Exercise protects against doxorubicin-induced oxidative stress and proteolysis in skeletal muscle.
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Skeletal Muscle an Active Compartment in the Sequestering and Metabolism of Doxorubicin ChemotherapyGlobal Proteome Changes in the Rat Diaphragm Induced by Endurance Exercise Training.Data on skeletal muscle apoptosis, autophagy, and morphology in mice treated with doxorubicin.Progressive resistance training and cancer testis (PROTRACT) - efficacy of resistance training on muscle function, morphology and inflammatory profile in testicular cancer patients undergoing chemotherapy: design of a randomized controlled trial.Voluntary exercise prevents cisplatin-induced muscle wasting during chemotherapy in miceHeat shock response and autophagy--cooperation and control.Effects of short-term endurance exercise training on acute doxorubicin-induced FoxO transcription in cardiac and skeletal muscle.Can endurance exercise preconditioning prevention disuse muscle atrophy?Mitochondrial-targeted antioxidants protect skeletal muscle against immobilization-induced muscle atrophyAutophagic signaling and proteolytic enzyme activity in cardiac and skeletal muscle of spontaneously hypertensive rats following chronic aerobic exerciseDoxorubicin acts via mitochondrial ROS to stimulate catabolism in C2C12 myotubesEndurance exercise attenuates ventilator-induced diaphragm dysfunction.Variations in oxidative stress markers in elite basketball players at the beginning and end of a season.Doxorubicin caused severe hyperglycaemia and insulin resistance, mediated by inhibition in AMPk signalling in skeletal muscle.The effects of acute doxorubicin treatment on proteome lysine acetylation status and apical caspases in skeletal muscle of fasted animals.Systemic blockade of ACVR2B ligands prevents chemotherapy-induced muscle wasting by restoring muscle protein synthesis without affecting oxidative capacity or atrogenes.The anticancer agent doxorubicin disrupts mitochondrial energy metabolism and redox balance in skeletal muscle.BGP-15 Protects against Oxaliplatin-Induced Skeletal Myopathy and Mitochondrial Reactive Oxygen Species Production in Mice.Protein breakdown in muscle wasting: role of autophagy-lysosome and ubiquitin-proteasomeNutritional strategies to counteract muscle atrophy caused by disuse and to improve recovery.Role of exercise-induced reactive oxygen species in the modulation of heat shock protein response.Mitochondria: Inadvertent targets in chemotherapy-induced skeletal muscle toxicity and wasting?Targeted overexpression of mitochondrial catalase protects against cancer chemotherapy-induced skeletal muscle dysfunction.Exercise training prevents skeletal muscle damage in an experimental sepsis model.Loss of MKP-5 promotes myofiber survival by activating STAT3/Bcl-2 signaling during regenerative myogenesis.Physical activity and physical fitness in lymphoma patients before, during, and after chemotherapy: a prospective longitudinal study.Fiber atrophy, oxidative stress, and oxidative fiber reduction are the attributes of different phenotypes in chronic obstructive pulmonary disease patients.Increased mitochondrial emission of reactive oxygen species and calpain activation are required for doxorubicin-induced cardiac and skeletal muscle myopathy.Tissue retention of doxorubicin and its effects on cardiac, smooth, and skeletal muscle function.Exercise Combined with Rhodiola sacra Supplementation Improves Exercise Capacity and Ameliorates Exhaustive Exercise-Induced Muscle Damage through Enhancement of Mitochondrial Quality Control.Effects of endurance exercise and doxorubicin on skeletal muscle myogenic regulatory factor expression.Metabolic Changes During Cancer Cachexia Pathogenesis.Creatine Supplementation and Doxorubicin-Induced Skeletal Muscle Dysfunction: An Ex Vivo Investigation.Chemotherapeutic Drugs and Mitochondrial Dysfunction: Focus on Doxorubicin, Trastuzumab, and Sunitinib.Acylated and unacylated ghrelin inhibit doxorubicin-induced apoptosis in skeletal muscleSecreted Frizzled-Related Protein-2 Inhibits Doxorubicin-Induced Apoptosis Mediated through the Akt-mTOR Pathway in Soleus Muscle
P2860
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P2860
Exercise protects against doxorubicin-induced oxidative stress and proteolysis in skeletal muscle.
description
2011 nî lūn-bûn
@nan
2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Exercise protects against doxo ...... roteolysis in skeletal muscle.
@ast
Exercise protects against doxo ...... roteolysis in skeletal muscle.
@en
Exercise protects against doxo ...... roteolysis in skeletal muscle.
@nl
type
label
Exercise protects against doxo ...... roteolysis in skeletal muscle.
@ast
Exercise protects against doxo ...... roteolysis in skeletal muscle.
@en
Exercise protects against doxo ...... roteolysis in skeletal muscle.
@nl
prefLabel
Exercise protects against doxo ...... roteolysis in skeletal muscle.
@ast
Exercise protects against doxo ...... roteolysis in skeletal muscle.
@en
Exercise protects against doxo ...... roteolysis in skeletal muscle.
@nl
P2093
P2860
P1476
Exercise protects against doxo ...... roteolysis in skeletal muscle.
@en
P2093
Andreas N Kavazis
Ashley J Smuder
Scott K Powers
P2860
P304
P356
10.1152/JAPPLPHYSIOL.00677.2010
P407
P577
2011-02-10T00:00:00Z