Human papillomaviruses and centrosome duplication errors: modeling the origins of genomic instability.
about
Mapping common deleted regions on 5p15 in cervical carcinoma and their occurrence in precancerous lesions.Excessive centrosome abnormalities without ongoing numerical chromosome instability in a Burkitt's lymphomaGenome stability pathways in head and neck cancersExpression of the HPV18/E6 oncoprotein induces DNA damage.Abnormal centrosome amplification in cells through the targeting of Ran-binding protein-1 by the human T cell leukemia virus type-1 Tax oncoproteinThe arithmetic of centrosome biogenesis.Deficiencies in the Fanconi anemia DNA damage response pathway increase sensitivity to HPV-associated head and neck cancerThe retroviral oncoprotein Tax targets the coiled-coil centrosomal protein TAX1BP2 to induce centrosome overduplication.Chlamydia induces anchorage independence in 3T3 cells and detrimental cytological defects in an infection model.High incidence of HPV-associated head and neck cancers in FA deficient mice is associated with E7's induction of DNA damage through its inactivation of pocket proteinsKaposi's sarcoma-associated herpesvirus infection of blood endothelial cells induces lymphatic differentiation.Mechanisms of genomic instability in human cancer: insights from studies with human papillomavirus oncoproteins.Molecular transitions from papillomavirus infection to cervical precancer and cancer: Role of stromal estrogen receptor signaling.Human papillomaviruses and cell signaling.Detection of human papillomavirus in Chinese esophageal squamous cell carcinoma and its adjacent normal epithelium.Recent advances in the search for antiviral agents against human papillomaviruses.Molecular mechanisms of human papillomavirus-induced carcinogenesis.Virology and molecular pathogenesis of HPV (human papillomavirus)-associated oropharyngeal squamous cell carcinomaCentrosome and retroviruses: the dangerous liaisons.Immune concept of human papillomaviruses and related antigens in local cancer milieu of human cervical neoplasia.Mechanisms of chromosomal instability.Chromosomal gains measured in cytology samples from women with abnormal cervical cancer screening results.Chromosome aberrations in cells infected with bovine papillomavirus: comparing cutaneous papilloma, esophagus papilloma, and urinary bladder lesion cells.Human papillomavirus (HPV) type 18 induces extended growth in primary human cervical, tonsillar, or foreskin keratinocytes more effectively than other high-risk mucosal HPVs.High incidence of female reproductive tract cancers in FA-deficient HPV16-transgenic mice correlates with E7's induction of DNA damage response, an activity mediated by E7's inactivation of pocket proteins.The centrosome as potential target for cancer therapy and prevention.Heterogeneity of tumor endothelial cells.Manipulation of cellular DNA damage repair machinery facilitates propagation of human papillomaviruses.Smad4 deficiency in cervical carcinoma cells.LGALS3BP regulates centriole biogenesis and centrosome hypertrophy in cancer cells.Merkel cell polyomavirus small T antigen induces genome instability by E3 ubiquitin ligase targeting.Disruption of the retinoblastoma pathway by small interfering RNA and ectopic expression of the catalytic subunit of telomerase lead to immortalization of human ovarian surface epithelial cells.The MCT-1 oncogene product impairs cell cycle checkpoint control and transforms human mammary epithelial cells.Beta human papillomavirus E6 expression inhibits stabilization of p53 and increases tolerance of genomic instability.Mitosis is not the only distributor of mutated cells: non-mitotic endopolyploid cells produce reproductive genome-reduced cells.Racial/ethnic differences in HPV 16/18 genotypes and integration status among women with a history of cytological abnormalities.A comprehensive analysis of HPV integration loci in anogenital lesions combining transcript and genome-based amplification techniques.Contribution of Tumor Endothelial Cells in Cancer Progression.The multistage model of cancer development: some implications.
P2860
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P2860
Human papillomaviruses and centrosome duplication errors: modeling the origins of genomic instability.
description
2002 nî lūn-bûn
@nan
2002 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Human papillomaviruses and cen ...... rigins of genomic instability.
@ast
Human papillomaviruses and cen ...... rigins of genomic instability.
@en
Human papillomaviruses and cen ...... rigins of genomic instability.
@nl
type
label
Human papillomaviruses and cen ...... rigins of genomic instability.
@ast
Human papillomaviruses and cen ...... rigins of genomic instability.
@en
Human papillomaviruses and cen ...... rigins of genomic instability.
@nl
prefLabel
Human papillomaviruses and cen ...... rigins of genomic instability.
@ast
Human papillomaviruses and cen ...... rigins of genomic instability.
@en
Human papillomaviruses and cen ...... rigins of genomic instability.
@nl
P2860
P356
P1433
P1476
Human papillomaviruses and cen ...... rigins of genomic instability.
@en
P2093
Karl Münger
Stefan Duensing
P2860
P2888
P304
P356
10.1038/SJ.ONC.1205709
P407
P577
2002-09-01T00:00:00Z
P5875
P6179
1035487595