Inducible depletion of satellite cells in adult, sedentary mice impairs muscle regenerative capacity without affecting sarcopenia. - Wikidata - awgldk - TriplyDB

Inducible depletion of satellite cells in adult, sedentary mice impairs muscle regenerative capacity without affecting sarcopenia.

Inducible depletion of satellite cells in adult, sedentary mice impairs muscle regenerative capacity without affecting sarcopenia.

http://www.wikidata.org/entity/Q34892571

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When stem cells grow old: phenotypes and mechanisms of stem cell aging The ins and outs of muscle stem cell aging Satellite cells in human skeletal muscle plasticity A muscle stem cell for every muscle: variability of satellite cell biology among different muscle groups Skeletal muscle wasting in cachexia and sarcopenia: molecular pathophysiology and impact of exercise training Klotho, stem cells, and aging Interactions between muscle stem cells, mesenchymal-derived cells and immune cells in muscle homeostasis, regeneration and disease The quasi-parallel lives of satellite cells and atrophying muscle The central role of muscle stem cells in regenerative failure with aging Regulation of Muscle Stem Cell Functions: A Focus on the p38 MAPK Signaling Pathway Ageing in relation to skeletal muscle dysfunction: redox homoeostasis to regulation of gene expression Myogenic Progenitor Cells Control Extracellular Matrix Production by Fibroblasts during Skeletal Muscle Hypertrophy Postexercise essential amino acid supplementation amplifies skeletal muscle satellite cell proliferation in older men 24 hours postexercise The effect of calorie restriction on mouse skeletal muscle is sex, strain and time-dependent.Hypothalamic stem cells control ageing speed partly through exosomal miRNAs.Age affects the contraction-induced mitochondrial redox response in skeletal muscle.The altered fate of aging satellite cells is determined by signaling and epigenetic changes.Regulation of Muscle Satellite Cell Function in Tissue Homeostasis and Aging Skeletal muscle morphology in sarcopenia defined using the EWGSOP criteria: findings from the Hertfordshire Sarcopenia Study (HSS).Increases of M2a macrophages and fibrosis in aging muscle are influenced by bone marrow aging and negatively regulated by muscle-derived nitric oxide.Inducible depletion of adult skeletal muscle stem cells impairs the regeneration of neuromuscular junctions.Narciclasine attenuates diet-induced obesity by promoting oxidative metabolism in skeletal muscle.Reduced voluntary running performance is associated with impaired coordination as a result of muscle satellite cell depletion in adult mice.Pervasive satellite cell contribution to uninjured adult muscle fibers Pharyngeal Satellite Cells Undergo Myogenesis Under Basal Conditions and Are Required for Pharyngeal Muscle Maintenance Osteogenic potential of alpha smooth muscle actin expressing muscle resident progenitor cells.Transcriptional profiling and muscle cross-section analysis reveal signs of ischemia reperfusion injury following total knee arthroplasty with tourniquet.Diabetic mice exhibited a peculiar alteration in body composition with exaggerated ectopic fat deposition after muscle injury due to anomalous cell differentiation.High Intensity Training May Reverse the Fiber Type Specific Decline in Myogenic Stem Cells in Multiple Sclerosis Patients.Molecular circuitry of stem cell fate in skeletal muscle regeneration, ageing and disease.Targeting β1-integrin signaling enhances regeneration in aged and dystrophic muscle in mice.Reduced Notch signalling leads to postnatal skeletal muscle hypertrophy in Pofut1cax/cax mice.The effect of lengthening contractions on neuromuscular junction structure in adult and old mice.Loss of niche-satellite cell interactions in syndecan-3 null mice alters muscle progenitor cell homeostasis improving muscle regeneration.Glucocorticoids increase skeletal muscle NF-κB inducing kinase (NIK): links to muscle atrophy.Aged Muscle Demonstrates Fiber-Type Adaptations in Response to Mechanical Overload, in the Absence of Myofiber Hypertrophy, Independent of Satellite Cell Abundance.Klotho gene silencing promotes pathology in the mdx mouse model of Duchenne muscular dystrophy.Changes in Communication between Muscle Stem Cells and their Environment with Aging.Requirement of myomaker-mediated stem cell fusion for skeletal muscle hypertrophy.Dynamic changes in heparan sulfate during muscle differentiation and ageing regulate myoblast cell fate and FGF2 signalling.

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Inducible depletion of satellite cells in adult, sedentary mice impairs muscle regenerative capacity without affecting sarcopenia.

http://www.wikidata.org/entity/Q34892571

description

2014 nî lūn-bûn

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2014 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Inducible depletion of satelli ...... without affecting sarcopenia.

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Inducible depletion of satelli ...... without affecting sarcopenia.

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Inducible depletion of satelli ...... without affecting sarcopenia.

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Inducible depletion of satelli ...... without affecting sarcopenia.

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Inducible depletion of satelli ...... without affecting sarcopenia.

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Inducible depletion of satelli ...... without affecting sarcopenia.

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Inducible depletion of satelli ...... without affecting sarcopenia.

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P2093

Charlotte A Peterson

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Christopher S Fry

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Esther E Dupont-Versteegden

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Fujun Liu

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Janna R Jackson

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John J McCarthy

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Jonah D Lee

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Jyothi Mula

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Lin Yang

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P2860

Decreased specific force and power production of muscle fibers from myostatin-deficient mice are associated with a suppression of protein degradation

Reduced satellite cell numbers and myogenic capacity in aging can be alleviated by endurance exercise

Restoring systemic GDF11 levels reverses age-related dysfunction in mouse skeletal muscle

Epidemiology of sarcopenia among the elderly in New Mexico

Adult pancreatic beta-cells are formed by self-duplication rather than stem-cell differentiation

Rejuvenation of aged progenitor cells by exposure to a young systemic environment

Increased Wnt signaling during aging alters muscle stem cell fate and increases fibrosis

p38 MAPK signaling underlies a cell-autonomous loss of stem cell self-renewal in skeletal muscle of aged mice.

The aged niche disrupts muscle stem cell quiescence.

Geriatric muscle stem cells switch reversible quiescence into senescence.

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10.1038/NM.3710

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1

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Christopher L. Mendias

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2014-12-15T00:00:00Z

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269717222

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25501907

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4289085

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