Cardiomyocyte overexpression of iNOS in mice results in peroxynitrite generation, heart block, and sudden death - Wikidata - awgldk - TriplyDB

Cardiomyocyte overexpression of iNOS in mice results in peroxynitrite generation, heart block, and sudden death

Cardiomyocyte overexpression of iNOS in mice results in peroxynitrite generation, heart block, and sudden death

http://www.wikidata.org/entity/Q34789751

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Nitric oxide and peroxynitrite in health and disease Altered Nitric Oxide System in Cardiovascular and Renal Diseases Electrical vagus nerve stimulation for the treatment of chronic heart failure Cardiovascular risks associated with low dose ionizing particle radiation The homeodomain transcription factor Irx5 establishes the mouse cardiac ventricular repolarization gradient Arrhythmia and neuronal/endothelial myocyte uncoupling in hyperhomocysteinemia The valosin-containing protein is a novel mediator of mitochondrial respiration and cell survival in the heart in vivo.TNF-TNFR2/p75 signaling inhibits early and increases delayed nontargeted effects in bone marrow-derived endothelial progenitor cells.Dysautonomia due to reduced cholinergic neurotransmission causes cardiac remodeling and heart failure.KMUP-1 attenuates isoprenaline-induced cardiac hypertrophy in rats through NO/cGMP/PKG and ERK1/2/calcineurin A pathways Hypoxia-induced left ventricular dysfunction in myoglobin-deficient mice.TRPV1 Activation Attenuates High-Salt Diet-Induced Cardiac Hypertrophy and Fibrosis through PPAR-δ Upregulation.Cardiac function is preserved following 4 weeks of voluntary wheel running in a rodent model of chronic kidney disease Resveratrol improves left ventricular diastolic relaxation in type 2 diabetes by inhibiting oxidative/nitrative stress: in vivo demonstration with magnetic resonance imaging Decreased vascular lesion formation in mice with inducible endothelial-specific expression of protein kinase Akt Netrin-1 improves post-injury cardiac function in vivo via DCC/NO-dependent preservation of mitochondrial integrity, while attenuating autophagy.Obligatory role of heat shock protein 90 in iNOS induction.Synergism between arrhythmia and hyperhomo-cysteinemia in structural heart disease.Protective effects of chinese traditional medicine buyang huanwu decoction on myocardial injury.Activation of the poly(ADP-ribose) polymerase pathway in human heart failure.Regulation of Inducible Nitric Oxide Synthase (iNOS) and its Potential Role in Insulin Resistance, Diabetes and Heart Failure Reactive oxygen species in cardiovascular disease.Nitric oxide control of cardiac function: is neuronal nitric oxide synthase a key component?Macrophage polarization by angiotensin II-type 1 receptor aggravates renal injury-acceleration of atherosclerosis.Particle Radiation-Induced Nontargeted Effects in Bone-Marrow-Derived Endothelial Progenitor Cells.Nitric oxide signaling and the regulation of myocardial function.Inducible nitric oxide synthase mediates DNA double strand breaks in Human T-Cell Leukemia Virus Type 1-induced leukemia/lymphoma.Differential modulation of nitric oxide synthases in aging: therapeutic opportunities.Nitrosative stress and pharmacological modulation of heart failure.Lack of inducible NO synthase reduces oxidative stress and enhances cardiac response to isoproterenol in mice with deoxycorticosterone acetate-salt hypertension Isothiocyanates ameliorate the symptom of heart dysfunction and mortality in a murine AIDS model by inhibiting apoptosis in the left ventricle Reciprocal regulation of airway rejection by the inducible gas-forming enzymes heme oxygenase and nitric oxide synthase.Low-dose radiation affects cardiac physiology: gene networks and molecular signaling in cardiomyocytes.Modulation of myocardial contraction by peroxynitrite Pathophysiology of hypertension in the absence of nitric oxide/cyclic GMP signaling.Inducible nitric oxide synthase deficiency protects the heart from systolic overload-induced ventricular hypertrophy and congestive heart failure.Sabiporide reduces ischemia-induced arrhythmias and myocardial infarction and attenuates ERK phosphorylation and iNOS induction in rats.Relevance of nitric oxide for myocardial remodeling.Biphasic effect of SIN-1 is reliant upon cardiomyocyte contractile state.Nitric oxide homeostasis as a target for drug additives to cardioplegia

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P2860

Cardiomyocyte overexpression of iNOS in mice results in peroxynitrite generation, heart block, and sudden death

http://www.wikidata.org/entity/Q34789751

description

2002 nî lūn-bûn

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2002 թվականի մարտին հրատարակված գիտական հոդված

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

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Cardiomyocyte overexpression o ...... heart block, and sudden death

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Cardiomyocyte overexpression o ...... heart block, and sudden death

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Cardiomyocyte overexpression o ...... heart block, and sudden death

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Cardiomyocyte overexpression o ...... heart block, and sudden death

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Cardiomyocyte overexpression o ...... heart block, and sudden death

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Cardiomyocyte overexpression o ...... heart block, and sudden death

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Cardiomyocyte overexpression o ...... heart block, and sudden death

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Cardiomyocyte overexpression o ...... heart block, and sudden death

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Journal of Clinical Investigation

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Cardiomyocyte overexpression o ...... heart block, and sudden death

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Asif Pirani

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Azar Azad

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Duncan J Stewart

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Jagdish Butany

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Mansoor Husain

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Richard Schulz

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Robert Gros

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Tamas Csont

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Xiaomang You

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P2860

Nitric oxide synthase complexed with dystrophin and absent from skeletal muscle sarcolemma in Duchenne muscular dystrophy

Nitric oxide synthases in mammals

Tight control of gene expression in mammalian cells by tetracycline-responsive promoters

A novel genetic pathway for sudden cardiac death via defects in the transition between ventricular and conduction system cell lineages

The role of neutrophils in myocardial ischemia-reperfusion injury.

Interactions between NO and reactive oxygen species: pathophysiological importance in atherosclerosis, hypertension, diabetes and heart failure.

Inducible nitric oxide synthase gene expression in the brain during systemic inflammation.

Verapamil ameliorates the clinical and pathological course of murine myocarditis

Beneficial effects and improved survival in rodent models of septic shock with S-methylisothiourea sulfate, a potent and selective inhibitor of inducible nitric oxide synthase

Complete heart block and sudden death in mice overexpressing calreticulin.

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735-743

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10.1172/JCI0213265

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6

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109

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Imran N. Mungrue

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2002-03-01T00:00:00Z

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11901182

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