancreatic bet ...... helial-mesenchymal transition.
@en
P2093
Miranda Anderson
Parker B Antin
Sean W Limesand
P2860
P356
10.1677/JOE-09-0072
P407
P577
2009-07-16T00:00:00Z