HES1, a target of Notch signaling, is elevated in canine osteosarcoma, but reduced in the most aggressive tumors - Wikidata - awgldk - TriplyDB

HES1, a target of Notch signaling, is elevated in canine osteosarcoma, but reduced in the most aggressive tumors

HES1, a target of Notch signaling, is elevated in canine osteosarcoma, but reduced in the most aggressive tumors

http://www.wikidata.org/entity/Q34788468

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Methylseleninic acid sensitizes Notch3-activated OVCA429 ovarian cancer cells to carboplatin Germline and somatic genetics of osteosarcoma - connecting aetiology, biology and therapy.Meta-analysis of differentially expressed genes in osteosarcoma based on gene expression data.Biological significance and therapeutic implication of resveratrol-inhibited Wnt, Notch and STAT3 signaling in cervical cancer cells miR-381 Regulates Neural Stem Cell Proliferation and Differentiation via Regulating Hes1 Expression Hes1: a key role in stemness, metastasis and multidrug resistance.Notch Signaling and the Skeleton.MiR-34a-5p promotes multi-chemoresistance of osteosarcoma through down-regulation of the DLL1 gene.Hes4: A potential prognostic biomarker for newly diagnosed patients with high-grade osteosarcoma.The clinicopathological significance of HES1 promoter hypomethylation in patients with colorectal cancer.Up-regulation of microRNA-340 promotes osteosarcoma cell apoptosis while suppressing proliferation, migration, and invasion by inactivating the CTNNB1-mediated Notch signaling pathway

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P2860

HES1, a target of Notch signaling, is elevated in canine osteosarcoma, but reduced in the most aggressive tumors

http://www.wikidata.org/entity/Q34788468

description

2013 nî lūn-bûn

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2013 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի հուլիսին հրատարակված գիտական հոդված

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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name

HES1, a target of Notch signal ...... in the most aggressive tumors

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HES1, a target of Notch signal ...... in the most aggressive tumors

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HES1, a target of Notch signal ...... in the most aggressive tumors

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HES1, a target of Notch signal ...... in the most aggressive tumors

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HES1, a target of Notch signal ...... in the most aggressive tumors

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HES1, a target of Notch signal ...... in the most aggressive tumors

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HES1, a target of Notch signal ...... in the most aggressive tumors

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HES1, a target of Notch signal ...... in the most aggressive tumors

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HES1, a target of Notch signal ...... in the most aggressive tumors

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BMC Veterinary Research

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HES1, a target of Notch signal ...... in the most aggressive tumors

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Barbara E Powers

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Dawn L Duval

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Deanna D Dailey

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E J Ehrhart

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J Brad Charles

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Kristin P Anfinsen

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Liza E Pfaff

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Thora J Jonasdottir

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Tina B Bønsdorff

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P2860

Integrative genomic analyses on HES/HEY family: Notch-independent HES1, HES3 transcription in undifferentiated ES cells, and Notch-dependent HES1, HES5, HEY1, HEY2, HEYL transcription in fetal tissues, adult tissues, or cancer

Structure, chromosomal locus, and promoter analysis of the gene encoding the mouse helix-loop-helix factor HES-1. Negative autoregulation through the multiple N box elements

Gene expression levels assessed by oligonucleotide microarray analysis and quantitative real-time RT-PCR -- how well do they correlate?

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

HES and HERP families: multiple effectors of the Notch signaling pathway

Oscillations in notch signaling regulate maintenance of neural progenitors

Activating the PARP-1 sensor component of the groucho/ TLE1 corepressor complex mediates a CaMKinase IIdelta-dependent neurogenic gene activation pathway

Relative expression software tool (REST©) for group-wise comparison and statistical analysis of relative expression results in real-time PCR

Microarray analysis of differentially expressed genes of primary tumors in the canine central nervous system.

Evidence based selection of housekeeping genes.

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Douglas H Thamm

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