Plasma concentrations of Gas6 and sAxl correlate with disease activity in systemic lupus erythematosus. - Wikidata - awgldk - TriplyDB

Plasma concentrations of Gas6 and sAxl correlate with disease activity in systemic lupus erythematosus.

Plasma concentrations of Gas6 and sAxl correlate with disease activity in systemic lupus erythematosus.

http://www.wikidata.org/entity/Q34979215

about

Design, synthesis, and validation of Axl-targeted monoclonal antibody probe for microPET imaging in human lung cancer xenograft.GAS6 receptor status is associated with dormancy and bone metastatic tumor formation Increased plasma levels of the soluble Mer tyrosine kinase receptor in systemic lupus erythematosus relate to disease activity and nephritis.Identification of the soluble form of tyrosine kinase receptor Axl as a potential biomarker for intracranial aneurysm rupture.Axl receptor blockade ameliorates pulmonary pathology resulting from primary viral infection and viral exacerbation of asthma Growth Arrest-Specific 6 Protein in Patients with Sjögren Syndrome: Determination of the Plasma Level and Expression in the Labial Salivary Gland Identification of Axl as a downstream effector of TGF-β1 during Langerhans cell differentiation and epidermal homeostasis Growth arrest-specific gene 6 (Gas6) levels are elevated in patients with chronic renal failure.Mer or Axl receptor tyrosine kinase inhibition promotes apoptosis, blocks growth and enhances chemosensitivity of human non-small cell lung cancer Circulating growth arrest-specific protein 6 levels are associated with erythropoietin resistance in hemodialysis patients.Mannose binding lectin: a biomarker of systemic lupus erythematosus disease activity.Vitamin K-dependent proteins GAS6 and Protein S and TAM receptors in patients of systemic lupus erythematosus: correlation with common genetic variants and disease activity Heightened cleavage of Axl receptor tyrosine kinase by ADAM metalloproteases may contribute to disease pathogenesis in SLE.Effect of GAS6 and AXL Gene Polymorphisms on Adiposity, Systemic Inflammation, and Insulin Resistance in Adolescents.Circulating levels of soluble MER in lupus reflect M2c activation of monocytes/macrophages, autoantibody specificities and disease activity O death where is thy sting? Immunologic tolerance to apoptotic self.Promising biomarkers for systemic lupus erythematosus.Incomplete clearance of apoptotic cells in systemic lupus erythematosus: pathogenic role and potential biomarker.Increased Mer and Axl receptor tyrosine kinase expression on glomeruli in lupus nephritis.Diminished soluble levels of growth arrest specific protein 6 and tyrosine kinase receptor Axl in patients with rheumatoid arthritis.Soluble Axl is an accurate biomarker of cirrhosis and hepatocellular carcinoma development: results from a large scale multicenter analysis.Axl, Ferritin, Insulin-Like Growth Factor Binding Protein 2, and Tumor Necrosis Factor Receptor Type II as Biomarkers in Systemic Lupus Erythematosus.Serum growth arrest-specific protein 6 levels are a reliable biomarker of disease activity in systemic lupus erythematosus.Plasma concentrations of growth arrest specific protein 6 and the soluble form of its tyrosine kinase receptor Axl in patients with systemic lupus erythematosus and Behçets disease.Soluble TAM receptor tyrosine kinases in rheumatoid arthritis: correlation with disease activity and bone destruction.Recombinant Gas6 augments Axl and facilitates immune restoration in an intracerebral hemorrhage mouse model.Antiphospholipid Antibodies Inhibit Trophoblast Toll-like Receptor and Inflammasome Negative Regulators.GAS6 intron 8 c.834 + 7G > A gene polymorphism in diabetic nephropathy.Plasma growth arrest-specific protein 6 levels in premenopausal and postmenopausal women: the role of endogenous estrogen.GAS6/TAM Pathway Signaling in Hemostasis and Thrombosis.

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P2860

Plasma concentrations of Gas6 and sAxl correlate with disease activity in systemic lupus erythematosus.

http://www.wikidata.org/entity/Q34979215

description

2011 nî lūn-bûn

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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2011 թվականի հունվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Plasma concentrations of Gas6 ...... systemic lupus erythematosus.

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Plasma concentrations of Gas6 ...... systemic lupus erythematosus.

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Plasma concentrations of Gas6 ...... systemic lupus erythematosus.

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type

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Plasma concentrations of Gas6 ...... systemic lupus erythematosus.

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Plasma concentrations of Gas6 ...... systemic lupus erythematosus.

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Plasma concentrations of Gas6 ...... systemic lupus erythematosus.

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Plasma concentrations of Gas6 ...... systemic lupus erythematosus.

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Plasma concentrations of Gas6 ...... systemic lupus erythematosus.

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Plasma concentrations of Gas6 ...... systemic lupus erythematosus.

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Plasma concentrations of Gas6 ...... systemic lupus erythematosus.

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Anders A Bengtsson

http://www.w3.org/2001/XMLSchema#string

Andreas Jönsen

http://www.w3.org/2001/XMLSchema#string

Björn Dahlbäck

http://www.w3.org/2001/XMLSchema#string

Carl Ekman

http://www.w3.org/2001/XMLSchema#string

Gunnar Sturfelt

http://www.w3.org/2001/XMLSchema#string

P2860

Vitamin K-dependent Gas6 activates ERK kinase and stimulates growth of cardiac fibroblasts

TNF-alpha, IL-6, and IL-1 expression is inhibited by GAS6 in monocytes/macrophages

axl, a transforming gene isolated from primary human myeloid leukemia cells, encodes a novel receptor tyrosine kinase

The protein encoded by a growth arrest-specific gene (gas6) is a new member of the vitamin K-dependent proteins related to protein S, a negative coregulator in the blood coagulation cascade

The Axl/Gas6 pathway is required for optimal cytokine signaling during human natural killer cell development

Dominant-negative inhibition of the Axl receptor tyrosine kinase suppresses brain tumor cell growth and invasion and prolongs survival

Gas6 regulates mesangial cell proliferation through Axl in experimental glomerulonephritis

Interferon-inducible gene expression signature in peripheral blood cells of patients with severe lupus

TAM receptors are pleiotropic inhibitors of the innate immune response

The 1982 revised criteria for the classification of systemic lupus erythematosus

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1064-1069

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scholarly article

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10.1093/RHEUMATOLOGY/KEQ459

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6

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50

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21278074

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3093930

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