EZH2-Mediated H3K27me3 Is Involved in Epigenetic Repression of Deleted in Liver Cancer 1 in Human Cancers - Wikidata - awgldk - TriplyDB

EZH2-Mediated H3K27me3 Is Involved in Epigenetic Repression of Deleted in Liver Cancer 1 in Human Cancers

EZH2-Mediated H3K27me3 Is Involved in Epigenetic Repression of Deleted in Liver Cancer 1 in Human Cancers

http://www.wikidata.org/entity/Q34797908

about

Deleted in liver cancer-1 (DLC1): an emerging metastasis suppressor gene Aberrant upregulation of 14-3-3σ and EZH2 expression serves as an inferior prognostic biomarker for hepatocellular carcinoma DLC1 is the principal biologically-relevant down-regulated DLC family member in several cancers ChromNet: Learning the human chromatin network from all ENCODE ChIP-seq data.Significance of genetic variants in DLC1 and their association with hepatocellular carcinoma Mariner Transposons Contain a Silencer: Possible Role of the Polycomb Repressive Complex 2.Mutual regulation between CHD5 and EZH2 in hepatocellular carcinoma.The Polycomb Group Protein EZH2 Impairs DNA Damage Repair Gene Expression in Human Uterine Fibroids Hepatoepigenetic Alterations in Viral and Nonviral-Induced Hepatocellular Carcinoma.Epigenetic modifications in prostate cancer.Targeting of cancer stem cells by inhibitors of DNA and histone methylation.Integrative functional genomic analysis identifies epigenetically regulated fibromodulin as an essential gene for glioma cell migration.Nutritional Epigenetics and the Prevention of Hepatocellular Carcinoma with Bioactive Food Constituents.Epigenetic mechanisms regulating the development of hepatocellular carcinoma and their promise for therapeutics.H3K27me3 is an Epigenetic Mark of Relevance in Endometriosis.EZH2 regulates cancer cell migration through repressing TIMP-3 in non-small cell lung cancer.EZH2 regulates neuroepithelium structure and neuroblast proliferation by repressing p21.The expression and significance of histone lysine methylation in endometrial cancer.EZH2-mediated suppression of lncRNA-LET promotes cell apoptosis and inhibits the proliferation of post-burn skin fibroblasts.

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EZH2-Mediated H3K27me3 Is Involved in Epigenetic Repression of Deleted in Liver Cancer 1 in Human Cancers

http://www.wikidata.org/entity/Q34797908

description

2013 nî lūn-bûn

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2013 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2013 թվականի հունիսին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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name

EZH2-Mediated H3K27me3 Is Invo ...... iver Cancer 1 in Human Cancers

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EZH2-Mediated H3K27me3 Is Invo ...... iver Cancer 1 in Human Cancers

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EZH2-Mediated H3K27me3 Is Invo ...... iver Cancer 1 in Human Cancers

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EZH2-Mediated H3K27me3 Is Invo ...... iver Cancer 1 in Human Cancers

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EZH2-Mediated H3K27me3 Is Invo ...... iver Cancer 1 in Human Cancers

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EZH2-Mediated H3K27me3 Is Invo ...... iver Cancer 1 in Human Cancers

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EZH2-Mediated H3K27me3 Is Invo ...... iver Cancer 1 in Human Cancers

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Carmen Chak-Lui Wong

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Chun-Ming Wong

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Joyce Man-Fong Lee

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Sandy Leung-Kuen Au

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P2860

DLC-1, a Rho GTPase-activating protein with tumor suppressor function, is essential for embryonic development

DLC-1 suppresses non-small cell lung cancer growth and invasion by RhoGAP-dependent and independent mechanisms

An oncogene-tumor suppressor cascade drives metastatic prostate cancer by coordinately activating Ras and nuclear factor-kappaB

Interaction of deleted in liver cancer 1 with tensin2 in caveolae and implications in tumor suppression

The phosphotyrosine-independent interaction of DLC-1 and the SH2 domain of cten regulates focal adhesion localization and growth suppression activity of DLC-1

DLC1 negatively regulates angiogenesis in a paracrine fashion

EZH2 is a marker of aggressive breast cancer and promotes neoplastic transformation of breast epithelial cells

A bivalent chromatin structure marks key developmental genes in embryonic stem cells

Chromatin modifications and their function

Role of histone H3 lysine 27 methylation in Polycomb-group silencing

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e68226

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scholarly article

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10.1371/JOURNAL.PONE.0068226

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6

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23826380

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3694912

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