Negative regulation of type I IFN expression by OASL1 permits chronic viral infection and CD8⁺ T-cell exhaustion.
about
Decoding the complexity of type I interferon to treat persistent viral infectionsOligoadenylate synthase-like (OASL) proteins: dual functions and associations with diseasesOASL-a new player in controlling antiviral innate immunityBacterial amyloid curli acts as a carrier for DNA to elicit an autoimmune response via TLR2 and TLR9New insights into type I interferon and the immunopathogenesis of persistent viral infections.OASL1 deficiency promotes antiviral protection against genital herpes simplex virus type 2 infection by enhancing type I interferon productionType I interferons in viral control and immune regulationCisplatin induces tolerogenic dendritic cells in response to TLR agonists via the abundant production of IL-10, thereby promoting Th2- and Tr1-biased T-cell immunity.Porcine 2', 5'-oligoadenylate synthetases inhibit Japanese encephalitis virus replication in vitro.RNAseq reveals hypervirulence-specific host responses to M. tuberculosis infection2'-5' Oligoadenylate synthetase-like 1 (OASL1) deficiency in mice promotes an effective anti-tumor immune response by enhancing the production of type I interferons.Dynamic Changes of Microglia/Macrophage M1 and M2 Polarization in Theiler's Murine Encephalomyelitis.Controlled infection with a therapeutic virus defines the activation kinetics of human natural killer cells in vivo.Transcriptomic Signatures of Tacaribe Virus-Infected Jamaican Fruit Bats.The Significance of Type-I Interferons in the Pathogenesis and Therapy of Human Immunodeficiency Virus 1 Infection.The Role of Interferon in Persistent Viral Infection: Insights from Murine Norovirus.OASL1 Traps Viral RNAs in Stress Granules to Promote Antiviral Responses.Type I IFN signaling blockade by a PASylated antagonist during chronic SIV infection suppresses specific inflammatory pathways but does not alter T cell activation or virus replication
P2860
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P2860
Negative regulation of type I IFN expression by OASL1 permits chronic viral infection and CD8⁺ T-cell exhaustion.
description
2013 nî lūn-bûn
@nan
2013 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Negative regulation of type I ...... on and CD8⁺ T-cell exhaustion.
@ast
Negative regulation of type I ...... on and CD8⁺ T-cell exhaustion.
@en
Negative regulation of type I ...... on and CD8⁺ T-cell exhaustion.
@nl
type
label
Negative regulation of type I ...... on and CD8⁺ T-cell exhaustion.
@ast
Negative regulation of type I ...... on and CD8⁺ T-cell exhaustion.
@en
Negative regulation of type I ...... on and CD8⁺ T-cell exhaustion.
@nl
prefLabel
Negative regulation of type I ...... on and CD8⁺ T-cell exhaustion.
@ast
Negative regulation of type I ...... on and CD8⁺ T-cell exhaustion.
@en
Negative regulation of type I ...... on and CD8⁺ T-cell exhaustion.
@nl
P2093
P2860
P1433
P1476
Negative regulation of type I ...... ion and CD8⁺ T-cell exhaustion
@en
P2093
Chan Hee Park
Myeong Sup Lee
Young-Joon Kim
Yun Hee Jeong
P2860
P304
P356
10.1371/JOURNAL.PPAT.1003478
P50
P577
2013-07-18T00:00:00Z