A resveratrol and polyphenol preparation suppresses oxidative and inflammatory stress response to a high-fat, high-carbohydrate meal. - Wikidata - awgldk - TriplyDB

A resveratrol and polyphenol preparation suppresses oxidative and inflammatory stress response to a high-fat, high-carbohydrate meal.

A resveratrol and polyphenol preparation suppresses oxidative and inflammatory stress response to a high-fat, high-carbohydrate meal.

http://www.wikidata.org/entity/Q34894173

about

Resveratrol and clinical trials: the crossroad from in vitro studies to human evidence Resveratrol Inhibition of Cellular Respiration: New Paradigm for an Old Mechanism Neuroprotective properties and mechanisms of resveratrol in in vitro and in vivo experimental cerebral stroke models Nitric oxide, oxidative stress, and p66Shc interplay in diabetic endothelial dysfunction Polyphenols and the human brain: plant “secondary metabolite” ecologic roles and endogenous signaling functions drive benefits Systemic and renal oxidative stress in the pathogenesis of hypertension: modulation of long-term control of arterial blood pressure by resveratrol Flavonoids Affect Host-Microbiota Crosstalk through TLR Modulation Resveratrol improves cardiac contractility following trauma-hemorrhage by modulating Sirt1 Specific types of alcoholic beverage consumption and risk of type 2 diabetes: A systematic review and meta-analysis Resveratrol, from experimental data to nutritional evidence: the emergence of a new food ingredient.Comparisons of the Postprandial Inflammatory and Endotoxaemic Responses to Mixed Meals in Young and Older Individuals: A Randomised Trial.Physicochemical properties of dietary phytochemicals can predict their passive absorption in the human small intestine.Trans-Resveratrol Supplementation and Endothelial Function during the Fasting and Postprandial Phase: A Randomized Placebo-Controlled Trial in Overweight and Slightly Obese Participants.Combined phytochemistry and chemotaxis assays for identification and mechanistic analysis of anti-inflammatory phytochemicals in Fallopia japonica.Immunomodulatory effects of high-protein diet with resveratrol supplementation on radiation-induced acute-phase inflammation in rats.Resveratrol levels and all-cause mortality in older community-dwelling adults.Resveratrol inhibits enterovirus 71 replication and pro-inflammatory cytokine secretion in rhabdosarcoma cells through blocking IKKs/NF-κB signaling pathway.Resveratrol--pills to replace a healthy diet?Telmisartan ameliorates lipopolysaccharide-induced innate immune response through peroxisome proliferator-activated receptor-γ activation in human monocytes Interventions to Slow Aging in Humans: Are We Ready?The journey of resveratrol from yeast to human.Resveratrol pretreatment attenuates injury and promotes proliferation of neural stem cells following oxygen-glucose deprivation/reoxygenation by upregulating the expression of Nrf2, HO-1 and NQO1 in vitro.Diet and aging.Toxins, Toxicity, and Endotoxemia: A Historical and Clinical Perspective for Chiropractors.Sirtuin biology and relevance to diabetes treatment.Age-associated proinflammatory secretory phenotype in vascular smooth muscle cells from the non-human primate Macaca mulatta: reversal by resveratrol treatment.Resveratrol and fenofibrate ameliorate fructose-induced nonalcoholic steatohepatitis by modulation of genes expression Pilot study of resveratrol in older adults with impaired glucose tolerance.Skeletal muscle SIRT1 and the genetics of metabolic health: therapeutic activation by pharmaceuticals and exercise Resveratrol suppresses PAI-1 gene expression in a human in vitro model of inflamed adipose tissue.Resveratrol: How Much Wine Do You Have to Drink to Stay Healthy?A healthier approach to clinical trials evaluating resveratrol for primary prevention of age‐related diseases in healthy populations.Resveratrol: why is it a promising therapy for chronic kidney disease patients?Small molecule SIRT1 activators for the treatment of aging and age-related diseases.Resveratrol and health--a comprehensive review of human clinical trials.Pathogenic obesity and nutraceuticals.Resveratrol and diabetic cardiac function: focus on recent in vitro and in vivo studies.Anti-inflammatory effects of resveratrol and its potential use in therapy of immune-mediated diseases.Plant-based modulation of Toll-like receptors: an emerging therapeutic model.Anti-inflammatory properties of orange juice: possible favorable molecular and metabolic effects.

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P2860

A resveratrol and polyphenol preparation suppresses oxidative and inflammatory stress response to a high-fat, high-carbohydrate meal.

http://www.wikidata.org/entity/Q34894173

description

2011 nî lūn-bûn

@nan

2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2011 թվականի փետրվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

A resveratrol and polyphenol p ...... h-fat, high-carbohydrate meal.

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A resveratrol and polyphenol p ...... h-fat, high-carbohydrate meal.

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The Journal of Clinical Endocrinology and Metabolism

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A resveratrol and polyphenol p ...... h-fat, high-carbohydrate meal.

@en

P2093

Ajay Chaudhuri

http://www.w3.org/2001/XMLSchema#string

Anuritha Marumganti

http://www.w3.org/2001/XMLSchema#string

Chang Ling Sia

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Husam Ghanim

http://www.w3.org/2001/XMLSchema#string

Kelly Korzeniewski

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Paresh Dandona

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Sanaa Abuaysheh

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Teekam Lohano

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P2860

Orange juice neutralizes the proinflammatory effect of a high-fat, high-carbohydrate meal and prevents endotoxin increase and Toll-like receptor expression

SOCS-1 and SOCS-3 block insulin signaling by ubiquitin-mediated degradation of IRS1 and IRS2

The role of SOCS-3 in leptin signaling and leptin resistance

A rapid micropreparation technique for extraction of DNA-binding proteins from limiting numbers of mammalian cells

TLR4 links innate immunity and fatty acid-induced insulin resistance

An antiinflammatory and reactive oxygen species suppressive effects of an extract of Polygonum cuspidatum containing resveratrol.

Decline in transcriptional activity of Nrf2 causes age-related loss of glutathione synthesis, which is reversible with lipoic acid.

Molecular mechanism of nrf2 activation by oxidative stress.

Increase in plasma endotoxin concentrations and the expression of Toll-like receptors and suppressor of cytokine signaling-3 in mononuclear cells after a high-fat, high-carbohydrate meal: implications for insulin resistance.

SOCS-3 inhibits insulin signaling and is up-regulated in response to tumor necrosis factor-alpha in the adipose tissue of obese mice.

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1409-1414

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10.1210/JC.2010-1812

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5

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96

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2011-02-02T00:00:00Z

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21289251

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3085195

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