Increased C5a receptor expression in sepsis. - Wikidata - awgldk - TriplyDB

Increased C5a receptor expression in sepsis.

Increased C5a receptor expression in sepsis.

http://www.wikidata.org/entity/Q34793122

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Molecular mechanisms of inflammation and tissue injury after major trauma--is complement the "bad guy"?On the Functional Overlap between Complement and Anti-Microbial Peptides Disruption of the C5a receptor gene increases resistance to acute Gram-negative bacteremia and endotoxic shock: opposing roles of C3a and C5a.Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) prevents lipopolysaccharide (LPS)-induced, sepsis-related severe acute lung injury in mice.Beneficial and detrimental effects of plasmin(ogen) during infection and sepsis in mice.Complement activation in emergency department patients with severe sepsis Sphingosine kinase 1 mediation of expression of the anaphylatoxin receptor C5L2 dampens the inflammatory response to endotoxin Complement in animal development: unexpected roles of a highly conserved pathway.Nrf2 is a critical regulator of the innate immune response and survival during experimental sepsis.Elimination of C5aR prevents intestinal mucosal damage and attenuates neutrophil infiltration in local and remote organs.Adenovirus-mediated in vivo silencing of anaphylatoxin receptor C5aR.Cutting edge: the membrane attack complex of complement is required for the development of murine experimental cerebral malaria.Properdin levels in human sepsis.Anti-inflammatory strategies for the treatment of sepsis.C5a-induced gene expression in human umbilical vein endothelial cells.Disturbed homeostasis of lung intercellular adhesion molecule-1 and vascular cell adhesion molecule-1 during sepsis.The enigma of sepsis.A key role of C5a/C5aR activation for the development of sepsis.MyD88-dependent production of IL-17F is modulated by the anaphylatoxin C5a via the Akt signaling pathway.Thrombin-activatable procarboxypeptidase B regulates activated complement C5a in vivo.MHC class I-specific antibody binding to nonhematopoietic cells drives complement activation to induce transfusion-related acute lung injury in mice Manipulation of the complement system for benefit in sepsis.Alternative Pathway Inhibition by Exogenous Factor H Fails to Attenuate Inflammation and Vascular Leakage in Experimental Pneumococcal Sepsis in Mice.Role of Complement C5 in Experimental Blunt Chest Trauma-Induced Septic Acute Lung Injury (ALI).An essential role for complement C5a in the pathogenesis of septic cardiac dysfunction.Acute lung injury and the acute respiratory distress syndrome: four decades of inquiry into pathogenesis and rational management.Emerging drugs for the treatment of sepsis.Neutrophils orchestrate their own recruitment in murine arthritis through C5aR and FcγR signaling.The role of complement, C5a and its receptors in sepsis and multiorgan dysfunction syndrome.C5 deficiency and C5a or C5aR blockade protects against cerebral malaria Therapeutic potential of HMGB1-targeting agents in sepsis.Is CaMKII a link between inflammation and hypertrophy in heart?Complement component C5a mediates hemorrhage-induced intestinal damage Renal dysfunction in burns: a review.Inhibition of complement C5a prevents breakdown of the blood-brain barrier and pituitary dysfunction in experimental sepsis Immunomodulatory and Antimicrobial Activity of Babassu Mesocarp Improves the Survival in Lethal Sepsis.Sepsis, apoptosis and complement.Anticomplement therapy.Experimental therapeutic strategies for severe sepsis: mediators and mechanisms.Attenuation of IgG immune complex-induced acute lung injury by silencing C5aR in lung epithelial cells.

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Increased C5a receptor expression in sepsis.

http://www.wikidata.org/entity/Q34793122

description

2002 nî lūn-bûn

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2002 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2002 թվականի հուլիսին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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name

Increased C5a receptor expression in sepsis.

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Increased C5a receptor expression in sepsis.

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Increased C5a receptor expression in sepsis.

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type

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Increased C5a receptor expression in sepsis.

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Increased C5a receptor expression in sepsis.

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Increased C5a receptor expression in sepsis.

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prefLabel

Increased C5a receptor expression in sepsis.

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Increased C5a receptor expression in sepsis.

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Increased C5a receptor expression in sepsis.

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Journal of Clinical Investigation

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Increased C5a receptor expression in sepsis.

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P2093

Carl L Pierson

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Christoph W Strey

http://www.w3.org/2001/XMLSchema#string

Dimitrios Mastellos

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Ines J Laudes

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John D Lambris

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Katie A Keller

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Maciej M Markiewski

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Niels C Riedemann

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Ren-Feng Guo

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Thomas A Neff

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P2860

Expression of receptors for C5a anaphylatoxin (CD88) on human bronchial epithelial cells: enhancement of C5a-mediated release of IL-8 upon exposure to cigarette smoke

Expression of the complement anaphylatoxin C3a and C5a receptors on bronchial epithelial and smooth muscle cells in models of sepsis and asthma

Chemotactic and anaphylatoxic fragment cleaved from the fifth component of guinea pig complement

The chemotactic receptor for human C5a anaphylatoxin

Inhibition of immune-complex mediated dermal inflammation in rats following either oral or topical administration of a small molecule C5a receptor antagonist

A new small molecule C5a receptor antagonist inhibits the reverse-passive Arthus reaction and endotoxic shock in rats

Identification of the C3b receptor-binding domain in third component of complement

Selection of a C5a receptor antagonist from phage libraries attenuating the inflammatory response in immune complex disease and ischemia/reperfusion injury.

Expression of the anaphylatoxin C5a receptor in non-myeloid cells.

Endotoxin-induced shock in the rat. A role for C5a

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101-108

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10.1172/JCI0215409

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1

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110

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Firas S Zetoune

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2002-07-01T00:00:00Z

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12093893

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