Modification of the erythroid transcription factor GATA-1 by SUMO-1.
about
GATA6 promotes angiogenic function and survival in endothelial cells by suppression of autocrine transforming growth factor beta/activin receptor-like kinase 5 signalingMaster regulatory GATA transcription factors: mechanistic principles and emerging links to hematologic malignanciesGATA switches as developmental driversComplex SUMO-1 regulation of cardiac transcription factor Nkx2-5GATA-1 associates with and inhibits p53.Exploring cellular memory molecules marking competent and active transcriptions.Controlling hematopoiesis through sumoylation-dependent regulation of a GATA factor.GATA1 function, a paradigm for transcription factors in hematopoiesisSENP1-mediated GATA1 deSUMOylation is critical for definitive erythropoiesis.Sumoylation regulates interaction of FOG1 with C-terminal-binding protein (CTBP)SUMO conjugation contributes to immune deviation in nonobese diabetic mice by suppressing c-Maf transactivation of IL-4.PDSM, a motif for phosphorylation-dependent SUMO modification.The Ufm1-activating enzyme Uba5 is indispensable for erythroid differentiation in miceMyocardin sumoylation transactivates cardiogenic genes in pluripotent 10T1/2 fibroblasts.SUMO modification regulates MafB-driven macrophage differentiation by enabling Myb-dependent transcriptional repressionHeat shock factors: integrators of cell stress, development and lifespan.Role of the GATA family of transcription factors in endocrine development, function, and diseaseSumoylation and regulation of cardiac gene expression.Transcriptional mechanisms underlying hemoglobin synthesis.GATA1 Binding Kinetics on Conformation-Specific Binding Sites Elicit Differential Transcriptional Regulation.Differential requirement for Gata1 DNA binding and transactivation between primitive and definitive stages of hematopoiesis in zebrafish.Emerging roles of the SUMO pathway in development.The GATA factor revolution in hematology.RSRC1 SUMOylation enhances SUMOylation and inhibits transcriptional activity of estrogen receptor β.PIASx acts as an Elk-1 coactivator by facilitating derepression.SUMO-1 modification of MEF2A regulates its transcriptional activity.Identification of estrogen receptor β as a SUMO-1 target reveals a novel phosphorylated sumoylation motif and regulation by glycogen synthase kinase 3β.Pias1 is essential for erythroid and vascular development in the mouse embryo.Sumoylation in Development and Differentiation.A CK2-RNF4 interplay coordinates non-canonical SUMOylation and degradation of nuclear receptor FXR.A 15q24 microdeletion in transient myeloproliferative disease (TMD) and acute megakaryoblastic leukaemia (AMKL) implicates PML and SUMO3 in the leukaemogenesis of TMD/AMKL
P2860
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P2860
Modification of the erythroid transcription factor GATA-1 by SUMO-1.
description
2004 nî lūn-bûn
@nan
2004 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Modification of the erythroid transcription factor GATA-1 by SUMO-1.
@ast
Modification of the erythroid transcription factor GATA-1 by SUMO-1.
@en
Modification of the erythroid transcription factor GATA-1 by SUMO-1.
@nl
type
label
Modification of the erythroid transcription factor GATA-1 by SUMO-1.
@ast
Modification of the erythroid transcription factor GATA-1 by SUMO-1.
@en
Modification of the erythroid transcription factor GATA-1 by SUMO-1.
@nl
prefLabel
Modification of the erythroid transcription factor GATA-1 by SUMO-1.
@ast
Modification of the erythroid transcription factor GATA-1 by SUMO-1.
@en
Modification of the erythroid transcription factor GATA-1 by SUMO-1.
@nl
P2093
P2860
P356
P1476
Modification of the erythroid transcription factor GATA-1 by SUMO-1
@en
P2093
Antonella Ronchi
Claudio Santoro
Fabio Avolio
Licio Collavin
Monica Gostissa
Paola Secco
P2860
P304
P356
10.1073/PNAS.0308605101
P407
P577
2004-06-01T00:00:00Z