Virulence factors of the human opportunistic pathogen Serratia marcescens identified by in vivo screening - Wikidata - awgldk - TriplyDB

Virulence factors of the human opportunistic pathogen Serratia marcescens identified by in vivo screening

Virulence factors of the human opportunistic pathogen Serratia marcescens identified by in vivo screening

http://www.wikidata.org/entity/Q34911227

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Comparative and functional genomic analyses of the pathogenicity of phytopathogen Xanthomonas campestris pv. campestris.Worms and flies as genetically tractable animal models to study host-pathogen interactions The phytopathogen Dickeya dadantii (Erwinia chrysanthemi 3937) is a pathogen of the pea aphid Running with the Red Queen: host-parasite coevolution selects for biparental sex Serratia marcescens internalization and replication in human bladder epithelial cells Diversity and specificity in the interaction between Caenorhabditis elegans and the pathogen Serratia marcescens The native microbiome of the nematode Caenorhabditis elegans: gateway to a new host-microbiome model Open-Air Biowarfare Testing and the Evolution of Values Microbial synergy via an ethanol-triggered pathway Genotypic comparison of Pantoea agglomerans plant and clinical strains Caenorhabditis elegans semi-automated liquid screen reveals a specialized role for the chemotaxis gene cheB2 in Pseudomonas aeruginosa virulence Insights into cross-kingdom plant pathogenic bacteria Evolution of the innate immune system: the worm perspective Serratia marcescens is injurious to intestinal epithelial cells.Molecular detection and analysis of a novel metalloprotease gene of entomopathogenic Serratia marcescens strains in infected Galleria mellonella.Serratia infections: from military experiments to current practice.From in vitro to in vivo Models of Bacterial Biofilm-Related Infections.Population dynamics and habitat sharing of natural populations of Caenorhabditis elegans and C. briggsae Cryptococcus neoformans gene involved in mammalian pathogenesis identified by a Caenorhabditis elegans progeny-based approach.Staphylococcus aureus virulence factors identified by using a high-throughput Caenorhabditis elegans-killing model Proteomic analysis of the systemic immune response of Drosophila.A model of bacterial intestinal infections in Drosophila melanogaster.High-throughput isolation and mapping of C. elegans mutants susceptible to pathogen infection.Virulent synergistic effect between Enterococcus faecalis and Escherichia coli assayed by using the Caenorhabditis elegans model.Predation on multiple trophic levels shapes the evolution of pathogen virulence.Phenotypic covariance of longevity, immunity and stress resistance in the caenorhabditis nematodes.Molecular bases of proliferation of Francisella tularensis in arthropod vectors.The Caenorhabditis elegans germ line regulates distinct signaling pathways to control lifespan and innate immunity.Capsule Production and Glucose Metabolism Dictate Fitness during Serratia marcescens Bacteremia.The RssAB two-component signal transduction system in Serratia marcescens regulates swarming motility and cell envelope architecture in response to exogenous saturated fatty acids Caenorhabditis elegans-based model systems for antifungal drug discovery.Caenorhabditis elegans as an alternative model host for legionella pneumophila, and protective effects of Bifidobacterium infantis.Serratia marcescens is able to survive and proliferate in autophagic-like vacuoles inside non-phagocytic cells Characterization of the dapA-nlpB genetic locus involved in regulation of swarming motility, cell envelope architecture, hemolysin production, and cell attachment ability in Serratia marcescens Activated and inactivated immune responses in Caenorhabditis elegans against Photorhabdus luminescens TT01 Genome-wide RNAi screen identifies genes involved in intestinal pathogenic bacterial infection RssAB-FlhDC-ShlBA as a major pathogenesis pathway in Serratia marcescens Role of pore-forming toxins in bacterial infectious diseases.Yersinia pestis kills Caenorhabditis elegans by a biofilm-independent process that involves novel virulence factors.Worms need microbes too: microbiota, health and aging in Caenorhabditis elegans

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Virulence factors of the human opportunistic pathogen Serratia marcescens identified by in vivo screening

http://www.wikidata.org/entity/Q34911227

description

2003 nî lūn-bûn

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2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2003 թվականի ապրիլին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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Virulence factors of the human ...... dentified by in vivo screening

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Virulence factors of the human ...... dentified by in vivo screening

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Virulence factors of the human ...... dentified by in vivo screening

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The EMBO Journal

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Virulence factors of the human ...... dentified by in vivo screening

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Alain Filloux

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B Brett Finlay

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C Léopold Kurz

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Gérard P F Michel

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Isabelle Vallet

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Ivo Steinmetz

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Jules A Hoffmann

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Marianne Aurouze

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Mitch Uh

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Sophie Chauvet

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Iron transport systems of Serratia marcescens

Basic local alignment search tool

Tricine-sodium dodecyl sulfate-polyacrylamide gel electrophoresis for the separation of proteins in the range from 1 to 100 kDa

Common virulence factors for bacterial pathogenicity in plants and animals

Pseudomonas aeruginosa GacA, a factor in multihost virulence, is also essential for biofilm formation

The Pseudomonas aeruginosa algC gene encodes phosphoglucomutase, required for the synthesis of a complete lipopolysaccharide core

Caenorhabditis elegans: a model genetic host to study Pseudomonas aeruginosa pathogenesis.

Two-component and phosphorelay signal transduction.

Inducible antibacterial defense system in C. elegans.

The SPI-3 pathogenicity island of Salmonella enterica

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1451-1460

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scholarly article

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10.1093/EMBOJ/CDG159

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7

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22

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Jean-Pierre Gorvel

Jonathan J. Ewbank

Dominique Ferrandon

Emmanuel Andres

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2003-04-01T00:00:00Z

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10835820

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12660152

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152903

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