Mitochondrial transcription factor A, an endogenous danger signal, promotes TNFα release via RAGE- and TLR9-responsive plasmacytoid dendritic cells. - Wikidata - awgldk - TriplyDB

Mitochondrial transcription factor A, an endogenous danger signal, promotes TNFα release via RAGE- and TLR9-responsive plasmacytoid dendritic cells.

Mitochondrial transcription factor A, an endogenous danger signal, promotes TNFα release via RAGE- and TLR9-responsive plasmacytoid dendritic cells.

http://www.wikidata.org/entity/Q34951563

about

Bioenergetic dysfunction and inflammation in Alzheimer's disease: a possible connection Tolerance and Cross-Tolerance following Toll-Like Receptor (TLR)-4 and -9 Activation Are Mediated by IRAK-M and Modulated by IL-7 in Murine Splenocytes Mitochondria in lung disease The Roles of Mitochondrial Damage-Associated Molecular Patterns in Diseases Mitochondria in lung biology and pathology: more than just a powerhouse.BMPR2 preserves mitochondrial function and DNA during reoxygenation to promote endothelial cell survival and reverse pulmonary hypertension The danger model approach to the pathogenesis of the rheumatic diseases.Mitochondrial DNA damage-associated molecular patterns mediate a feed-forward cycle of bacteria-induced vascular injury in perfused rat lungs.Mechanism of Mitochondrial Transcription Factor A Attenuation of CpG-Induced Antibody Production.Mitochondrial ROS Induces Cardiac Inflammation via a Pathway through mtDNA Damage in a Pneumonia-Related Sepsis Model.Mitochondrial DNA in the regulation of innate immune responses Intra-Peritoneal Administration of Mitochondrial DNA Provokes Acute Lung Injury and Systemic Inflammation via Toll-Like Receptor 9.Innate Immune Activity in Glomerular Podocytes Circulating soluble receptor for advanced glycation end products (sRAGE) as a biomarker of emphysema and the RAGE axis in the lung.RAGE Enhances TLR Responses through Binding and Internalization of RNA.Plasma nuclear and mitochondrial DNA levels in acute myocardial infarction patients.Mitochondrial DNA and TLR9 drive muscle inflammation upon Opa1 deficiency.Mitochondrial Damage-Associated Molecular Patterns: From Inflammatory Signaling to Human Diseases.Scavenging Circulating Mitochondrial DNA as a Potential Therapeutic Option for Multiple Organ Dysfunction in Trauma Hemorrhage.

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Mitochondrial transcription factor A, an endogenous danger signal, promotes TNFα release via RAGE- and TLR9-responsive plasmacytoid dendritic cells.

http://www.wikidata.org/entity/Q34951563

description

2013 nî lūn-bûn

@nan

2013 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2013年の論文

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2013年論文

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2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

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2013年論文

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2013年论文

@wuu

name

Mitochondrial transcription fa ...... plasmacytoid dendritic cells.

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Mitochondrial transcription fa ...... plasmacytoid dendritic cells.

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Mitochondrial transcription fa ...... plasmacytoid dendritic cells.

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type

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Mitochondrial transcription fa ...... plasmacytoid dendritic cells.

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Mitochondrial transcription fa ...... plasmacytoid dendritic cells.

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Mitochondrial transcription fa ...... plasmacytoid dendritic cells.

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Mitochondrial transcription fa ...... plasmacytoid dendritic cells.

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Mitochondrial transcription fa ...... plasmacytoid dendritic cells.

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Mitochondrial transcription fa ...... plasmacytoid dendritic cells.

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Mitochondrial transcription fa ...... plasmacytoid dendritic cells.

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Elliott D Crouser

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Guohong Shao

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Mark W Julian

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Tracey L Papenfuss

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Zachary C Vangundy

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P2860

A critical cysteine is required for HMGB1 binding to Toll-like receptor 4 and activation of macrophage cytokine release

Plasmacytoid dendritic cells: linking innate and adaptive immunity

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

Human mitochondria-derived N-formylated peptides are novel agonists equally active on FPR and FPRL1, while Listeria monocytogenes-derived peptides preferentially activate FPR

Similarity of human mitochondrial transcription factor 1 to high mobility group proteins

Type I interferon: friend or foe?

IRF-7 is the master regulator of type-I interferon-dependent immune responses

Late endosome/lysosome-localized Rab7b suppresses TLR9-initiated proinflammatory cytokine and type I IFN production in macrophages

Endothelin-converting enzyme-1 regulates trafficking and signalling of the neurokinin 1 receptor in endosomes of myenteric neurones

Splenectomy inactivates the cholinergic antiinflammatory pathway during lethal endotoxemia and polymicrobial sepsis

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8

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Toll-like receptor

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