miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination - Wikidata - awgldk - TriplyDB

miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination

miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination

http://www.wikidata.org/entity/Q34956268

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Regulation of miRNA 219 and miRNA Clusters 338 and 17-92 in Oligodendrocytes miRNAs As Emerging Regulators of Oligodendrocyte Development and Differentiation miRNAs Participate in MS Pathological Processes and Its Therapeutic Response Tuning the cell fate of neurons and glia by microRNAs The nuclear envelope: an intriguing focal point for neurogenetic disease Extrinsic and Intrinsic Regulation of Axon Regeneration by MicroRNAs after Spinal Cord Injury The Emerging Role of microRNAs in Schizophrenia and Autism Spectrum Disorders Reduction of Dicer impairs Schwann cell differentiation and myelination Nuclear lamins are differentially expressed in retinal neurons of the adult rat retina MicroRNA-23a promotes myelination in the central nervous system Ancestral exposure to stress epigenetically programs preterm birth risk and adverse maternal and newborn outcomes Expression of progerin in aging mouse brains reveals structural nuclear abnormalities without detectible significant alterations in gene expression, hippocampal stem cells or behavior.¹H-MR spectroscopy of adult-onset autosomal dominant leukodystrophy with autonomic symptoms.MicroRNA profiles in hippocampal granule cells and plasma of rats with pilocarpine-induced epilepsy--comparison with human epileptic samples.MicroRNA expression profiling of oligodendrocyte differentiation from human embryonic stem cells.Identification of miRNomes associated with adult neurogenesis after stroke using Argonaute 2-based RNA sequencing Lamin-binding Proteins Lamin B1 levels modulate differentiation into neurons during embryonic corticogenesis Dicer ablation in oligodendrocytes provokes neuronal impairment in mice Nuclear lamins and neurobiology.Lamin B1 overexpression increases nuclear rigidity in autosomal dominant leukodystrophy fibroblasts.Adult-onset autosomal dominant leukodystrophy: linking nuclear envelope to myelin.Shaping the oligodendrocyte identity by epigenetic control MicroRNAs: novel regulators of oligodendrocyte differentiation and potential therapeutic targets in demyelination-related diseases.Inner nuclear membrane proteins: impact on human disease.MicroRNAs in oligodendrocyte and Schwann cell differentiation.Integrative nucleophosmin mutation-associated microRNA and gene expression pattern analysis identifies novel microRNA - target gene interactions in acute myeloid leukemia.Fine-Tuning Oligodendrocyte Development by microRNAs.Oxidative stress induces an ATM-independent senescence pathway through p38 MAPK-mediated lamin B1 accumulation.Specification and maintenance of oligodendrocyte precursor cells from neural progenitor cells: involvement of microRNA-7a.Coordinated control of oligodendrocyte development by extrinsic and intrinsic signaling cues Nuclear lamins in the brain - new insights into function and regulation.MicroRNA dysregulation in multiple sclerosis.Unwrapping myelination by microRNAs Lamin B1 fluctuations have differential effects on cellular proliferation and senescence.Nuclear lamin functions and disease.Expression and Genetic Analysis of MicroRNAs Involved in Multiple Sclerosis.Epigenetics, nervous system tumors, and cancer stem cells.The contrasting roles of lamin B1 in cellular aging and human disease LMNB1-related autosomal-dominant leukodystrophy: Clinical and radiological course.

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miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination

http://www.wikidata.org/entity/Q34956268

description

2009 nî lūn-bûn

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2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2009 թվականի փետրվարին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年论文

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name

miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination

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miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination

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miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination

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type

label

miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination

@ast

miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination

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miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination

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prefLabel

miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination

@ast

miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination

@en

miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination

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Disease Models & Mechanisms

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miR-23 regulation of lamin B1 is crucial for oligodendrocyte development and myelination

@en

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Shu-Ting Lin

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Ying-Hui Fu

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P2860

Common themes and cell type specific variations of higher order chromatin arrangements in the mouse

Modeling of a human circadian mutation yields insights into clock regulation by PER2

Myelin deficient mice: expression of myelin basic protein and generation of mice with varying levels of myelin

Lamin B1 is required for mouse development and nuclear integrity

Cre-lox-regulated conditional RNA interference from transgenes

A mammalian microRNA expression atlas based on small RNA library sequencing

Nuclear pore complexes form immobile networks and have a very low turnover in live mammalian cells

The microRNA miR-124 antagonizes the anti-neural REST/SCP1 pathway during embryonic CNS development

Lamin A-dependent nuclear defects in human aging

alpha-Synuclein locus triplication causes Parkinson's disease

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178-188

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10.1242/DMM.001065

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3-4

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oligodendrocyte

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