CCL5 promotes VEGF-dependent angiogenesis by down-regulating miR-200b through PI3K/Akt signaling pathway in human chondrosarcoma cells. - Wikidata - awgldk - TriplyDB

CCL5 promotes VEGF-dependent angiogenesis by down-regulating miR-200b through PI3K/Akt signaling pathway in human chondrosarcoma cells.

CCL5 promotes VEGF-dependent angiogenesis by down-regulating miR-200b through PI3K/Akt signaling pathway in human chondrosarcoma cells.

http://www.wikidata.org/entity/Q34786547

about

Overexpression of the miR-141/200c cluster promotes the migratory and invasive ability of triple-negative breast cancer cells through the activation of the FAK and PI3K/AKT signaling pathways by secreting VEGF-A WISP-3 inhibition of miR-452 promotes VEGF-A expression in chondrosarcoma cells and induces endothelial progenitor cells angiogenesis.Dimethyloxaloylglycine Promotes the Angiogenic Activity of Mesenchymal Stem Cells Derived from iPSCs via Activation of the PI3K/Akt Pathway for Bone Regeneration.NF1 germline mutation differentially dictates optic glioma formation and growth in neurofibromatosis-1.Antitumor activity and inhibitory effects on cancer stem cell-like properties of Adeno-associated virus (AAV) -mediated Bmi-1 interference driven by Bmi-1 promoter for gastric cancer.CCL5 promotes VEGF-C production and induces lymphangiogenesis by suppressing miR-507 in human chondrosarcoma cells.Adipocyte miR-200b/a/429 ablation in mice leads to high-fat-diet-induced obesity.Novel therapeutic approaches in chondrosarcoma.MicroRNAs as biomarkers in the diagnosis and treatment of chondrosarcoma.Onco-GPCR signaling and dysregulated expression of microRNAs in human cancer.Endothelin-1 promotes epithelial-mesenchymal transition in human chondrosarcoma cells by repressing miR-300.Pro-angiogenic effect of RANTES-loaded polysaccharide-based microparticles for a mouse ischemia therapy.Mesenchymal stem cells in inflammatory microenvironment potently promote metastatic growth of cholangiocarcinoma via activating Akt/NF-κB signaling by paracrine CCL5.G Protein-Coupled Receptors at the Crossroad between Physiologic and Pathologic Angiogenesis: Old Paradigms and Emerging Concepts.Targeting tumor microenvironment to curb chemoresistance via novel drug delivery strategies.Induction of chemokine (C-C motif) ligand 5 by Epstein-Barr virus infection enhances tumor angiogenesis in nasopharyngeal carcinoma.Clinical Benefit of Pazopanib in a Patient with Metastatic Chondrosarcoma: A Case Report and Review of the Literature.AKT as Locus of Cancer Unknown Primary Site.

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P2860

CCL5 promotes VEGF-dependent angiogenesis by down-regulating miR-200b through PI3K/Akt signaling pathway in human chondrosarcoma cells.

http://www.wikidata.org/entity/Q34786547

description

2014 nî lūn-bûn

@nan

2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2014年の論文

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Chih-Hsin Tang

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Guan-Ting Liu

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Hsi-Kai Tsou

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Hsien-Te Chen

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Jui-Chieh Chen

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Po-Chen Chen

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Shih-Wei Wang

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Tzu-Wei Tan

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Wei-Hung Yang

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Yi-Chin Fong

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P2860

PI3K/AKT/mTOR Pathway in Angiogenesis

The functions of animal microRNAs

Oncomirs - microRNAs with a role in cancer

Angiogenesis in cancer and other diseases

Microarray analysis shows that some microRNAs downregulate large numbers of target mRNAs

MicroRNAs: genomics, biogenesis, mechanism, and function

MiR-200c increases the radiosensitivity of non-small-cell lung cancer cell line A549 by targeting VEGF-VEGFR2 pathway

CCL5 and CCR5 interaction promotes cell motility in human osteosarcoma.

miR-200b targets Ets-1 and is down-regulated by hypoxia to induce angiogenic response of endothelial cells.

MiR-200a inhibits epithelial-mesenchymal transition of pancreatic cancer stem cell.

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