Malonyl CoA control of fatty acid oxidation in the ischemic heart.
about
The role of trimetazidine in cardiovascular disease: beyond an anti-anginal agentMalonyl-CoA decarboxylase is present in the cytosolic, mitochondrial and peroxisomal compartments of rat hepatocytesSubclinical cardiovascular damage and fat utilization in overweight/obese individuals receiving the same dietary and pharmacological interventionsMyocardial hypertrophy and the maturation of fatty acid oxidation in the newborn human heart.Second window of preconditioning normalizes palmitate use for oxidation and improves function during low-flow ischaemia.Myocardial energy metabolism during ischemia and the mechanisms of metabolic therapies.Biochemical dysfunction in heart mitochondria exposed to ischaemia and reperfusion.Molecular system bioenergetics: regulation of substrate supply in response to heart energy demands.The malonyl CoA axis as a potential target for treating ischaemic heart disease.Trimetazidine and cardioprotection: facts and perspectives.Peroxisome proliferator-activated receptors in cardiac energy metabolism and cardiovascular disease.Pleiotropic mechanisms of action of perhexiline in heart failure.Metabolic enzymes dysregulation in heart failure: the prospective therapy.Malonyl-CoA decarboxylase inhibition is selectively cytotoxic to human breast cancer cells.Hypoglycemia and revisable ST-elevation induced by Movento.beta-Hydroxybutyrate inhibits myocardial fatty acid oxidation in vivo independent of changes in malonyl-CoA content.Moderate severity heart failure does not involve a downregulation of myocardial fatty acid oxidation.Attenuation of fatty acid-induced apoptosis by low-dose alcohol in neonatal rat cardiomyocytes.5-Hydroxydecanoate is metabolised in mitochondria and creates a rate-limiting bottleneck for beta-oxidation of fatty acids.Malonyl-CoA decarboxylase inhibition suppresses fatty acid oxidation and reduces lactate production during demand-induced ischemia.Acute myocardial ischaemia induces specific alterations of ventricular mitochondrial function in experimental pigs.Metoprolol improves cardiac function and modulates cardiac metabolism in the streptozotocin-diabetic rat.Agmatine stimulates hepatic fatty acid oxidation: a possible mechanism for up-regulation of ureagenesis.EphrinA1-Fc attenuates myocardial ischemia/reperfusion injury in mice.Impact of Trimetazidine Treatment on 5-year Clinical Outcomes in Patients with Significant Coronary Artery Spasm: A Propensity Score Matching Study.Protective effects of ranolazine and propranolol, alone or combined, on the structural and functional alterations of cardiomyocyte mitochondria in a pig model of ischemia/reperfusion.Loss of Metabolic Flexibility in the Failing Heart.
P2860
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P2860
Malonyl CoA control of fatty acid oxidation in the ischemic heart.
description
2002 nî lūn-bûn
@nan
2002 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Malonyl CoA control of fatty acid oxidation in the ischemic heart.
@ast
Malonyl CoA control of fatty acid oxidation in the ischemic heart.
@en
Malonyl CoA control of fatty acid oxidation in the ischemic heart.
@nl
type
label
Malonyl CoA control of fatty acid oxidation in the ischemic heart.
@ast
Malonyl CoA control of fatty acid oxidation in the ischemic heart.
@en
Malonyl CoA control of fatty acid oxidation in the ischemic heart.
@nl
prefLabel
Malonyl CoA control of fatty acid oxidation in the ischemic heart.
@ast
Malonyl CoA control of fatty acid oxidation in the ischemic heart.
@en
Malonyl CoA control of fatty acid oxidation in the ischemic heart.
@nl
P356
P1476
Malonyl CoA control of fatty acid oxidation in the ischemic heart.
@en
P2093
Gary D Lopaschuk
Jason R B Dyck
P304
P356
10.1006/JMCC.2002.2060
P577
2002-09-01T00:00:00Z