KSR1 is overexpressed in endometrial carcinoma and regulates proliferation and TRAIL-induced apoptosis by modulating FLIP levels.

KSR1 is overexpressed in endometrial carcinoma and regulates proliferation and TRAIL-induced apoptosis by modulating FLIP levels.

Item
http://www.wikidata.org/entity/Q34805471
Hierarchical scaffolding of an ERK1/2 activation pathwayGenetic and Pharmacological Screens Converge in Identifying FLIP, BCL2, and IAP Proteins as Key Regulators of Sensitivity to the TRAIL-Inducing Anticancer Agent ONC201/TIC10.Suppression of Ras/Mapk pathway signaling inhibits Myc-induced lymphomagenesis.AMPK Promotes Aberrant PGC1β Expression To Support Human Colon Tumor Cell Survival.Endometrial carcinoma: molecular alterations involved in tumor development and progression.Molecular pathology of endometrial carcinoma.FLIP: molecular switch between apoptosis and necroptosis.Functional genomics identifies novel genes essential for clear cell renal cell carcinoma tumor cell proliferation and migration.Endometrial Carcinoma: Specific Targeted Pathways.Combination of Vorinostat and caspase-8 inhibition exhibits high anti-tumoral activity on endometrial cancer cells.Sorafenib induces endometrial carcinoma apoptosis by inhibiting Elk-1-dependent Mcl-1 transcription and inducing Akt/GSK3β-dependent protein degradation.ERK controls epithelial cell death receptor signalling and cellular FLICE-like inhibitory protein (c-FLIP) in ulcerative colitis.The scaffold protein KSR1, a novel therapeutic target for the treatment of Merlin-deficient tumors.Expression of the scaffold connector enhancer of kinase suppressor of Ras 1 (CNKSR1) is correlated with clinical outcome in pancreatic cancer.praja2 regulates KSR1 stability and mitogenic signalingGenetic variants of kinase suppressors of Ras (KSR1) to predict survival in patients with ERα-positive advanced breast cancer.Cellular FLICE-like inhibitory protein protects against cardiac hypertrophy by blocking ASK1/p38 signaling in mice.
P2860

KSR1 is overexpressed in endometrial carcinoma and regulates proliferation and TRAIL-induced apoptosis by modulating FLIP levels.

Item
http://www.wikidata.org/entity/Q34805471
description
2011 nî lūn-bûn
@nan
2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
KSR1 is overexpressed in endom ...... sis by modulating FLIP levels.
@ast
KSR1 is overexpressed in endom ...... sis by modulating FLIP levels.
@en
KSR1 is overexpressed in endom ...... sis by modulating FLIP levels.
@nl
type
label
KSR1 is overexpressed in endom ...... sis by modulating FLIP levels.
@ast
KSR1 is overexpressed in endom ...... sis by modulating FLIP levels.
@en
KSR1 is overexpressed in endom ...... sis by modulating FLIP levels.
@nl
prefLabel
KSR1 is overexpressed in endom ...... sis by modulating FLIP levels.
@ast
KSR1 is overexpressed in endom ...... sis by modulating FLIP levels.
@en
KSR1 is overexpressed in endom ...... sis by modulating FLIP levels.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P50
P577
P5875
P698
P921
P932
P356
P1433
P1476
KSR1 is overexpressed in endom ...... sis by modulating FLIP levels.
@en
P2093
P2860
P304
P31
P356
10.1016/J.AJPATH.2010.12.041
P407
P433
P478
P50
P577
P5875
P698
P921
P932