Mutational analysis of open reading frames 62 and 71, encoding the varicella-zoster virus immediate-early transactivating protein, IE62, and effects on replication in vitro and in skin xenografts in the SCID-hu mouse in vivo.
about
Herpes simplex virus and varicella zoster virus, the house guests who never leaveGenome-wide mutagenesis reveals that ORF7 is a novel VZV skin-tropic factorRed-mediated transposition and final release of the mini-F vector of a cloned infectious herpesvirus genomeRole of the varicella-zoster virus gene product encoded by open reading frame 35 in viral replication in vitro and in differentiated human skin and T cells in vivo.Molecular mechanisms of varicella zoster virus pathogenesis.Varicella-zoster virus T cell tropism and the pathogenesis of skin infectionVaricella-Zoster virus pathogenesis and immunobiology: new concepts emerging from investigations with the SCIDhu mouse model.Comparison of virus transcription during lytic infection of the Oka parental and vaccine strains of Varicella-Zoster virusMutational analysis of the varicella-zoster virus ORF62/63 intergenic regionDownregulation of varicella-zoster virus (VZV) immediate-early ORF62 transcription by VZV ORF63 correlates with virus replication in vitro and with latency.Mouse models with human immunity and their application in biomedical research.A sequence within the varicella-zoster virus (VZV) OriS is a negative regulator of DNA replication and is bound by a protein complex containing the VZV ORF29 proteinThe immediate-early 63 protein of Varicella-Zoster virus: analysis of functional domains required for replication in vitro and for T-cell and skin tropism in the SCIDhu model in vivo.Natural selection for rash-forming genotypes of the varicella-zoster vaccine virus detected within immunized human hosts.Identification of non-essential loci within the Meleagrid herpesvirus 1 genome.The varicella-zoster virus genomeThe attenuated genotype of varicella-zoster virus includes an ORF0 transitional stop codon mutationAn Sp1/Sp3 site in the downstream region of varicella-zoster virus (VZV) oriS influences origin-dependent DNA replication and flanking gene transcription and is important for VZV replication in vitro and in human skinVaricella-zoster virus ORF 58 gene is dispensable for viral replication in cell culture.Mutational analysis of varicella-zoster virus (VZV) immediate early protein (IE62) subdomains and their importance in viral replication.Regulation of the varicella-zoster virus ORF3 promoter by cellular and viral factorsVaricella-zoster virus infection of human neural cells in vivo.Cellular transcription factor YY1 mediates the varicella-zoster virus (VZV) IE62 transcriptional activation.Differential requirement for cell fusion and virion formation in the pathogenesis of varicella-zoster virus infection in skin and T cells.Functions of the C-terminal domain of varicella-zoster virus glycoprotein E in viral replication in vitro and skin and T-cell tropism in vivo.Postherpetic neuralgia: from preclinical models to the clinic.Molecular studies of the Oka varicella vaccine.Insights into the function of tegument proteins from the varicella zoster virus.Phenotypic and genetic characterization of varicella-zoster virus mutants resistant to acyclovir, brivudine and/or foscarnet.Mutagenesis of varicella-zoster virus glycoprotein B: putative fusion loop residues are essential for viral replication, and the furin cleavage motif contributes to pathogenesis in skin tissue in vivo.Genetic analysis of varicella-zoster virus ORF0 to ORF4 by use of a novel luciferase bacterial artificial chromosome system.Varicella-Zoster Virus Activates CREB, and Inhibition of the pCREB-p300/CBP Interaction Inhibits Viral Replication In Vitro and Skin Pathogenesis In Vivo.Cyclin-dependent kinase 1/cyclin B1 phosphorylates varicella-zoster virus IE62 and is incorporated into virions.Replication of varicella-zoster virus in human skin organ cultureThe replication cycle of varicella-zoster virus: analysis of the kinetics of viral protein expression, genome synthesis, and virion assembly at the single-cell level.Development of recombinant varicella-zoster viruses expressing luciferase fusion proteins for live in vivo imaging in human skin and dorsal root ganglia xenografts.Varicella-Zoster Virus ORF9p Binding to Cellular Adaptor Protein Complex 1 Is Important for Viral Infectivity
P2860
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P2860
Mutational analysis of open reading frames 62 and 71, encoding the varicella-zoster virus immediate-early transactivating protein, IE62, and effects on replication in vitro and in skin xenografts in the SCID-hu mouse in vivo.
description
2003 nî lūn-bûn
@nan
2003 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Mutational analysis of open re ...... in the SCID-hu mouse in vivo.
@ast
Mutational analysis of open re ...... in the SCID-hu mouse in vivo.
@en
Mutational analysis of open re ...... in the SCID-hu mouse in vivo.
@nl
type
label
Mutational analysis of open re ...... in the SCID-hu mouse in vivo.
@ast
Mutational analysis of open re ...... in the SCID-hu mouse in vivo.
@en
Mutational analysis of open re ...... in the SCID-hu mouse in vivo.
@nl
prefLabel
Mutational analysis of open re ...... in the SCID-hu mouse in vivo.
@ast
Mutational analysis of open re ...... in the SCID-hu mouse in vivo.
@en
Mutational analysis of open re ...... in the SCID-hu mouse in vivo.
@nl
P2093
P2860
P1433
P1476
Mutational analysis of open re ...... in the SCID-hu mouse in vivo.
@en
P2093
Ann M Arvin
Bunji Sato
Hideki Ito
Leigh Zerboni
Marvin H Sommer
Stewart Hinchliffe
P2860
P304
P356
10.1128/JVI.77.10.5607-5620.2003
P407
P577
2003-05-01T00:00:00Z