Increased adherence to Caco-2 cells caused by disruption of the yhiE and yhiF genes in enterohemorrhagic Escherichia coli O157:H7
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Coping with low pH: molecular strategies in neutralophilic bacteriaFrom ingestion to colonization: the influence of the host environment on regulation of the LEE encoded type III secretion system in enterohaemorrhagic Escherichia coliNsrR, GadE, and GadX interplay in repressing expression of the Escherichia coli O157:H7 LEE pathogenicity island in response to nitric oxideExpression and regulation of the Escherichia coli O157:H7 effector proteins NleH1 and NleH2Global transcriptional response of Escherichia coli O157:H7 to growth transitions in glucose minimal medium.Differential expression of virulence and stress fitness genes between Escherichia coli O157:H7 strains with clinical or bovine-biased genotypes.Differential gene expression and adherence of Escherichia coli O157:H7 in vitro and in ligated pig intestinesClpXP protease controls expression of the type III protein secretion system through regulation of RpoS and GrlR levels in enterohemorrhagic Escherichia coli.Inactivation of alternative sigma factor 54 (RpoN) leads to increased acid resistance, and alters locus of enterocyte effacement (LEE) expression in Escherichia coli O157 : H7.Characterization of enterohemorrhagic Escherichia coli strains based on acid resistance phenotypesLysogeny with Shiga toxin 2-encoding bacteriophages represses type III secretion in enterohemorrhagic Escherichia coli.Adherence of Escherichia coli O157:H7 to epithelial cells in vitro and in pig gut loops is affected by bacterial culture conditionsThe GrlR-GrlA regulatory system coordinately controls the expression of flagellar and LEE-encoded type III protein secretion systems in enterohemorrhagic Escherichia coli.The host metabolite D-serine contributes to bacterial niche specificity through gene selection.Coordinate control of the locus of enterocyte effacement and enterohemolysin genes by multiple common virulence regulators in enterohemorrhagic Escherichia coli.Transcription of the ehx enterohemolysin gene is positively regulated by GrlA, a global regulator encoded within the locus of enterocyte effacement in enterohemorrhagic Escherichia coli.Enteropathogenic and enterohemorrhagic Escherichia coli virulence gene regulation.Escherichia coli O157:H7 survives within human macrophages: global gene expression profile and involvement of the Shiga toxinsCharacterization of the Escherichia coli O157:H7 Sakai GadE regulon.Pathogenesis of human enterovirulent bacteria: lessons from cultured, fully differentiated human colon cancer cell linesTranscriptome of enterohemorrhagic Escherichia coli O157 adhering to eukaryotic plasma membranes.Novel compounds targeting the enterohemorrhagic Escherichia coli type three secretion system reveal insights into mechanisms of secretion inhibition.Transient shielding of intimin and the type III secretion system of enterohemorrhagic and enteropathogenic Escherichia coli by a group 4 capsule.A positive regulatory loop controls expression of the locus of enterocyte effacement-encoded regulators Ler and GrlA.Differential virulence of clinical and bovine-biased enterohemorrhagic Escherichia coli O157:H7 genotypes in piglet and Dutch belted rabbit models.Characterization of enteropathogenic Escherichia coli mutants that fail to disrupt host cell spreading and attachment to substratum.Regulators encoded in the Escherichia coli type III secretion system 2 gene cluster influence expression of genes within the locus for enterocyte effacement in enterohemorrhagic E. coli O157:H7.Global regulation by horizontally transferred regulators establishes the pathogenicity of Escherichia coli.Identification and Characterization of a Phase-Variable Element That Regulates the Autotransporter UpaE in Uropathogenic Escherichia coliTranscriptional regulators of the GAD acid stress island are carried by effector protein-encoding prophages and indirectly control type III secretion in enterohemorrhagic Escherichia coli O157:H7
P2860
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P2860
Increased adherence to Caco-2 cells caused by disruption of the yhiE and yhiF genes in enterohemorrhagic Escherichia coli O157:H7
description
2003 nî lūn-bûn
@nan
2003 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Increased adherence to Caco-2 ...... hagic Escherichia coli O157:H7
@ast
Increased adherence to Caco-2 ...... hagic Escherichia coli O157:H7
@en
Increased adherence to Caco-2 ...... hagic Escherichia coli O157:H7
@nl
type
label
Increased adherence to Caco-2 ...... hagic Escherichia coli O157:H7
@ast
Increased adherence to Caco-2 ...... hagic Escherichia coli O157:H7
@en
Increased adherence to Caco-2 ...... hagic Escherichia coli O157:H7
@nl
prefLabel
Increased adherence to Caco-2 ...... hagic Escherichia coli O157:H7
@ast
Increased adherence to Caco-2 ...... hagic Escherichia coli O157:H7
@en
Increased adherence to Caco-2 ...... hagic Escherichia coli O157:H7
@nl
P2093
P2860
P1476
Increased adherence to Caco-2 ...... hagic Escherichia coli O157:H7
@en
P2093
Chihiro Sasakawa
Hiroshi Mori
Ichiro Tatsuno
Kazuki Taguchi
P2860
P304
P356
10.1128/IAI.71.5.2598-2606.2003
P407
P50
P577
2003-05-01T00:00:00Z