Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation. - Wikidata - awgldk - TriplyDB

Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.

Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.

http://www.wikidata.org/entity/Q34905557

about

Survival regulation of leukemia stem cells Inhibiting CCN1 blocks AML cell growth by disrupting the MEK/ERK pathway.Wnt signaling is involved in 6-benzylthioinosine-induced AML cell differentiation.KRas(G12D)-evoked leukemogenesis does not require β-catenin.γ-Catenin is overexpressed in acute myeloid leukemia and promotes the stabilization and nuclear localization of β-catenin Activation of wnt/β-catenin signaling blocks monocyte-macrophage differentiation through antagonizing PU.1-targeted gene transcription.Drosophila as a model to study the role of blood cells in inflammation, innate immunity and cancer.Multifaceted roles of GSK-3 and Wnt/β-catenin in hematopoiesis and leukemogenesis: opportunities for therapeutic intervention.Understanding of leukemic stem cells and their clinical implications.Pathways involved in Drosophila and human cancer development: the Notch, Hedgehog, Wingless, Runt, and Trithorax pathway.Identification of leukemia stem cells in acute myeloid leukemia and their clinical relevance.Interleukin-3-mediated regulation of β-catenin in myeloid transformation and acute myeloid leukemia.Regulation of normal and leukemic stem cells through cytokine signaling and the microenvironment.The viral oncogene Np9 acts as a critical molecular switch for co-activating β-catenin, ERK, Akt and Notch1 and promoting the growth of human leukemia stem/progenitor cells.Wnt/ß-catenin: a new therapeutic approach to acute myeloid leukemia.Caveolin-1 Influences LFA-1 Redistribution upon TCR Stimulation in CD8 T Cells.Development of gene expression-based risk score in cytogenetically normal acute myeloid leukemia patients.Pharmacological targeting of β-catenin in normal karyotype acute myeloid leukemia blasts.Reporting imaging flow cytometry data for publication: why mask the detail?The Cks1/Cks2 axis fine-tunes Mll1 expression and is crucial for MLL-rearranged leukaemia cell viability.Disruption of Wnt/β-catenin exerts anti-leukemia activity and synergizes with FLT3 inhibition in FLT3-mutant acute myeloid leukemia.Novel strategies for targeting leukemia stem cells: sounding the death knell for blood cancer.Focal adhesion kinase splice variants maintain primitive acute myeloid leukemia cells through altered Wnt signaling.

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P2860

Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.

http://www.wikidata.org/entity/Q34905557

description

2011 nî lūn-bûn

@nan

2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի փետրվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.

@ast

Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.

@en

Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.

@nl

type

label

Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.

@ast

Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.

@en

Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.

@nl

prefLabel

Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.

@ast

Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.

@en

Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.

@nl

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Heterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.

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Andrew Filby

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Arnaud Gandillet

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Dominique Bonnet

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François Lassailly

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Jacques Vargaftig

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T Andrew Lister

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P2860

Human acute myeloid leukemia is organized as a hierarchy that originates from a primitive hematopoietic cell

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Granulocyte-macrophage progenitors as candidate leukemic stem cells in blast-crisis CML

Wnt proteins are lipid-modified and can act as stem cell growth factors

Principles of cancer therapy: oncogene and non-oncogene addiction

Leukemia-initiating cells from some acute myeloid leukemia patients with mutated nucleophosmin reside in the CD34(-) fraction.

A murine model of CML blast crisis induced by cooperation between BCR/ABL and NUP98/HOXA9

Cdx4 dysregulates Hox gene expression and generates acute myeloid leukemia alone and in cooperation with Meis1a in a murine model.

The molecular basis of leukemia.

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770-780

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scholarly article

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10.1038/LEU.2011.17

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5

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25

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Emmanuel Griessinger

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2011-02-22T00:00:00Z

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49853830

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