TNFR1 signaling and IFN-gamma signaling determine whether T cells induce tumor dormancy or promote multistage carcinogenesis. - Wikidata - awgldk - TriplyDB

TNFR1 signaling and IFN-gamma signaling determine whether T cells induce tumor dormancy or promote multistage carcinogenesis.

TNFR1 signaling and IFN-gamma signaling determine whether T cells induce tumor dormancy or promote multistage carcinogenesis.

http://www.wikidata.org/entity/Q34785288

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A human colonic commensal promotes colon tumorigenesis via activation of T helper type 17 T cell responses Tumour progression and metastasis Glioblastoma Circulating Cells: Reality, Trap or Illusion?Mechanisms of disseminated cancer cell dormancy: an awakening field Dormancy of growth-stunted malignant melanoma: sustainable and smoldering patterns From mice to humans: developments in cancer immunoediting Direct crosstalk between mast cell-TNF and TNFR1-expressing endothelia mediates local tissue inflammation.Lipoxygenase mediates invasion of intrametastatic lymphatic vessels and propagates lymph node metastasis of human mammary carcinoma xenografts in mouse.T-helper cell-mediated proliferation and cytokine responses against recombinant Merkel cell polyomavirus-like particles.Blockade of IDO-kynurenine-AhR metabolic circuitry abrogates IFN-γ-induced immunologic dormancy of tumor-repopulating cells Screening of human tumor antigens for CD4 T cell epitopes by combination of HLA-transgenic mice, recombinant adenovirus and antigen peptide libraries.Role of endothelial progenitors and other bone marrow-derived cells in the development of the tumor vasculature Tumor cells disseminate early, but immunosurveillance limits metastatic outgrowth, in a mouse model of melanoma.Early tumor dissemination, but late metastasis: insights into tumor dormancy.The best of both worlds - managing the cancer, saving the bone.Oncogene withdrawal engages the immune system to induce sustained cancer regression.Pathway-based evaluation in early onset colorectal cancer suggests focal adhesion and immunosuppression along with epithelial-mesenchymal transition.CD4(+) T cells contribute to the remodeling of the microenvironment required for sustained tumor regression upon oncogene inactivation An essential role for the immune system in the mechanism of tumor regression following targeted oncogene inactivation.Tumor expression of CD200 inhibits IL-10 production by tumor-associated myeloid cells and prevents tumor immune evasion of CTL therapy.64Cu antibody-targeting of the T-cell receptor and subsequent internalization enables in vivo tracking of lymphocytes by PET.Endogenous T cell responses to antigens expressed in lung adenocarcinomas delay malignant tumor progression.Tumor necrosis factor induces tumor promoting and anti-tumoral effects on pancreatic cancer via TNFR1.Tumor-specific cytotoxic T lymphocyte activity determines colorectal cancer patient prognosis.Effects of inflammatory factors on mesenchymal stem cells and their role in the promotion of tumor angiogenesis in colon cancer CD160 is essential for NK-mediated IFN-γ production Associations between polymorphisms in the IL-4 and IL-4 receptor genes and urinary carcinomas: a meta-analysis.Opposing roles for IL-23 and IL-12 in maintaining occult cancer in an equilibrium state.New insights into cancer immunoediting and its three component phases--elimination, equilibrium and escape ¹H NMR based serum metabolic profiles associated with pathological progression of pancreatic islet β cell tumor in Rip1-Tag2 mice.IL-10 contributes to the suppressive function of tumour-associated myeloid cells and enhances myeloid cell accumulation in tumours.Rapid generation of NY-ESO-1-specific CD4(+) THELPER1 cells for adoptive T-cell therapy.The role of immunosuppression of mesenchymal stem cells in tissue repair and tumor growth Effective anti-neu-initiated antitumor responses require the complex role of CD4+ T cells.Radiation-induced equilibrium is a balance between tumor cell proliferation and T cell-mediated killing Noncanonical roles of the immune system in eliciting oncogene addiction Regulated expression of a tumor-associated antigen reveals multiple levels of T-cell tolerance in a mouse model of lung cancer.Usp18 deficient mammary epithelial cells create an antitumour environment driven by hypersensitivity to IFN-λ and elevated secretion of Cxcl10 Cancer risk in immune-mediated inflammatory diseases (IMID).Biological resonance for cancer metastasis, a new hypothesis based on comparisons between primary cancers and metastases.

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P2860

TNFR1 signaling and IFN-gamma signaling determine whether T cells induce tumor dormancy or promote multistage carcinogenesis.

http://www.wikidata.org/entity/Q34785288

description

2008 nî lūn-bûn

@nan

2008 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի հունիսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

TNFR1 signaling and IFN-gamma ...... ote multistage carcinogenesis.

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TNFR1 signaling and IFN-gamma ...... ote multistage carcinogenesis.

@en

TNFR1 signaling and IFN-gamma ...... ote multistage carcinogenesis.

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type

label

TNFR1 signaling and IFN-gamma ...... ote multistage carcinogenesis.

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TNFR1 signaling and IFN-gamma ...... ote multistage carcinogenesis.

@en

TNFR1 signaling and IFN-gamma ...... ote multistage carcinogenesis.

@nl

prefLabel

TNFR1 signaling and IFN-gamma ...... ote multistage carcinogenesis.

@ast

TNFR1 signaling and IFN-gamma ...... ote multistage carcinogenesis.

@en

TNFR1 signaling and IFN-gamma ...... ote multistage carcinogenesis.

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J.CCR.2008.04.001

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TNFR1 signaling and IFN-gamma ...... ote multistage carcinogenesis.

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P2093

Amir Yazdi

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Bernd Pichler

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Christian A Sander

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Heidi Braumüller

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Kamran Ghoreschi

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Katrin Schaak

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Manfred Kneilling

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Martin Röcken

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Nele Müller-Hermelink

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Ralph Huss

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507-518

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scholarly article

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10.1016/J.CCR.2008.04.001

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13

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Irmgard Förster

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2008-06-01T00:00:00Z

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5318239

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18538734

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