Canonical Wnt signaling is critical to estrogen-mediated uterine growth.
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PAX2 activates WNT4 expression during mammalian kidney developmentWNT4 is a key regulator of normal postnatal uterine development and progesterone signaling during embryo implantation and decidualization in the mouse.Mechanisms of uterine estrogen signaling during early pregnancy in mice: an updateParticipation of WNT and β-Catenin in Physiological and Pathological Endometrial Changes: Association with AngiogenesisClinicopathological and molecular analysis of endometrial carcinoma associated with tamoxifenWNTs in the neonatal mouse uterus: potential regulation of endometrial gland developmentCooperative control via lymphoid enhancer factor 1/T cell factor 3 and estrogen receptor-alpha for uterine gene regulation by estrogenThe expression of Wnt4 is regulated by estrogen via an estrogen receptor alpha-dependent pathway in rat pituitary growth hormone-producing cellsbeta-catenin mediates glandular formation and dysregulation of beta-catenin induces hyperplasia formation in the murine uterus.Modulation of Wnt signaling influences fracture repairWnt genes in the mouse uterus: potential regulation of implantationTransforming growth factor-beta1 attenuates expression of both the progesterone receptor and Dickkopf in differentiated human endometrial stromal cellsGenomic profiling of microRNAs and messenger RNAs reveals hormonal regulation in microRNA expression in human endometriumInteraction between bone morphogenetic protein receptor type 2 and estrogenic compounds in pulmonary arterial hypertensionUterine sensing of the embryoEstrogen induces distinct patterns of microRNA expression within the mouse uterusWnt signaling can substitute for estrogen to induce division of ERalpha-positive cells in a mouse mammary tumor model.Suppression of estrogen receptor transcriptional activity by connective tissue growth factor.Involvement of {beta}-catenin and unusual behavior of CBP and p300 in glucocorticosteroid signaling in Schwann cellsEgr1 protein acts downstream of estrogen-leukemia inhibitory factor (LIF)-STAT3 pathway and plays a role during implantation through targeting Wnt4.Wnt signalling in implantation, decidualisation and placental differentiation--review.Endometrial regeneration and endometrial stem/progenitor cells.Physiological and molecular determinants of embryo implantationWhat have we learned about GPER function in physiology and disease from knockout mice?Wnt signaling in estrogen-induced lactotroph proliferation.Integration of estrogen and Wnt signaling circuits by the polycomb group protein EZH2 in breast cancer cells.Increased level of cellular Bip critically determines estrogenic potency for a xenoestrogen kepone in the mouse uterus.GPR30 activation opposes estrogen-dependent uterine growth via inhibition of stromal ERK1/2 and estrogen receptor alpha (ERα) phosphorylation signalsSignalling pathways in endometrial cancerBip is a molecular link between the phase I and phase II estrogenic responses in uterusAvian WNT4 in the female reproductive tracts: potential role of oviduct development and ovarian carcinogenesis.Chromatin immunoprecipitation assay detects ERalpha recruitment to gene specific promoters in uterus.Progesterone action in breast, uterine, and ovarian cancers.Postnatal deletion of Wnt7a inhibits uterine gland morphogenesis and compromises adult fertility in mice.Prokineticin 1 induces Dickkopf 1 expression and regulates cell proliferation and decidualization in the human endometriumDehydroepiandrosterone administration or G{alpha}q overexpression induces {beta}-catenin/T-Cell factor signaling and growth via increasing association of estrogen receptor-{beta}/Dishevelled2 in androgen-independent prostate cancer cells.Uterine epithelial cell estrogen receptor alpha-dependent and -independent genomic profiles that underlie estrogen responses in mice.Conditional deletion of Msx homeobox genes in the uterus inhibits blastocyst implantation by altering uterine receptivity.The regulation of embryo implantation and endometrial decidualization by progesterone receptor signaling.Nucleolar Sik-similar protein (Sik-SP) is required for the maintenance of uterine estrogen signaling mechanism via ERα.
P2860
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P2860
Canonical Wnt signaling is critical to estrogen-mediated uterine growth.
description
2004 nî lūn-bûn
@nan
2004 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Canonical Wnt signaling is critical to estrogen-mediated uterine growth.
@ast
Canonical Wnt signaling is critical to estrogen-mediated uterine growth.
@en
Canonical Wnt signaling is critical to estrogen-mediated uterine growth.
@nl
type
label
Canonical Wnt signaling is critical to estrogen-mediated uterine growth.
@ast
Canonical Wnt signaling is critical to estrogen-mediated uterine growth.
@en
Canonical Wnt signaling is critical to estrogen-mediated uterine growth.
@nl
prefLabel
Canonical Wnt signaling is critical to estrogen-mediated uterine growth.
@ast
Canonical Wnt signaling is critical to estrogen-mediated uterine growth.
@en
Canonical Wnt signaling is critical to estrogen-mediated uterine growth.
@nl
P2093
P2860
P356
P1476
Canonical Wnt signaling is critical to estrogen-mediated uterine growth.
@en
P2093
Meiling Li
Sanjoy K Das
Sudhansu K Dey
Xiaonan Hou
P2860
P304
P356
10.1210/ME.2004-0259
P577
2004-09-09T00:00:00Z