Identification of renal Cd36 as a determinant of blood pressure and risk for hypertension. - Wikidata - awgldk - TriplyDB

Identification of renal Cd36 as a determinant of blood pressure and risk for hypertension.

Identification of renal Cd36 as a determinant of blood pressure and risk for hypertension.

http://www.wikidata.org/entity/Q34790141

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Genetic analysis of the cardiac methylome at single nucleotide resolution in a model of human cardiovascular disease From integrative genomics to systems genetics in the rat to link genotypes to phenotypes Targeted mutation of SLC4A5 induces arterial hypertension and renal metabolic acidosis Genome-wide co-expression analysis in multiple tissues.Recent advances in genetics of the spontaneously hypertensive rat.Natural variation of histone modification and its impact on gene expression in the rat genome.Metabolic syndrome: from epidemiology to systems biology.Cellular fatty acid uptake: a pathway under construction.CD36 gene deletion decreases oleoylethanolamide levels in small intestine of free-feeding mice.The genome sequence of the spontaneously hypertensive rat: Analysis and functional significance High-resolution identity by descent mapping uncovers the genetic basis for blood pressure differences between spontaneously hypertensive rat lines Defining a rat blood pressure quantitative trait locus to a <81.8 kb congenic segment: comprehensive sequencing and renal transcriptome analysis Genetics of hypertension: from experimental animals to humans.Multi-tissue analysis of co-expression networks by higher-order generalized singular value decomposition identifies functionally coherent transcriptional modules.Integrated genomic approaches to identification of candidate genes underlying metabolic and cardiovascular phenotypes in the spontaneously hypertensive rat.Evidence for a link between gut microbiota and hypertension in the Dahl rat.Common variation in the CD36 (fatty acid translocase) gene is associated with left-ventricular mass.Translational regulation shapes the molecular landscape of complex disease phenotypes.Gene expression suggests spontaneously hypertensive rats may have altered metabolism and reduced hypoxic tolerance The emerging role for rat models in gene discovery.New Wistar Kyoto and spontaneously hypertensive rat transgenic models with ubiquitous expression of green fluorescent protein Consequences of perinatal treatment with L-arginine and antioxidants for the renal transcriptome in spontaneously hypertensive rats.Genome-wide identification of allelic expression in hypertensive rats.Genetic, physiological and comparative genomic studies of hypertension and insulin resistance in the spontaneously hypertensive rat The stroke-prone spontaneously hypertensive rat: still a useful model for post-GWAS genetic studies?Epigenetics and Control of RNAs.Associations between CD36 gene polymorphisms and metabolic response to a short-term endurance-training program in a young-adult population.Dual Linkage of a Locus to Left Ventricular Mass and a Cardiac Gene Co-Expression Network Driven by a Chromosome Domain.CD36 Gene Polymorphisms Are Associated with Intracerebral Hemorrhage Susceptibility in a Han Chinese Population.Systems-level approaches reveal conservation of trans-regulated genes in the rat and genetic determinants of blood pressure in humans.Genetic susceptibility to HIV-associated nephropathy.Whole genome survey of copy number variation in the spontaneously hypertensive rat: relationship to quantitative trait loci, gene expression, and blood pressure.Congenic strains provide evidence that four mapped loci in chromosomes 2, 4, and 16 influence hypertension in the SHR.ANO1 taking center stage: blood pressure regulation in SHRs.The involvement of CD36 in monocyte activation by antiphospholipid antibodies.Mapping genetic determinants of coronary microvascular remodeling in the spontaneously hypertensive rat.Towards Precision Medicine for Hypertension: A Review of Genomic, Epigenomic, and Microbiomic Effects on Blood Pressure in Experimental Rat Models and Humans.Genetic mechanisms of human hypertension and their implications for blood pressure physiology.Diverse development and higher sensitivity of the circadian clocks to changes in maternal-feeding regime in a rat model of cardio-metabolic disease.Genetics of hypertension: an assessment of progress in the spontaneously hypertensive rat.

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P2860

Identification of renal Cd36 as a determinant of blood pressure and risk for hypertension.

http://www.wikidata.org/entity/Q34790141

description

2008 nî lūn-bûn

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2008 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2008年の論文

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Identification of renal Cd36 a ...... ure and risk for hypertension.

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Identification of renal Cd36 a ...... ure and risk for hypertension.

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Nature Genetics

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Identification of renal Cd36 a ...... ure and risk for hypertension.

@en

P2093

Anil Bidani

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Caroline A Wallace

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Enrico Petretto

http://www.w3.org/2001/XMLSchema#string

Heike Zimdahl

http://www.w3.org/2001/XMLSchema#string

Jiaming Wang

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Joseph Dunbar

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Karen Griffin

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Ludmila Kazdova

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Martina Maxova

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Michal Pravenec

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P2860

Transgenic rescue of defective Cd36 ameliorates insulin resistance in spontaneously hypertensive rats

Genetics of Cd36 and the clustering of multiple cardiovascular risk factors in spontaneous hypertension.

Role of nitric oxide in the control of renal function and salt sensitivity.

Finding genes that underlie complex traits.

Pathophysiology of human genetic CD36 deficiency.

Myristic acid stimulates endothelial nitric-oxide synthase in a CD36- and an AMP kinase-dependent manner.

Hypercholesterolemia promotes a CD36-dependent and endothelial nitric-oxide synthase-mediated vascular dysfunction.

Quantitative trait transcripts for nicotine resistance in Drosophila melanogaster.

CD36 expression contributes to age-induced cardiomyopathy in mice.

Identification of Cd36 (Fat) as an insulin-resistance gene causing defective fatty acid and glucose metabolism in hypertensive rats.

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952-954

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10.1038/NG.164

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8

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40

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2008-06-29T00:00:00Z

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18587397

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